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Blood Physiology and Erythropoiesis Overview

The document provides an overview of blood physiology, detailing the composition, functions, and characteristics of red blood cells (RBCs). It discusses the processes of hematopoiesis, erythropoiesis, and the role of various nutrients and hormones in red blood cell production. Additionally, it covers types of hemoglobin, anemia classification, symptoms, diagnosis, and treatment options.

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0% found this document useful (0 votes)
3 views41 pages

Blood Physiology and Erythropoiesis Overview

The document provides an overview of blood physiology, detailing the composition, functions, and characteristics of red blood cells (RBCs). It discusses the processes of hematopoiesis, erythropoiesis, and the role of various nutrients and hormones in red blood cell production. Additionally, it covers types of hemoglobin, anemia classification, symptoms, diagnosis, and treatment options.

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aboalroos005
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© All Rights Reserved
We take content rights seriously. If you suspect this is your content, claim it here.
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LECTURE NO (-) Blood Physiology

Red Blood Cells


RBCs

Faculty Of Medicine [Link] Physiology


Blood

▪ Blood is a type of connective tissues.


➢ It consist of :
❖ Cells
❖ Intracellular substance
➢ The cells are:
▪ Red blood cells (RBCs) or erythrocytes.
▪ White blood cells (WBCs) or leukocytes.
▪ Platelets or thrombocytes.
▪ Volume of blood is about 5 litres in an average
adult male.
▪ 8% of the total body weight.
▪ Volume of plasma is about 3.5 L (5% of total
body weight)
The packed cell volume

▪ The cells in the blood can be separated by


centrifugation.
▪ The percentage of cells in blood is called the
packed cell volume (PCV) or the hematocrit.
▪ Most of these cells are RBCs.
➢ Normal values = 40-45% in males and 37-47% in
females.
▪ Low PCV indicate anemia.
▪ High PCV indicates polycythemia.
Functions of blood
➢ Transport of :
▪ Gases (e.g. oxygen & carbon dioxide).
▪ Nutrients (e.g. glucose, amino acids & free fatty
acids ).
▪ Waste products (e.g. urea & uric acid).
▪ Hormones (e.g. catecholamines, insulin & cortisol)
➢ Defense by WBCs.
➢ Homeostasis: maintains constant internal
environment.
➢ Hemostasis: prevention of blood loss &
maintenance of blood in fluid state.
Sites of hematopoiesis
❖The sites during pregnancy:
▪ First trimester: yolk sac.
▪ Second trimester: liver & spleen.
▪ Third trimester: red bone marrow.
❖The sites after delivery:
▪ Red bone marrow of all bones at first.
▪ Then the active red bone marrow becomes
gradually replaced by inactive yellow marrow.
▪ At about age of 20y the red marrow is confined to:
• The flat bones (skull, scapula, ribs, sternum,
vertebrae & iliac bone ).
• The end of long bones.
• Small bones of the hand & feet.
The red blood cells
➢ Characteristics of RBCs:-
▪ Shape: biconcave disks.
▪ Diameter: 7.5 microns.
▪ Thickness: 1-2 microns.
▪ Content: no nucleus & no organelles.
▪ Each cell contains hemoglobin (a red pigment for
transport of O2)
❖ RBCs count: 5 million/mm3 in males & 4.5
million/mm3 in females.
Functions of RBCs
▪ Transport oxygen.
▪ Transport carbon dioxide.
▪ Contains buffers to control pH.
Stages of erythropoiesis
➢ Stages in the red bone marrow:-
❖ Pluripotent stem cells (can form any type of cell).
❖ Committed stem cells (can form one type of cell
e.g. RBC).
❖ Proerythroblasts.
❖ Normoblasts.
➢ Stages in the blood:-
❖ Reticulocytes:
▪ The first form that enters the blood from the bone
marrow.
▪ Constitute about 1% of total RBCs.
▪ High reticulocyte count (more than 2%) indicates
hyperactivity in the bone marrow e.g. in hemolytic
anemia.
❖ Erythrocytes (the mature cells).
Haemopoiesis
Control of erythropoiesis
➢ Normal erythropoiesis requires:
▪ Normal bone marrow.
▪ Certain cytokines & growth factors.
▪ Certain hormones (erythropoietin).
▪ Certain nutrients (like iron, folic acid & vitamin
B12).
Erythropoietin
▪ Glycoprotein.
▪ Produced by the kidneys.
▪ Stimulated by hypoxia.
▪ Stimulates replication of stem cells & formation of
erythroblasts.
➢ Excess erythropoietin e.g. in high altitude or
chronic respiratory diseases causes polycythemia.
➢ Deficiency of erythropoietin e.g. in chronic renal
failure causes anemia (normocytic normochromic
anemia).
▪ Used for treatment of anemia of chronic renal
failure.
Nutrient for erythropoiesis
➢ Amino acids:-
▪ For formation of globin in hemoglobin.
▪ Found in animal or plant origin food.
▪ Absorbed in the small intestine.
➢ Iron:-
▪ For formation of heme in hemoglobin.
▪ Found in animal or plant origin food.
▪ Absorbed in the duodenum.
▪ Deficency causes microcytic hypochromic anemia.
➢ Vitamin B12:-
▪ For DNA synthesis (cell division).
▪ Found in food of animal origin.
▪ Absorbed in the terminal ileum.
▪ Deficiency causes macrocytic normochromic
anemia
➢ Folic acid:-
▪ For DNA synthesis (cell division).
▪ Found in food of plant origin.
▪ Absorbed in the jejunum.
▪ Deficiency causes macrocytic normochromic
anemia.
Hemoglobin
▪ The red pigment within the RBCs.
▪ Its concentration in the blood:
▪ 14-16 g/dl in adult males
▪ 13-15 g/dl in adult females.
▪ 18-20 g/dl in neonates.
Structure of Hb
▪ Consist of 4 subunits.
▪ Each subunit consist of heme + a poly peptide
chain.
▪ Each heme contains iron in reduced state (ferrous
or Fe2+).
▪ The 4 polypeptide chains are known as globin.
Function of Hb
▪ Carries 98% of oxygen in the blood.
▪ Carries some carbon dioxide.
▪ Act as a buffer.
Normal types of Hb
➢ Hb A:-
▪ Adult Hb = 98% of all Hb in adults.
▪ The 4 polypeptide chains are 2 alpha & 2 beta
chains (α2β2).
➢ Hb A2:-
▪ Adult Hb = 2.5 % of all Hb in adults.
▪ The 4 polypeptide chains are 2 alpha & 2delta
chains (α2δ2).
➢ Hb F :-
▪ Fetal Hb
▪ Replaced by Hb A 6 monthes after birth.
▪ The 4 polypeptide chains are 2 alpha & 2 gamma
chains (α2γ2).
➢ Hb A1c:-
▪ Subtype of Hb A, can bind glucose.
▪ Normally = 5% of total Hb.
▪ Its excess indicates poor control of diabetes
mellitus.
Abnormal types of Hb
➢ Hb S :-
▪ Sickle cell Hb.
▪ It is a Hb A but the amino acid number 6 in beta
chain (glutamic acid) is replaced by (valine).
▪ This abnormal Hb precipitate in cases of hypoxia,
changing the shape of RBCs to a sickle shape.
➢ Coplication of Hb S:
▪ Hemolytic anemia.
▪ Jaundice.
▪ Obstruction of small capillaries = atrophy of
organs.
Anemia
➢The definition :-
▪ State of reduction in Hb concentration below the
normal range.
➢The symptoms :-
▪ Headache.
▪ Weakness & fatiguability.
▪ Palpitation.
▪ Pallor in the skin & mucous membranes.
➢Classification of anemia :-
❖Decreased production :
▪ Bone marrow diseases e.g. tumors.
▪ Lack of nutrients e.g. iron deficiency anemia,
vitamin B12 deficiency anemia & folic acid
deficiency anemia.
▪ Lack of erythropoietin as in chronic renal failure.
❖Increased destruction :-
▪ Abnormal RBC shape as in sickle cell anemia or
congenital spherocytosis.
▪ Lack of RBC enzymes like glucose 6 phosphate
dehydrogenase.
▪ Mechanical destruction of RBCs as in malaria.
❖Blood loss :-
▪ Acute blood loss e.g. following trauma.
▪ Chronic blood loss e.g. due to chronic peptic ulcer.
➢ Diagnosis of anemia:-
▪ Low Hb.
▪ Low PCV.
➢ Treatment of anemia:-
▪ Treatment of the cause e.g. correction of the
deficient nutrient.
▪ Blood transfusion if Hb is very low.
A tissue section
showing red cells in a
small blood vessel.

The red cell must be able to


change shape and squeeze
through small capillaries.

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