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Understanding Genetic Code Degeneracy

Chapter 14 discusses the genetic code, highlighting its degeneracy where multiple codons can specify the same amino acid, and the wobble mechanism that allows flexibility in codon-anticodon pairing. It also covers the rules governing the genetic code, types of mutations that can alter it, and the concept of suppressor mutations that can reverse harmful mutations. Additionally, the chapter emphasizes the nearly universal nature of the genetic code, which facilitates comparative genomics and gene expression across different organisms.

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0% found this document useful (0 votes)
4 views27 pages

Understanding Genetic Code Degeneracy

Chapter 14 discusses the genetic code, highlighting its degeneracy where multiple codons can specify the same amino acid, and the wobble mechanism that allows flexibility in codon-anticodon pairing. It also covers the rules governing the genetic code, types of mutations that can alter it, and the concept of suppressor mutations that can reverse harmful mutations. Additionally, the chapter emphasizes the nearly universal nature of the genetic code, which facilitates comparative genomics and gene expression across different organisms.

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chen0009563
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© All Rights Reserved
We take content rights seriously. If you suspect this is your content, claim it here.
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Chapter 14 Opener

The Genetic Code

Lin, Ching-Hsuan (林晉玄)


Phone: 3366-4449
E-mail: chinghsuanlin@[Link]
The Code Is Degenerate

Codon: degeneracy
Anticodon: wobble
64 possibilities, but only have 20-amino-acids language

 Degeneracy: each amino


acid is specified by more
than one codon

 Synonyms: codons
specifying the same amino
acid
Coding role - Degeneracy

 Often, when the first two


nucleotides are identical, the
third nucleotide can be either
C or U without changing the
code.

 A and G at the third position


are interchangeable as well.
Minimize the deleterious effects of mutations
Reasons for degeneracy

 Some tRNAs (a total of 40) could


recognize more than one different codons
(a total of 61).
 The wobble feature of the base at the 5’
end of the anticodon (also called the
wobble position).
Wobble in the Anticodon
[Link]
ki/Francis_Crick

In 1966, Francis Crick devised the wobble


concept. It states that the base at the 5’ end
of the anticodon is not as spatially confined
as the other two, allowing it to form
hydrogen bonds with more than one bases
located at the 3’ end of a codon.
Why wobble is allowed at the 5’
anticodon? --the structural basis

 The 3-D structure of tRNA shows that the stacking


interactions between the flat surfaces of the 3 anticodon
bases + 2 followed bases, and the first (5’) anticodon base
is positioned at the end of the stack, thus less restricted in
its movements.
 The 3’ base appears in the middle of the stack, resulting in
the restriction of its movements.
The first (5’) anticodon base is positioned at the
end of the stack
Wobble in the Anticodon
Inosine Is the Fifith Base in the Anticodon

[Link]

Inosine arises through enzymatic modification


of adenine to give the 6-keto group
The Wobble Rules
 The pairings permitted are those give
ribose-ribose distances close to that of
the standard A:U or G:C base pairs.
 The ribose-ribose distances:
---Purine-purine: too long
---Pyrimidine-pyrimidine: too short
The ribose-ribose distances for
the wobble pairs are close to
those of A:U or G:C base pairs
Three Codons Direct Chain Termination

 Three codons, UAA, UAG, and UGA signify


chain termination.
 They are not read by tRNAs but by proteins
called release factors (RF1 and RF2 in bacteria
and eRF1 in eukaryotes).
Assignment of Codons Using Repeating
Copolymers Built from Two or Three Nucleotides
Three Rules Govern the Genetic Code

Codons are read in a 5’ to 3’ direction in units of three


nucleotides

Codons are nonoverlapping and the message contains


no gaps

The message is translated in a fixed reading frame


which is set by the initiation codon.
Three Kinds of Point Mutations
Alter the Genetic Code
 Missense mutation: An alternation that changes a codon
specific for one amino acid to a codon specific for another
amino acid [Sense mutations do not alter genetic code]

 Nonsense or stop mutation: An alternation causing a


change to a chain-termination codon

 Frameshift mutation: mutations are insertions or


deletions of one or a small number of base pairs that
alter the reading frame
Ala Ala Ala Ala Ala Ala Ala Ala
5’ GCU GCU GCU GCU GCU GCU GCU GCU 3’

Frameshit

Ala Ala Ser Cys Cys Cys Cys Cys


5’ GCU GCU AGC UGC UGC UGC UGC UGC 3’

Frameshit 3 nucleotides

Ala Ala Ser Met Leu His Ala Ala


5’ GCU GCU AGC AUG CUG CAU GCU GCU 3’
The Harmful Mutations Can Be Reversed by a
Second Genetic Change
 Reverse (back) mutations: change an altered nucleotide
sequence back to its original arrangement

 Suppressor mutations: suppress the change due to


mutation at site A by producing an additional genetic
change at site B
(1) Intragenic suppression: same gene
(2) Intergenic suppression: another genes

Suppressor genes: genes that cause suppression of


mutations in other genes.
Suppression of Frame-Shift Mutations
(intragenic suppression)
Intergenic Suppression Involves Mutant tRNAs

Mutant tRNA genes suppress the effects of


nonsense mutations in protein-coding genes

They act by reading a stop codon as if it were a


signal for a specific amino acid
Intergenic Suppression Involves Mutant tRNAs
The Code Is Nearly Universal

Benefits of the universal codes


 Allow us to directly compare the protein coding
sequences among all organisms (comparative
genomics)

 Make it possible to express cloned copies of genes


encoding useful protein in different host organism.
Example: Human insulin expression in bacteria)
In certain subcellular organelles, the
genetic code is slightly different

 Mitochondrial tRNAs are unusual in the way


that they decode mitochondrial messages

 Only 22 tRNAs are present in mammalian


mitochondria. The U in the 5’ wobble position of
a tRNA is capable of recognizing all four bases in
the 3’ of the codon
Key points of the chapter
1. What is the degeneracy of genetic code? what is its importance?
and what is its molecular mechanisms?

2. What are the three roles governing the genetic code? What are
the mutations altering genetic code? What is the suppressor
mutation? and What are the best known intergenic suppressor
genes?

3. What are the benefits of the code universality?

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