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Sle 2

Systemic Lupus Erythematosus (SLE) is a complex autoimmune disease characterized by a wide range of clinical manifestations, including constitutional symptoms, musculoskeletal issues, cutaneous manifestations, and organ involvement. Diagnosis is based on clinical features and immunological markers, with management focusing on disease control through pharmacological treatments and supportive care. The prognosis has improved significantly, with a five-year survival rate exceeding 90%, although long-term morbidity remains a concern.

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0% found this document useful (0 votes)
3 views6 pages

Sle 2

Systemic Lupus Erythematosus (SLE) is a complex autoimmune disease characterized by a wide range of clinical manifestations, including constitutional symptoms, musculoskeletal issues, cutaneous manifestations, and organ involvement. Diagnosis is based on clinical features and immunological markers, with management focusing on disease control through pharmacological treatments and supportive care. The prognosis has improved significantly, with a five-year survival rate exceeding 90%, although long-term morbidity remains a concern.

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draditiadvilkar
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We take content rights seriously. If you suspect this is your content, claim it here.
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Clinical Features

SLE is known as “the disease with a thousand faces” due to its wide
spectrum of manifestations.

1. Constitutional Symptoms
• Fatigue
• Fever
• Weight loss
• Malaise

2. Musculoskeletal System
• Symmetrical, non-erosive arthritis
• Commonly involves small joints
• Myalgia and muscle weakness

3. Cutaneous Manifestations
• Malar (butterfly) rash
• Photosensitivity
• Discoid rash
• Oral and nasal ulcers
• Alopecia
• Livedo reticularis

4. Renal Involvement (Lupus Nephritis)


• Proteinuria
• Hematuria
• Hypertension
• Edema
• Can progress to renal failure
• Classified into six classes based on renal biopsy (ISN/RPS
classification)

5. Hematological Manifestations
• Anemia (often hemolytic)
• Leukopenia
• Lymphopenia
• Thrombocytopenia

6. Cardiovascular System
6. Cardiovascular System
• Pericarditis (most common)
• Myocarditis
• Libman–Sacks endocarditis
• Accelerated atherosclerosis

7. Respiratory System
• Pleuritis
• Pleural effusion
• Interstitial lung disease
• Pulmonary hypertension

8. Neuropsychiatric Lupus
• Seizures
• Psychosis
• Cognitive dysfunction
• Headache
• Stroke

9. Gastrointestinal and Other Systems


• Vasculitis
• Mesenteric ischemia
• Pancreatitis
Investigations

Laboratory Findings

Hematological
• Anemia
• Leukopenia
• Thrombocytopenia
• Raised ESR (CRP often normal unless
infection)

Immunological Tests
• ANA (positive in >95% cases)
• Anti-dsDNA (disease activity marker)
• Anti-Sm (highly specific)
• Antiphospholipid antibodies
• Low complement levels (C3, C4)

Renal Tests
• Urinalysis (proteinuria, casts)
• Serum creatinine
• Renal biopsy if indicated
Diagnostic Criteria

ACR / EULAR Criteria

Diagnosis is based on a combination of:


• Clinical features
• Immunological markers

A weighted scoring system is used, with ANA


positivity as an entry criterion.

Differential Diagnosis
• Rheumatoid arthritis
• Dermatomyositis
• Systemic sclerosis
• Mixed connective tissue disease
• Vasculitis
• Antiphospholipid antibody syndrome
Management

There is no cure for SLE, but disease control is possible with


appropriate therapy.

General Measures
• Patient education
• Sun protection
• Regular follow-up
• Vaccinations

Pharmacological Treatment

1. NSAIDs
• For mild arthritis and serositis

2. Antimalarials
• Hydroxychloroquine (cornerstone therapy)
• Reduces flares and improves survival

3. Corticosteroids
• Used for moderate to severe disease
• Oral or intravenous depending on severity

4. Immunosuppressive Agents
• Azathioprine
• Methotrexate
• Mycophenolate mofetil
• Cyclophosphamide (for severe lupus nephritis)

5. Biologic Therapy
• Belimumab
• Rituximab (off-label in refractory cases)
Complications
• Chronic kidney disease
• Infections (due to immunosuppression)
• Cardiovascular disease
• Osteoporosis
• Pregnancy complications

Prognosis

The prognosis of SLE has improved significantly due to:


• Early diagnosis
• Better immunosuppressive therapy
• Improved supportive care

Five-year survival now exceeds 90%, though long-term morbidity


remains a concern.

Conclusion

Systemic Lupus Erythematosus is a complex autoimmune disease


with diverse clinical manifestations and unpredictable course. Early
recognition, appropriate investigation, and individualized treatment
are essential to prevent organ damage and improve quality of life. A
multidisciplinary approach involving rheumatologists, nephrologists,
and other specialists plays a crucial role in optimal disease
management.

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