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Understanding Neurons and Action Potentials

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15 views50 pages

Understanding Neurons and Action Potentials

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© All Rights Reserved
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5.

Specialized cells

• Previously we have talked about ‘generic’ cells

• This week we are going to look at cells that have become


‘specialized’

• Neurons and muscle cell are examples of specialized cells


Nerves

• NEURONS are specialized cells

• Neurons transmit signals through the body of multi-cellular


eukaryotes

• Action potentials travel down the neuron

• At the end of a neuron NEUROTRANSMITTERS are released


That allow the signal to carry on to other neurons
Anatomy of a neuron

• DENDRITES: Dendrites extend from the cell body and receive


chemical signals from the axon termini of other neurons

• CELL BODY: The cell body is where the nucleus is located


and is the site of protein synthesis

• AXON HILLOCK: The axon hillock is the junction of the


cell body and the axon

•AXONS: The axon is an extended part od the cell that the action
potential travels down

• AXON TERMINALS: The axon terminals are small branches


of the axon that form the synapses
The resting potential

• Cells have difference in charge between the inside and outside


of the plasma membrane

• This is known as the RESTING POTENTIAL

• The resting potential requires two types of membrane pore

1. Active transport by the Na+/K+ ATPase ion pumps

2. Non-gated passive K+ channels


The resting potential (2)

• Biomembranes can be selectively permeable to ions

• Molecules diffuse down their concentration gradient

• A build-up of charge will resist the migration down the


concentration gradient until a equilibrium is reached

• A voltage difference will then exist between the two sides of


the membrane

• This is known as the RESTING POTENTIAL


More about the resting potential

• The equilibrium potential in volts can be calculated by the


NERNST EQUATION

ENa= RT/ZF ln [Na1]/[Nar]

• Most cells have a resting potential of 60-70mV

• The bulk of the voltage difference comes from K+ ions

• The concentration gradient that is the ultimate cause of the


resting potential is maintained by active transport of ions
Na+ /K+ ATPase ion pumps

• Na+/K+ ATPase ion pumps require ATP as they work against


a concentration gradient

• Na+/K+ ATPase ion pumps transport three Na+ ions out of the
cell, and two K+ ions into the cell per ATP hydrolyzed

• As a consequence intracellular K+ concentration is high and


Na+ concentration is low compared to the extra cellular
environment
Passive K+ channels

• Passive K+ channels allow K+ ions to travel down their


gradient from the inside to the outside of the cell

• They are non-gated and selectively permeable only to K+

• Like all pores passive K+ channels are trans-membrane

• They are composed of four subunits of largely α-helices


The action potential

• Nerve cells can use the potential difference across the plasma
membrane as a method for transferring signals

• An action potential is sudden transient depolarization of the


resting potential followed by a repolarization

• Depolarization is dependent on VOLTAGE GATED Na+


CHANNELS

• Repolarization is dependent on VOLTAGE GATED K+


CHANNELS
VOLTAGE GATED Na+ CHANNELS

• In a non-nerve cell or resting nerve cell these channels are


closed

• A small depolarization will start to open the voltage gated Na+


channels

• As the depolarization increases more voltage gated Na+


channels open

• Once the depolarization reaches a peak the gate is closed for


a REFRACTORY PERIOD
VOLTAGE GATED K+ CHANNELS

• Voltage gated K+ channels are important in the repolarization of


the neuron

• Voltage gated K+ channels open while the membrane is


depolarized

• There are many types of voltage gated K+ channels varying in


voltages they are activated

• Inactivation of voltage gated K+ channels is timed to occur


a few milliseconds after the channel is opened by repolarization
The action potential is unidirectional

• The refractory period insures the action potential is


Unidirectional

• Passive spread of membrane depolarization goes in all directions

• The ‘lag’ time of the refractory period of the voltage gated ion
Channels stops the action potential going back

• This affect is enhanced by HYPERPOLARIZATION


The action potential is to slow

• The velocity of the action potential is roughly proportional


to the diameter of the axon

• Many action potential travel at 1m/s

• However in humans the axons in the spinal cord are 1 m


in length!!

• There would be to much of a time gap to permit complex


movement
Myelination increases the velocity of the impulse

• MYELINATION is a stack of specialized plasma membrane sheets


wrapped around the axon

• The myelin membrane is laid down by GLIAL cells

• The myelin sheath is a phospholipid bilayer but containing


far fewer membrane proteins
Myelination allows the action potential to jump

• Voltage gated pumps are found at nodes were the the myelin
sheath is absent

• These nodes are the only place were the axon contacts the
extra-cellular fluid

• Positive ions spread quickly down the myelinated axon as they


cannot get to the membrane

• Once they reach a node a new depolarization can begin

• The signal can travel at 10-100 times the signal speed of un-
Myelinated neurons.
Summary

• Nerve cells are long thin cells designed for transmitting signal

• The signal is a rapid depolarization of the membrane potential


that travels down the axon

• The depolarization is a consequence of the sequential opening of


Voltage gated Na+ ion channels

• Repolarization occurs as a consequence of opening of voltage


gated K+ ion channels

• Myelination of axons increases the speed of signal transmission


Release of neurotransmitters

• NEUROTRANSMITTERS are released when the action potential


reaches the axon terminals

• The action potential triggers VOLTAGE GATED Ca2+


CHANNELS

• Ca2+ diffuses into the cytosol

• Synaptic vesicles bound to membrane proteins are induced to


to fuse with the membrane

• Neurotransmitters are released into the synaptic cleft


Excess neurotransmitters is removed from the synaptic cleft

• If the neurotransmitter is not removed from the cleft the signal


will continue for to long.

• Diffusion is to slow a process to rely on to remove the


neurotransmitter

• Most types of excess neurotransmitter is removed by reuptake

• The neurotransmitter acetylcholine is unusual in that its


signaling is terminated by its hydrolysis
Types of neurotransmitter

• Neurotransmitters are generally amino acids, nucleotides


or derivatives thereof

• Neurotransmitter are generally synthesized in the cytosol


and imported into SYNAPTIC VESICLES

• Synaptic vesicles take up neurotransmitters by an active


transport process
Post-synaptic affects

• The effects on the post-synaptic neuron can be excitatory or


inhibitory

• The effects depend on the receptor and the type of gate that is
activated

• EXCITATORY RECEPTORS depolarise the membrane

• INHIBITORY RECEPTORS hyperpolarise the membrane


Changes in charge at the post synaptic neuron

• The dendrites of the post-synaptic neuron can either cause excitatory


or inhibitory signals

• Excitatory signals are a consequence of depolarization

• Inhibitory signals are a consequence of hyperpolarization

• The change in polarization is a consequence of gated-ion channels


opening
Ligand gated-channels

• The change in polarization of the dendrite is a consequence of


LIGAND GATED CHANNELS

• Ligand gated channels open when they are activated by their


ligand (neurotransmitter)

• Inhibitory receptors open Cl- channels

• Excitatory receptors open Na+ channels


Acetylcholine receptor

• The ACETYLCHOLINE RECEPTOR is a ligand


gated channel

• When open the acetylcholine receptor allows passage of


Na+ ions

• Two acetylcholine molecules are required to be bound


to the receptor to open the gate
Summing the signal

• A neuron will fire (or not fire) an action potential


down its axon dependent upon the sum of signals from
its dendrites

• The depolarizations and hyperpolrizations are summed


at the AXON HILLOCK

• The action potential will be activated if the sum of the


polarizations is greater than a THRESHOLD VALUE

• Firing of the action potential is an all or nothing event


Motor neurons trigger muscles

• Neurotransmitters can also cause acvtivate muscles

• Acetylcholine and the acetylcholine receptor are important for


muscle signaling

• A depolarisation in the muscle cell membrane leads to release of


Ca2+ which results muscle contraction
A signaling circuit

• There are three types of nerve:


1. Sensory neuron
2. Interneuron
3. Motor neuron

• These nerves can be put into networks

• The very large number of possible nerve connections leads to


complex responses
Summary

• All cells have a resting potential produced by the action of active


and passive ion channel transport

• Neurons can depolarise the resting potential by the action of


voltage gated ion channels

• This depolarisation travels down the axon

• Neurons exchange signals via neurotransmitters

• Neurons are arranged in complex ‘circuits’

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