0% found this document useful (0 votes)
21 views7 pages

Understanding Diabetes Mellitus: Symptoms & Treatment

Diabetes Mellitus is characterized by impaired glucose metabolism due to insulin deficiency or decreased sensitivity. Key clinical features include polyuria, polydipsia, and potential complications such as neuropathy, retinopathy, and cardiovascular disease. Treatment involves medications like Metformin and lifestyle modifications, with specific guidelines for managing blood glucose, hypertension, and cholesterol levels.
Copyright
© All Rights Reserved
We take content rights seriously. If you suspect this is your content, claim it here.
Available Formats
Download as DOCX, PDF, TXT or read online on Scribd
0% found this document useful (0 votes)
21 views7 pages

Understanding Diabetes Mellitus: Symptoms & Treatment

Diabetes Mellitus is characterized by impaired glucose metabolism due to insulin deficiency or decreased sensitivity. Key clinical features include polyuria, polydipsia, and potential complications such as neuropathy, retinopathy, and cardiovascular disease. Treatment involves medications like Metformin and lifestyle modifications, with specific guidelines for managing blood glucose, hypertension, and cholesterol levels.
Copyright
© All Rights Reserved
We take content rights seriously. If you suspect this is your content, claim it here.
Available Formats
Download as DOCX, PDF, TXT or read online on Scribd

Diabetes Mellitus

Definition
Impaired glucose metabolism secondary to an absolute/relative insulin deficiency or
decreased sensitivity, or both

Diagnostic criteria (SEMDSA 2012)

1
Clinical features
 Polyuria
 Polydipsia
 Polyphagia with unexplained weight loss
 Can be asymptomatic

Complications (NB!!)

 Acute  Uncontrolled hyperglycaemia, DKA, HHS (hyperosmolar hyperglycaemic


state)
 Chronic  Microvascular, macrovascular or avascular (infection)

Microvascular
 Neuropathy
o Peripheral neuropathy (symmetrical)
 Glove and stocking sensory loss
 Paraesthesia
 Motor loss
o Autonomic neuropathy
 Gastroparesis
 Neurogenic bladder
 Impotence
 Orthostatic hypotension
o Mononeuropathy
 Sudden onset peripheral or CN deficit (e.g. foot drop)
 Retinopathy
o Non-proliferative (damage)
 ‘dot and blot’ and retinal haemorrhages, cotton wool/protein
exudates
o Proliferative
 Neovascularization, retinal detachment, blindness
o Both treated with photocoagulation
 Nephropathy
o Microalbuminuria  Proteinuria +/- Nephrotic syndrome  Renal failure
o Glomerular basement membrane thickening
o Rx  BP control, ACE-I, low protein diet

2
Macrovascular
 Atherosclerosis (DM is high risk for MI, CVA, foot ulcers)
o Stroke
o CAD  MI
o Peripheral arterial disease (PAD)  NB Ulcers
Avascular
 Diabetic foot (microvascular  damage  infection  gangrene)
 Candidiasis
 Mucormycosis

Complications screening

I. Nephropathy
a. Urine Albumin/Creatinine ratio  Annually
b. If CKD  Monitor potassium & CMP
II. Retinopathy
a. Annual eye exam
III. Neuropathy
a. Monofilament, vibration sense with annual foot exam
IV. Diabetic foot risk
a. Current or previous ulcer/amputation = @ Risk
b. Absent pulses +/- claudication (PAD) = @ Risk
c. Annual foot exam
V. Cardiovascular disease (CVD)
a. Chest pain, SOB
b. HTN  Ischemic Heart Disease

3
Oral agents

Class Agent Action Contraindication


Biguanides Metformin Increase sensitivity CKD
by increasing Acidosis
peripheral glucose Alcohol abuse
uptake CCF
Sulphonylurias Glimeperide Stimulate insulin Severe liver or renal
release from B cells disease
Incretins Exenetide Stimulate B cells to Do not use with
Glucagon-like produce more DPP-4 inhibitors
peptide 1 (GLP-1)  insulin
Inject Pancreatitis or
Slows gastric history thereof
emptying
Dipeptidyl Vildagliptin Inhibit DPP-4 History of
Peptidase (DPP) 4 enzyme blocking pancreatitis
inhibitors inactivation of
incretin hormones Do not use with
GLP-1
Meglitinides Repaglinide Post-prandial
Nataglinide glucose regulator
Alpha glucosidase Acarbose Slows digestion
inhibitors

4
DM treatment (2012 guidelines)

Glucose
Type 2 DM
1. Start Metformin then titrate up to max dose 850 mg
2. ADD: Glimepiride then titrate up
3. ADD: Intermediate acting insulin @ night, start 10U then titrate up based on
morning fasting glucose methods (keep oral agents)
a. If > 20-30U needed then change to biphasic (mixed) insulin  2/3 (70%) in
morning and 1/3 (30%) at night @ 0.3U/kg
b. Start biphasic immediately rather than nocte bolus if:
i. HbA1c > 10%
c. Keep on metformin, stop SU (glimepiride)

Type 1 DM
1. Start basal bolus insulin  0.7 - 0.9U/kg
a. 40% basal at night (intermediate/long acting)
b. 60% bolus divided pre-meal (rapid acting/short acting)

5
c. Patients must have glucose meters

Hypertension
 ACE-I +/- CCB +/- Diuretic
 Enalapril, amlodipine, HCTZ

High LDL cholesterol


 LDL > 1.8 BUT < 3  Simvastatin 10mg
 LDL > 3 BUT < 3.5  20mg
 LDL > 3.4 But < 4  Atorva 40mg
 LDL > 4mg  Atorva 80mg
NB  6% rule (double dose reduced LDL with 6% only)

DM control and risk factor control (2012)

Glucose

6
FPG PPG HbA1c
Majority 4.0 - 7.0 5.0 - 10.0 < 7%
Elderly 4.0 - 7.0 < 12.0 < 7.5%
High risk

Test HbA1c 1-2x a year

LDL cholesterol
Test Lipogram 1x a year and follow-up LDL 1-2x a year
 LDL < 1.8

BP
 BP < 140/90 mmHg unless nephropathy (then lower)

You might also like