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ACID

The document provides an overview of various drug classes used for acid control, diarrhea management, laxatives, and antiemetics. It details the mechanisms, indications, contraindications, and nursing considerations for each drug class, including antacids, H2 receptor antagonists, proton pump inhibitors, and others. Additionally, it outlines the causes of constipation, types of laxatives, and the management of nausea and vomiting through different drug classes.

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0% found this document useful (0 votes)
3 views7 pages

ACID

The document provides an overview of various drug classes used for acid control, diarrhea management, laxatives, and antiemetics. It details the mechanisms, indications, contraindications, and nursing considerations for each drug class, including antacids, H2 receptor antagonists, proton pump inhibitors, and others. Additionally, it outlines the causes of constipation, types of laxatives, and the management of nausea and vomiting through different drug classes.

Uploaded by

reaperdiab
Copyright
© All Rights Reserved
We take content rights seriously. If you suspect this is your content, claim it here.
Available Formats
Download as DOCX, PDF, TXT or read online on Scribd

ACID-CONTROLLING,

ANTIDIARRHEAL, LAXATIVE, AND


ANTIEMETIC DRUGS
I. ACID-CONTROLLING AGENTS
Stomach Secretions:

Substance Function
HCl (hydrochloric acid) Made by parietal cells; aids digestion, kills microbes
Bicarbonate Buffers acid, prevents hyperacidity
Pepsinogen → Pepsin Breaks down proteins
Intrinsic factor Helps absorb vitamin B12
Mucus Protects stomach lining
Prostaglandins Promote mucus & bicarbonate, protect mucosa

Gastric Cell Types

Cell Function
Parietal Secrete HCl → maintain pH 1–4; site of action for acid-controlling drugs
Chief Secrete pepsinogen (protein digestion)
Mucous cells Secrete mucus barrier

Acid-Related Disorders

 GERD → reflux of acid → esophagitis, Barrett’s esophagus


 PUD (Peptic Ulcer Disease) → gastric or duodenal ulcers (often H. pylori)
 Hyperacidity/Dyspepsia → heartburn, indigestion
 Stress ulcers → in critically ill patients (e.g., Curling or Cushing ulcers)
 H. pylori treatment:
o Triple therapy: PPI + clarithromycin + (amoxicillin OR metronidazole)
o Quadruple therapy: PPI + bismuth + tetracycline + metronidazole

1. Antacids

Mechanism: Neutralize acid, ↑ gastric pH, stimulate mucus/bicarb/PG protection


Do NOT prevent acid production.
Indications:

 Heartburn, GERD, PUD, gastritis

Contraindications:

 Renal failure, GI obstruction, electrolyte imbalance, pregnancy (caution)

Drug Interactions:

 ↓ absorption of other drugs (chelation)


 ↑ stomach pH → affects absorption
 Take 1–2 hrs apart from other meds

Types:

Type Examples Notes


Aluminum hydroxide
Aluminum salts Constipation, hypophosphatemia
(Maalox)
Magnesium hydroxide (Milk
Magnesium salts Diarrhea; avoid in renal failure
of Magnesia)
Constipation, kidney stones, milk-alkali
Calcium salts Tums
syndrome
Sodium Quick relief, risk of metabolic alkalosis,
Alka-Seltzer
bicarbonate avoid in HF/renal pts

Nursing Tips:

 Take with 240 mL water


 Avoid long-term use (acid rebound)
 Chew thoroughly

2. H2 Receptor Antagonists (“-tidine”)

Examples:

 Cimetidine, Ranitidine (Zantac), Famotidine (Pepcid)

Mechanism:

 Blocks H2 receptors → ↓ HCl secretion, ↑ pH


Uses:

 GERD, PUD, esophagitis, stress ulcer prophylaxis

Adverse Effects:

 Confusion (elderly), hypotension (IV), impotence (Cimetidine), gynecomastia


 Thrombocytopenia (rare)

Interactions:

 Cimetidine inhibits liver enzymes → ↑ warfarin, phenytoin, theophylline


 Smoking ↓ drug effectiveness

Nursing Notes:

 Take 30–60 min before meals


 Do not take with antacids
 Monitor LFTs, renal labs

3. Proton Pump Inhibitors (PPIs) — “-prazole”

Examples: Omeprazole, Pantoprazole, Lansoprazole, Esomeprazole

Mechanism:

 Irreversibly blocks H⁺/K⁺ ATPase pump → stops acid secretion

Indications:

 GERD, PUD, Zollinger-Ellison syndrome, H. pylori therapy, stress ulcer prophylaxis

Adverse Effects:

 Long-term: Osteoporosis, ↓ magnesium, ↑ risk of C. difficile & pneumonia


 Possible kidney injury

Interactions:

 ↑ Warfarin, Phenytoin, Diazepam levels


 ↓ absorption of iron, digoxin, ampicillin

Nursing Notes:

 Take before meals, swallow whole


 If difficulty swallowing → open capsule, sprinkle on applesauce
 Avoid long-term use without follow-up

4. Other Acid-Controlling Drugs

Sucralfate:

 Coats ulcers → “liquid bandage”; promotes healing


 Give 1 hr before meals, 2 hr apart from other meds

Misoprostol:

 Synthetic prostaglandin → protects stomach lining


 Used for NSAID ulcer prevention
 🚫 Contraindicated in pregnancy (causes uterine contractions)

Simethicone (antiflatulent):

 Breaks gas bubbles; used for flatulence


 Found in Ovol, Maalox, Rolaids

II. ANTIDIARRHEAL DRUGS


💧 Diarrhea Overview

 Acute: < 2 weeks; infection or food-related


 Chronic: > 3 weeks; linked to IBS, diabetes, tumors

Goals: Decrease frequency, relieve cramps, restore fluids/electrolytes

Classes of Antidiarrheals

Class Mechanism Example Notes


Black stool/tongue, ↑
Coat intestinal Bismuth subsalicylate (Pepto-
Adsorbents bleeding time, avoid in
wall, bind toxins Bismol), Activated charcoal
kids → Reye’s syndrome
↓ peristalsis, dry Urinary retention, blurred
Anticholinergics Atropine, Hyoscyamine
secretions vision, 🚫 glaucoma
↓ bowel motility, Loperamide Constipation, CNS
Opiates ↑ water (Imodium), Diphenoxylate + depression, dependence
absorption Atropine (Lomotil) (Lomotil)
Restore normal
Probiotics Lactobacillus acidophilus Used after antibiotics
flora
Nursing Notes:

 Avoid in infectious diarrhea (keeps pathogen inside)


 Do not give bismuth to <16 yrs (Reye’s)
 Assess for dehydration, electrolytes

III. LAXATIVES
Causes of Constipation

 ↓ fiber, ↓ fluids, inactivity, meds (opioids, iron, anticholinergics)

🧾 Laxative Types & Key Points

Type Mechanism Example Notes


Absorb water → ↑
Safest; take with ≥240 mL
Bulk-forming stool bulk → reflex Psyllium (Metamucil)
water
peristalsis
Emollient (stool Lubricate & soften Docusate sodium Prevents straining; not
softener) stool (Colace), Mineral oil stimulant
Used for bowel prep,
Draw water into Lactulose, Polyethylene
Hyperosmotic hepatic encephalopathy
bowel → distention glycol (PEG), Glycerin
(Lactulose ↓ ammonia)
↑ osmotic pressure
Magnesium hydroxide, Fleet Quick action; avoid in
Saline → water in
enema renal/cardiac patients
intestines
Nerve stimulation Risk for dependency;
Stimulant Bisacodyl (Dulcolax), Senna
→ ↑ peristalsis fastest acting

Adverse Effects (General):

 Cramping, dehydration, electrolyte imbalance


 Bulk-forming → impaction
 Saline → magnesium toxicity

Nursing Notes:

 Do NOT give if bowel obstruction or undiagnosed pain


 Encourage fiber + fluids + activity
 Take with 6–8 oz water
 Long-term stimulant use → ↓ bowel tone
IV. ANTIEMETIC & ANTINAUSEA DRUGS
🧠 Pathway:

 Vomiting Center (VC) & Chemoreceptor Trigger Zone (CTZ) in brain


 Stimulated by neurotransmitters (ACh, dopamine, serotonin)

💊 Drug Classes

Class Mechanism Example Key Nursing Points


Block ACh in Motion sickness; 🚫
Anticholinergic Scopolamine patch
inner ear & VC glaucoma
Block
Antihistamines (H1 Dimenhydrinate Sedation, avoid
vestibular &
blockers) (Gravol), Meclizine, Promethazine alcohol
reticular ACh
Block
Antidopaminergic EPS, hypotension,
dopamine in Prochlorperazine, Promethazine
(Neuroleptic) sedation
CTZ
Risk for tardive
Block
dyskinesia; avoid
Prokinetic dopamine; ↑ Metoclopramide (Reglan)
with seizure
peristalsis
disorders
Block 5-HT3 Used for chemo &
Serotonin blockers in GI, CTZ, Ondansetron (Zofran) post-op N/V; HA
VC common
THC Act on cerebral For chemo or AIDS;
Dronabinol
(Cannabinoids) cortex stimulates appetite
Avoid in pregnancy
Natural
Herbal (Ginger) Zingiber officinale & with
antiemetic
anticoagulants

General Nursing Implications:

 Assess cause of nausea (drug, motion, chemo)


 Administer 30–60 min before chemo or travel
 Warn: Drowsiness → avoid driving/alcohol
 Monitor for dehydration, I&O
 Ondansetron may cause headache (treat with acetaminophen)
Antacids = neutralize acid (fast relief)

 H2 blockers = “-tidine” → ↓ acid production


 PPIs = “-prazole” → stop acid entirely
 Laxatives: “Bulk, Soft, Osmotic, Saline, Stimulate” 💩
 Antidiarrheals: “Slow down or coat”
 Antiemetics: target CTZ/VC — each class blocks a neurotransmitter

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