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Understanding Protein Structure and Folding

The document provides an overview of protein structure, detailing the four levels: primary, secondary, tertiary, and quaternary structures, along with protein folding, stability, and denaturation. It explains the significance of amino acid sequences, local arrangements like alpha helices and beta sheets, and the importance of protein folding for functionality. Additionally, it discusses the consequences of protein misfolding, linking it to diseases such as Alzheimer's and prion diseases.

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0% found this document useful (0 votes)
37 views38 pages

Understanding Protein Structure and Folding

The document provides an overview of protein structure, detailing the four levels: primary, secondary, tertiary, and quaternary structures, along with protein folding, stability, and denaturation. It explains the significance of amino acid sequences, local arrangements like alpha helices and beta sheets, and the importance of protein folding for functionality. Additionally, it discusses the consequences of protein misfolding, linking it to diseases such as Alzheimer's and prion diseases.

Uploaded by

noleen652
Copyright
© All Rights Reserved
We take content rights seriously. If you suspect this is your content, claim it here.
Available Formats
Download as PPTX, PDF, TXT or read online on Scribd

Protein Structure

Likando Chababa
Protein Structure

• What are proteins?


• Four levels of structure (primary,
secondary, tertiary, quaternary)
• Protein folding and stability
• Protein denaturation
• Protein misfolding and diseases
What are proteins?
 Proteins are polymers of amino
acids joined together by peptide
bonds
Primary Structure
 It is the linear sequence of amino
acids
Voet Biochemistry 3e Page 65
© 2004 John Wiley & Sons, Inc.
Voet Biochemistry 3e Page 278
© 2004 John Wiley & Sons, Inc.

Primary structure of proinsulin


Secondary Structure
 It is the local three-dimensional
arrangement of a polypeptide
backbone
 Excluding the conformations (3D

arrangements) of its side chains


a Helix
 a helix is right-handed
 It has 3.6 residues (amino acids) per
turn
 The helix is stabilized by hydrogen
bonding
 Between carboxylic group and 4th

N–H group
 The amino acid side chains point
outward and downward from the
helix
 The core of the helix is tightly
Voet Biochemistry 3e Page 224
© 2004 John Wiley & Sons, Inc.

The right-handed a helix


Voet Biochemistry 3e Page 65
© 2004 John Wiley & Sons, Inc.
b Sheets
 Two or more polypeptide chains
form hydrogen bonding with
each other
 Also called pleated sheets

 They appear as folded structures

with edges
Voet Biochemistry 3e Page 228
© 2004 John Wiley & Sons, Inc.

A two-stranded b antiparallel pleated sheet


Voet Biochemistry 3e Page 65
© 2004 John Wiley & Sons, Inc.
Antiparallel b sheets
 Two or more hydrogen-bonded
polypeptide chains run in
opposite direction
 Hydrogen bonding is more

stable
Voet Biochemistry 3e Page 227
© 2004 John Wiley & Sons, Inc.

b pleated sheets. (a) The antiparallel b pleated sheets


Parallel b sheets
 Two or more hydrogen-bonded
polypeptide chains run in the
same direction
 Hydrogen bonding is less stable

(distorted)
Voet Biochemistry 3e Page 227
© 2004 John Wiley & Sons, Inc.

b pleated sheets. (b) The parallel b pleated sheets.


Other secondary
structures
 Turns (reverse turns)
 Loops

 b bends

 Random coils
 Supersecondary structures or
motifs:
bab motif: a helix connects two

b sheets
b hairpin: reverse turns connect

antiparallel b sheets
aa motif: two a helices together

b barrels: rolls of b sheets


Voet Biochemistry 3e Page 249
© 2004 John Wiley & Sons, Inc.

bab motif b hairpin aa motif

Schematic diagrams of supersecondary structures


Voet Biochemistry 3e Page 252
© 2004 John Wiley & Sons, Inc.

b barrel
Voet Biochemistry 3e Page 229
© 2004 John Wiley & Sons, Inc.

Secondary structure of proteins


(a-helix, b-sheets, loops, turns, random coils)
Tertiary Structure
 It is the three-dimensional
structure of an entire
polypeptide chain including side
chains
 It is the folding of secondary

structure and side chains


 Helices, sheets and side chains

combined to form tertiary


structure
Voet Biochemistry 3e Page 229
© 2004 John Wiley & Sons, Inc.

Tertiary structure of proteins


(Secondary structure + side chains)
 Domains
Polypeptide chains (>200
amino acids) fold into two or
more clusters known as
domains
Domains are units that look like

globular proteins
Domains are part of protein

subunits
Voet Biochemistry 3e Page 248
© 2004 John Wiley & Sons, Inc.

One subunit with two domains


Voet Biochemistry 3e Page 294
© 2004 John Wiley & Sons, Inc.

One subunit with three domains


Quaternary
Structure
 Many proteins contain two or
more polypeptide chains
 Each chain forms a three-
dimensional structure called
subunit
 It is the 3D arrangement of
different subunits of a protein
Voet Biochemistry 3e Page 67
© 2004 John Wiley & Sons, Inc.
 Hemoglobin is a globular protein
A multisubunit protein is called

oligomer
Composed of a b subunits (4
2 2
chains, 4 subunits)
Two same subunits are called

protomers
Protein folding
 Forces that stabilize proteins
 Protein denaturation
Forces that stabilize
protein structure
 Hydrophobic effect:
Nonpolar groups to minimize

their contacts with water


Nonpolar side chains are in the

interior of a protein
 Hydrogen bonding
 Electrostatic interactions (ion
pairing):
Between positive and negative

charges
 van der Waals forces (weak

polar forces):
Weak attractive or repulsive
forces between molecules
Protein
denaturation
 Denaturation: A process in which a
protein looses its native structure
 Factors that cause denaturation:
 Heat: disrupts hydrogen bonding
 Change in pH: alters ionization

states of aa
 Detergents: interfere with

hydrophobic interactions
 Chaotropic agents: ions or small

organic molecules that disrupt


hydrophobic interactions
Protein misfolding
 Every protein must fold to achieve
its normal conformation and
function
 Abnormal folding of proteins leads to

a number of diseases in humans


Alzheimer’s disease:
 b-amyloid protein is a misfolded

protein
 It forms fibrous deposits or plaques

in the brains of Alzheimer’s patients


Creutzfeldt-Jacob or prion disease:
 Piron protein is present in normal

brain tissue
 In diseased brains, the same protein

is misfolded
 Therefore it forms insoluble fibrous

aggregates that damage brain cells


That’s all!

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