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Fever and Rash Differential Diagnosis Guide

The document discusses several diseases that present with fever and rash, including measles, rubella, roseola infantum, and scarlet fever. It provides details on the causative agents, clinical manifestations such as symptoms and rash appearance, potential complications, diagnosis, treatment and prevention. Differential diagnosis of fever and rash is important as incorrect initial diagnosis could lead to fatal outcomes.

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0% found this document useful (0 votes)
20 views50 pages

Fever and Rash Differential Diagnosis Guide

The document discusses several diseases that present with fever and rash, including measles, rubella, roseola infantum, and scarlet fever. It provides details on the causative agents, clinical manifestations such as symptoms and rash appearance, potential complications, diagnosis, treatment and prevention. Differential diagnosis of fever and rash is important as incorrect initial diagnosis could lead to fatal outcomes.

Uploaded by

Nur Octaviani
Copyright
© All Rights Reserved
We take content rights seriously. If you suspect this is your content, claim it here.
Available Formats
Download as PPT, PDF, TXT or read online on Scribd

Fever and Rash

DJATNIKA SETIABUDI

Tropical Medicine Block/System


Medical Faculty
Padjadjaran University
Introduction

 Common problem in clinic

 Wide range of severity :


self limited disease  life-threatening

 Wrong 1st suspicion  fatal outcome

 Knowledge of differential diagnosis !!!


Differential Diagnosis (1)

1. Past history of infectious disease and


immunization
2. Type of pro-dromal period
3. Feature of the rash
4. Presence of pathognomic or other
diagnostic signs
5. Laboratory diagnostic tests
Differential Diagnosis (2)

Feature of the rash :


 Category:
- macular or maculo-papular :
Morbilli, Rubella, Roseola infantum, Scarlatina
- papulo-vesicular:
varicella, herpes zoster, variola
- petechial or purpuric: meningococcemia, DHF
 Character : discrete or confluent
 Distribution , Duration
 The appearance associated with fever?
Key Questions:

• Acute or Chronic (Recurrent)?


• When did it start?
• Pattern of Spread?
• Sick or Well?
• Pruritic?
• Medications?
• Exposures?
Describe What You See
A. Pattern/Distribution
Diffuse or Localized?
Mucous Membranes?
Palms & Soles?
Exposed vs. Unexposed Areas?
B. Individual Lesions
Color
Size
Blanches?
Characteristics
C. Other Physical Findings
Primary Lesions

Macule : Flat, cannot be palpated


Papule : Palpable solid lesions <1cm
Nodule : Palpable solid lesions >1cm
Vesicle : Raised, fluid-filled lesions <0.5cm
Bulla : Larger vesicle
Pustule : Purulent vesicle
Plaques: Aggregations of any of the above
primary lesions
Secondary Lesions
(Resulting from infection, trauma, or therapy)

Scales
Ulcers
Excoriations
Fissures
Crusts
Scars
TABLE 1
Common Primary Skin Lesions
Macule : Circumscribed area of change in normal skin color, with no skin
elevation or depression; may be any size

Papule : Solid, raised lesion up to 0.5 cm in greatest diameter

Nodule : Similar to papule but located deeper in the dermis or


subcutaneous tissue; differentiated from papule by palpability
and depth, rather than size

Plaque : Elevation of skin occupying a relatively large area in relation to


height; often formed by confluence of papules

Vesicle : Circumscribed, elevated, fluid-containing lesion less than 0.5 cm in


greatest diameter; may be intraepidermal or subepidermal in origin

Bulla :Same as vesicle, except lesion is more than 0.5 cm in diameter

Pustule : Circumscribed elevation of skin containing purulent fluid of variable


character (i.e., fluid may be white, yellow, greenish or hemorrhagic)
MORBILLI
(Measles; Rubeola)

Acute infection, contagious, caused by morbilli


Virus ( Famili Paramyxoviridae)
 3 stadia : Prodromal
Erupstion
Convalescens
 Endemic in developing countries
 Effective imunization program
 cases decreasing
 prone to older age group
Pathology

 Lesion particularly at :
- Skin
- Mucous membranes :
respiratory : nasopharyng, bronchi
digestive : oral cavity, intestine
- Conjungtiva
 Serous exudate,
mononuclear cell predominant
Clinical manifestations

Incubation period : 10 – 12 days


 Stadium prodromal :

- Coryza, Cough, Conjungtivitis


- “Koplik spots”
- Fever
 Stadium eruption :
- High fever : 40 – 40,50C
- Typical rash: maculopapular eritromatosus
Head  truncus  extremities
 Stadium convalescens:

- rash : hyperpigmentation macule/squama


- sign and symptoms resolve
Diagnosis

 Anamnesis :
- symptoms
- history : - contact
- imunization

 Clinical signs: typical

 Laboratorium :
- leukopenia,
- relative lymphocytosis
Complication

 Pneumonia / Bronchopneumonia ; Otitis media


 CNS : - meningoensefalitis
- Subacute Sclerosing Pan Encephalitis
 Persisten diarrhea
 protein lossing enteropathy
 Exaserbation of TBC
 Keratoconjunctivitis  blindness
 Secondary bacterial infection of skin
 Myocarditis
 Noma
Prognosis

 Particularly good prognosis

 CFR decreased

 Mortality caused by complication


Treatment

 Symptomatic

 Supportive

 Vitamin A :
Unicef/WHO reccomendation

 Management of complication
Prevention

 Active immunization:
- Measles vaccine
- when ? 9 months old
- booster: 15 months --> MMR

 Passive immunization
RUBELLA (German Measles)
 Acute infection, contagious, caused by rubela
virus (family Togaviridae)
 prodromal sign : + / -
 Rash : short periode  3 days
 Typical sign: lymphadenopathy
post auricular – suboccipital –
posterior colli
 Problems in pregnant women 
Congenital rubella Syndrome
Clinical Manifestation
 Incubation period : 18 + 3 days

 Mild prodromal sign:


- mild fever
- adolescent : more severe

 Rash : maculopapular
face  sentrifugal to
neck – trunk – extremities
 24 hours all of body
 resolve in 3rd day
Congenital rubella Syndrome

 Depend on gestational age


 Abortus
 Stillbirth
 Congenital anomaly

 gravida 1 – 4 weeks : 61%


5 – 8 weeks : 26 %
9 – 12 weeks : 8%
Congenital rubella Syndrome

 Opthalmologic : cataract – Micropthalmia


Glaukoma - chorioretinitis

 Cardiac : Septal Defect – PDA

 Neurologic : Meningoencephalitis –
Microcephaly – mental Retardation

 Auditoric : sensorineural deafness


Exanthem subitum
( Roseola infantum )

 Acute infection caused by “Human Herpes


Virus 6” ( some HHV 7 )

 Mostly in infant

 Sporadic ( sometimes epidemic)

 Typical feature :
- Severity of clinical sign unproportionally with
degree of fever
- Simultaniously resolve of rash and clinical sign
Clinical Manifestation

 Incubation period : 7 – 17 days ( + 10 days )


 Most common in 6 – 18 months old
 Fever
- abruptly high ; 39,4 – 41,20C
- Duration: 1 – 5 days ( mostly 3 – 4 days )
- Convulsion can occur
 Mild clinical sign :
- mild pharyngitis and coryza
 Rash : not specific
 macule / maculopapular ; rose colour :
chest –> exremities and neck  face
Appear while temperature has return to normal
Disappear on 1 – 2 days with normal skin
Prognosis

 Particularly good prognosis

 Bad prognosis :
- hyperpyrexia with persistent convulsion
Treatment

 Symptomatic

 Supportive

 Prevention : ?
SCARLET FEVER (SCARLATINA)

 Grup A beta-hemolytic Streptococcus


 pyrogenic toxin (erytrogenic toxin)
 Clinical manifestation :
- Incubation period : 1 – 7 days (mean : 3 days)
- Acute symptoms:
high fever – headache – vomiting- chills
- Signs: severe pharyngitis
 hyperemis – edema – eksudate- dysphagia
- “Circum oral pallor” dan “Pastia lines”
- “white strawbey tongue”  desquamation
 “red strawberry tongue”
Typical rash:
- Reddish macule / papule
 blanching on pressure
- Firstly on axilla, groin and neck
 24 hours all of body
- Severe disease : miliaria sudamina
- Petechiae can occur
- Desquamation occur from end of 1stweek
to 6th week of disease
Diagnosis

 History and physical examination

 Pharyngeal swab : bacterial culture

 Serologic : ASTO/ ASLO/ ASO


Complete blood count : leukositosis
CRP increased or (+) : not specific
Complication

 Local spread / per continuitatum:


- Sinusitis – Otitis media - Mastoiditis
- Retro / para parapharyngeal absces
- Bronchopneumonia
- Servical adenitis

 Hematogenic spread:
- Meningitis – Osteomyelitis
– Arthritis (septic)

 Non-suppurative (late) complication:


- Acute rhematic fever
- Acute Glomerulonephritis
Treatment

 Antibiotics :
- Penicillin group
- Allergy to penicillin :
Erythromycin – lincomycin –
Clindamycin- Cephadroxil

 Symptomatic

 Supportive

 Management of Complication

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