Benefits of Therapeutic Exercise Explained
Benefits of Therapeutic Exercise Explained
Benefits of ex INCREASE
● Boost mood
● Combat chronic diseases
● Manage weight
● Boost energy level
● Promotes better sleep
● Self-confidence
● Strength and stamina
Effects of ex
● Increase HR
● Increase BP
● Increase breathing
● Increase body temp
● Increase metabolism
Mistakes of ex
● Valsalva:
○ Increase intrathoracic pressure
○ Slows HR
○ Increase venous pressure
● Cool-down:
○ Cessation (venous pooling)
○ Decrease the return of blood to the heart
● Warm-up:
○ Strain or injury
Stretches BENEFITS
● Flexibility and ROM
● Posture and stabilization
● Relaxation and circulation
Aerobic ex
● Use oxygen for energy
● Builds cardiovascular endurance
Fibers
● Slow-Twitch (type I) ENDURANCE → Aerobic
○ Slow contraction speed
○ Low force production, resistant to fatigue
● Fast Twitch (type IIa) POWER + ENDURANCE → Anaerobic Glycolysis
○ Fast contraction speed, low force production
○ Fatigue resistant
● Fast Twitch (type IIb) POWER → ATP-PC
○ Fast contraction speed, high force production
○ Susceptible to quick fatigue
Motion Types:
● Translations: whole body moves in a line
○ Rectilinear (straight): sled sliding
○ Curvilinear (curved): ball tossed in the air
● Rotation: body spins around the axis (elbow flexion)
● Active: done by muscles
● Passive: done by outside force (therapist, gravity)
Degrees of Freedom:
● Uniaxial: 1 movement
○ Hinge or pivot (humeroulnar, radioulnar)
● Biaxial: 2 movements
○ Condyloid/ellipsoid, and saddle (radiocarpal, carpometacarpal)
● Triaxial: 3 movements
○ Ball-and-socket (femoral-acetabular, glenoid-humeral)
Arthrokinematics:
● Roll: multiple points on another articular surface (tire rolling on ground)
● Slide: single point on one articular surface contacts multiple points on another articular
surface (box sliding on ground)
● Spin: single point on one articular surface rotates on a single point on another articular
surface (top spinning on one spot on the floor)
Types of Joints:
● Synarthrosis—no movement (skulls) STABILITY
● Syndesmosis—little movement (tibia/fibula)
● Gomphosis—peg in socket (teeth)
● Amphiarthrosis—slight movement (pubic symphysis) MOBILITY AND STABILITY
○ SHOCK ABSORPTION
● Diarthrosis—most common (freely movable) MOBILITY
Joint Positions:
Close-packed: joint surfaces fit tightly, least movement (STABILITY)
Open packed: joint surfaces move freely, unstable but flexible (MOVEMENT)
Levers:
1. 1st class: fulcrum in the middle (neck) BALANCE + POSTURE
2. 2nd class: resistance in the middle (calf) POWER
3. 3rd class: axis in the middle (elbow flexion) ROM + SPEED
Force Directions:
● Compression: push together
● Tension: pull apart
● Shear: parallel to surface (sliding)
● Bending: compression to one side, tension to the other
● Torsion: twisting
Newton's Laws:
● Inertia: an object stays still or in motion unless acted on
● Acceleration: heavier things need more force to move (F=MxA)
● Action: every action has an equal and opposite reaction
Types of Equilibrium:
● Stable: body returns to its former position after a light perturbation
● Unstable: body seeks a new position after a light perturbation
● Neutral: COG displaced but remains at the same level (rolling ball)
Movement System:
● Purposeful movement = muscle + nerves working together
● Brain → Nerves → Muscles
○ Afferent: sensory input ASCENDING
○ Efferent: sensory output DESCENDING
● Action potential: an electrical impulse that makes a muscle contract
● Depolarization: the cell becomes positive, triggering movement
Nervous System:
● Somatic: voluntary muscle and skin (moving arm)
● Visceral (autonomic): involuntary organs (heart rate)
● CNS: brain and SC (control center)
● PNS: nerves from CNS to body (communication lines)
Nerve Fibers:
● Type A: the fastest and thickest (MYELINATED)
○ Alpha:
■ Control skeletal muscle contraction (motor neurons)
■ Carry signals from muscle spindles for muscle position sense
(proprioception)
○ Beta:
■ Touch and pressure
○ Gamma:
■ Send signals to muscle spindles to help control muscle tone
○ Delta:
■ Pain and temperature (faster pain)
● Type B: medium size and speed (MYELINATED)
○ Carry AUTONOMIC signals: heart rate, smooth muscle, and glands
● Type C: smallest and slowest (UNMYELINATED)
○ Slow, dull, or aching pain
○ Autonomic nervous system
Motor Neurons:
● Alpha: activate EXTRAFUSAL fibers (main movers)
● Gamma: activate INTRAFUSAL fibers (muscle spindle sensors)
Skeletal muscle mechanics: study of how muscles’ internal + external forces affect strength
and movement
● Internal:
○ Muscle proteins (actin/myosin)
○ Length-tension
○ Motor unit recruitment
○ Fiber type and shape
● External:
○ Gravity
○ External load
○ Pulleys, position
○ Body orientation
Muscle Composition:
● Sarcomere: basic contractile tissue
● Contractile proteins:
○ Actin (thin) and Myosin (thick) = generate ACTIVE force
● Non-contractile proteins:
○ Support and create PASSIVE tension when stretched
Muscle Shapes:
● Fusiform: parallel → fast, LESS force (biceps)
● Pennate: angled → slower, MORE force (gastroc)
○ Pennate angle: angle between muscle fibers and tendons
■ Larger angle = more fibers fit = greater force
Pre-stretch concept:
● Pre-stretching before contraction = greater force output (squatting before jumping)
Muscular Insufficiency:
● Active: muscle SHORTENED too much → can't contract further (WEAK)
● Passive: muscle LENGTHENED too much → can’t lengthen further (TIGHT)
Bone Structure:
● Compact: hard outer layer STRENGTH AND STRUCTURE
● Cancellous (spongy): inner layer ABSORBS SHOCK
● Periosteum: tough outer covering that nourishes bone and serves as a muscle
attachment site FEEDS AND PROTECTS
● Marrow: makes blood cells
Neurology:
Brain Lobes and Key Structures:
Frontal Lobe: thinking, planning, movement, speech, personality
Parietal Lobe: sensation, touch, body awareness, spatial sense
Temporal Lobe: hearing, memory, understanding language
Occipital Lobe: vision and visual processing
Types of CVA?
- Ischemic: occlusive, depriving tissue of O₂ due to blockage (thrombosis, embolism)
- Hemorrhagic: bleeding in or around the brain due to a burst blood vessel
- Transient Ischemic Attack (TIA): “warning stroke,” temporary disruption of the blood
supply
Brunnstrom Stages:
Brunnstrom: uses reflexes and spasticity to regain movement
1. Flaccidity (approximation) → positioning, prevent contractures, PROM
2. Spasticity appears (approximation) → begin using reflexes and synergy patterns
3. Spasticity peaks (synergy patterns) → use synergies for function (facilitate
movement)
4. Movement begins out of synergy → work on isolated control
5. More complex movement → refine coordination
6. Near-normal control → functional retraining, speed, and accuracy
Flexor Synergy: UPPER extremity
● Scapula: elevation and retraction
● Shoulder: ABDuction and ER
● Elbow: flexion
● Forearm: supination
● Wrist: flexion
● Fingers: flexion with ADDuction
● Thumb: flexion with ADDuction
Positioning:
Supine (early phase) → keeps alignment; prevents pressure; good for relaxation and
tone reduction; REST AND RELAXATION
Sidelying on AFFECTED side → promotes weight bearing through the affected
arm/leg; normalizes tone; REGAIN SENSORY INPUT AND AWARENESS.
Sidelying on UNAFFECTED side → keeps affected arm protracted to avoid flexor tone;
supports leg in slight flexion; PREVENT NEGLECT, PROTECT SHOULDER
Sitting → encourages postural control, awareness of midline, and visual scanning;
BALANCE AND TRUNK RE-ED
Standing → improves WB, proprioception, and gait re-ed PRE-GAIT AND ENDURANCE
TRAINING
Clinical Concepts:
Pusher Syndrome: pushes toward the affected side due to midline misperception (R CVA
leans on the left side while seated) → visual cues (mirror), tactile input, vertical alignment
training.
Ramiste’s Phenomenon: resisted ABD/ADD on sound limb; same movement on
affected limb → initiates movement
Souques Phenomenon: raise arms above head, fingers automatically extend →
reduce hand flexor spasticity
PNF Progression:
1. Rhythmic Initiation → teach the pattern
2. Hold Relax Active Movement → wake up weak muscles
3. Repeated Contractions → build strength and activation
4. Hold Relax/Contract Relax → increase ROM
5. Alternating Isometrics → improve stability and endurance
6. Rhythmic Stabilization → strong postural stability and coordination
7. Agonist Reversal → improves control (STS)
8. Resisted Progression → builds coordination, timing, and endurance for
function (gait)
Kinesiology 2:
UE:
1. Nerves
2. Muscles, OINA
3. Mobs (arthrokinematics): shoulder, elbow, wrist
4. Axiohumeral, Scapulohumeral, Axioscapular
5. Force coupling
6. Planes and axes
7. End feels
8. Types of joints
9. Degrees of freedom
10. Grips
11. Functional position of the hand
End Feels:
● Normal:
○ Soft: elbow flexion (soft tissue)
○ Firm: most joints (capsule, muscle stretch)
○ Hard: elbow extension (bone on bone)
● Abnormal: empty (pain), spasm, boggy, springy
Muscle Groups
Axiohumeral (attach trunk → humerus)
● Latissimus Dorsi:
○ O: Lumbodorsal fascia, lower 3-4 ribs, iliac crest, SP T6-12
○ I: Bicipital groove of humerus
○ N: Thoracodorsal
○ A: ARM → Ext, ADD, IR; BACK/SPINE → thoracic flexion, lumbar
extension; SCAP → depression
● Pectoralis Major:
○ O: Sternal ½ of clavicle, ribs 1-6, rib cartilage, aponeurosis of external
oblique
○ I: Bicipital groove of humerus
○ N: Medial/Lateral Pec
○ A: ALL FIBERS → ADD, IR, elevate thoracic; UPPER → Flexion,
horizontal ADD; LOWER → Extension
● Subscapularis:
○ O: Subscapular fossa
○ I: Lesser tubercle of the humerus
○ N: Subscapular
○ A: IR
Force Couples:
Movement Muscles Working Together
Upward rotation of scap Upper trap + Lower trap + Serratus Anterior
Deltoid Lateral 3rd of the Deltoid Axillary Flex, IR, ABD, Ext,
clavicle, acromion. tuberosity ER
Spine of scapula
Pec Major Sternal ½ of clavicle, Bicipital groove Medial/Lateral Pec ADD, Flexion,
ribs 1-6, rib cartilage, Horizontal ADD,
aponeurosis of IR, Extension
external oblique
Flexor Carpi CFT Bases of 2nd & Medial Wrist flexion, radial
Radialis 3rd MC dev
Extensor Carpi Supracondylar ridge Base of 2nd Radial Wrist ext, radial
Radialis MC dev
Longus
Extensor Carpi CET, RCL Base of 3rd MC Radial Wrist ext, radial
Radialis Brevis dev
Extensor Carpi CET, post ulna Base of 5th MC Radial Wrist ext, ulnar
Ulnaris deviation
Precision Grip The thumb & one or more Writing, picking up a coin
fingers manipulate the object
Lateral (Key) Grip The thumb pad presses the Holding key
lateral side of the index finger
Shoulder Conditions
Impingement Syndrome:
Causes: Compression of the supraspinatus tendon, the subacromial bursa, and the long head
of the biceps under the acromion
● MOI:
○ Repetitive overhead activities
○ Poor posture → forward shoulder → decreased subacromial space
● Special Test:
○ Neer’s (IR + Flexion)
○ Hawkins-Kennedy (90° flex + IR)
○ Painful Arc (60-120°)
● Treatment:
○ Scapular strengthening: middle + lower traps, serratus anterior
○ RTC strengthening
○ Posterior capsule stretch
○ Postural training
○ Avoid impingement positions early (IR + ABD)
● Exercise Program:
○ Acute: Scapular squeezes, isometrics
○ Subacute: ER strengthening (bands), rows
○ Chronic: Overhead strengthening, functional training
AC Joint Dislocation:
● MOI:
○ FOOSH, direct blow
○ Clavicle pops up (step deformity)
● Special Test:
○ Crossarm
● Treatment:
○ Sling early
○ Gradual ROM
○ Avoid horizontal ADD early
○ Strengthen deltoid + scapular stabilizers
Shoulder Dislocation:
● MOI:
○ Forced ABD + ER = Anterior Dislocation
● Special Test:
○ Apprehension test
○ Relocation test (reduces symptoms)
● Associated lesions:
○ Hill-Sachs: compression fracture of the posterior humeral head
○ Bankart: anterior-inferior labrum tear
○ SLAP: superior labrum tear involving the biceps anchor
● Treatment:
○ Immobilization briefly
○ Strengthen RTC (ER) + scapular stabilizers
○ Avoid ABD + ER early
○ Closed-chain for joint protection
Subluxation:
● Common post-stroke
● The humeral head slides inferiorly
● Weak supraspinatus, deltoid
● Treatment:
○ Proper positioning
○ Strengthen the cuff & deltoid
○ No overhead pulling early
Adhesive Capsulitis:
● MOI:
○ Unknown; associated with diabetes
○ Capsular pattern loss: ER > ABD > IR
● Stages:
○ 1. Freezing (Painful) → pain dominates, ROM ↓
○ 2. Frozen → pain ↓, stiffness max
○ 3. Thawing → slow improvement in ROM
● Treatment:
○ AROM, PROM
○ Joint mobs:
■ ER → posterior glide
■ ABD → inferior glide
○ Heat + stretching
○ Scapular strengthening
● Exercise Program:
○ Pendulums
○ Wand ER
○ Table slides
○ Posterior capsule stretch
Thoracic Outlet Syndrome:
Structures compressed: Subclavian artery + vein, brachial plexus
Possible compression sites:
1. Between anterior + middle scalene
2. Between clavicle + 1st rib
3. Under pectoralis minor
● Symptoms:
○ Arm numbness, heaviness, fatigue
○ Color changes
○ Weak grip
● Test:
○ Roo’s
○ Adson’s
● Treatment:
○ Stretch scalenes, pec minor
○ Strengthen lower traps, serratus anterior
○ Postural training
○ First rib mobility
Therex Fundamentals:
Open Chain:
● Distal segment free
○ Bicep curl
● Better for isolating muscles
Closed Chain:
● Distal segment fixed
○ Push-up, wall slide
● Better for joint stability
Concentric:
● Muscle shortens
○ RAISING DB in bicep curl
Eccentric:
● Muscle lengthens
○ LOWERING DB in bicep curl
● Produces more force
● More DOMS
SCI: Injuries
What you can do functionally for each level
Complications at each level
Autonomic dysreflexia
HO
PD: Chronic, progressive neurological condition affecting the motor system due to
inability of dopamine receptors to work properly (basal ganglia)
- Symptoms:
- Bradykinesia (slow movement)
- Rigidity (know two types)
- Lead pipe: constant resistance / increased tone throughout entire
passive movement in any direction, regardless of speed
- Cog-wheel: Jerky, ratchet like response of tensing and letting go
- Tremors
- Akinesia (inability to initiate movement)
- Postural Instability: Leads to greater fear of falling due to loss of postural
extension
- Presentation
Here’s a clean, concise study sheet based on what you listed—organized by diagnosis
+ key points you’ll need for PTA exams/clinicals.
C1–C3
● Ventilator dependent
C4
● Power WC
C5
C6
C7
C8–T1
T2–T6
● Wheelchair independent
T7–T12
L1–L3
L4–S3
SCI Complications
● Autonomic Dysreflexia (T6 and above): BP↑, headache, flushing,
bradycardia → sit them up, find noxious stimulus
● Orthostatic Hypotension
● Pressure injuries
● Spasticity
● Neurogenic bowel/bladder
Autonomic Dysreflexia
● Intervention: sit upright, loosen clothing, check catheter, check bowel, call
medical team
● I – No Response
● IV – Confused / Agitated
● V – Confused / Inappropriate
● VI – Confused / Appropriate
Mechanisms of Injury
Key Symptoms
● Bradykinesia
● Rigidity
○ Lead Pipe: constant tone
● Resting Tremor
● Postural Instability
Presentation
● Masked face
Symptoms / Presentation
● Cognitive impairment
Treatment Goals
● Maintain independence
● Energy conservation
● Task-specific training
Neuro 2:
Complications:
● C4:
○ Diaphragm (partially), traps
○ Dependent for all mobility
● C5:
○ Elbow flexion (biceps)
○ Can feed self with adaptive equipment
○ Power WC
● C6:
○ Wrist extension (tenodesis)
○ Independent slide-board transfers possible
○ Manual WC with projections
○ Can drive with hand controls
● C7:
○ Triceps
○ Independent transfers
○ Manual WC
○ Pressure relief independently
● C8:
○ Hand intrinsics
○ Full independence with ADLs + WC mobility
Driving, Walking, Transfers:
● Coup-Contrecoup:
○ Coup: brain hits skull at site of impact
○ Contrecoup: brain rebounds → opposite side of impact
■ Predicts opposite lobe damage
● Lobes Function:
○ Frontal: judgement, motor planning, behavior
○ Parietal: sensation
○ Temporal: memory, hearing
○ Occipital: vision
○ Cerebellum: coordination
○ Brainstem: breathing, HR → MOST dangerous
● Signs of ICP:
○ Headache
○ Vomiting
○ Vision changes
○ Papilledema
○ LOC changes
○ Cushing’s Triad:
■ ↑ BP
■ ↓ HR
■ Irregular respirations
Total: 3-15
○ Scores:
■ 3-8 = severe
■ 9-12 = moderate
■ 13-15 = mild
● Rancho Levels:
○ 1-3 = Total A (severe)
○ 4-6 = MAX-MOD A (moderate)
○ 7-10 = MIN-I (mild)
● Parkinson’s Disease:
○ Symptoms:
■ Bradykinesia (MOST disabling)
■ Resting tremor
■ Rigidity:
● Cogwheel (jerky)
● Lead-pipe (constant)
■ Festination (short, shuffling steps)
■ Decreased arm swing
■ Stooped posture
■ Freezing episodes
○ Treatment:
■ BIG MOVEMENTS (LSVT BIG)
■ Rhythmic cueing (metronome)
■ Ambulation with trekking poles for arm swing
■ Trunk rotation exercises
■ Avoid dual-task early
■ Stretch flexors (chest, hip flexors)
● Multiple Sclerosis:
○ Symptoms:
■ Vision changes
■ Spasticity
■ Weakness
■ Fatigue
■ Heat intolerance
■ Sensory changes
■ Balance deficits
■ Intention tremor
○ Treatment Principles:
■ Cool environment
■ Energy conservation
■ Avoid overheating (Uhthoff’s phenomenon)
■ Submaximal strengthening
■ Frequent rest breaks
■ Functional training
■ Aerobic activity: morning is best