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MP PSC Zoology Genetics Online Course

The document provides an extensive overview of genetics, covering key terms, inheritance patterns, chromosomal variations, and genetic disorders. It includes explanations of concepts such as incomplete dominance, crossing over, and population genetics, along with examples of genetic disorders like hemophilia and sickle-cell anemia. Additionally, it discusses the Hardy-Weinberg equilibrium and the significance of chromosome mapping in understanding gene linkage and inheritance.

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0% found this document useful (0 votes)
8 views29 pages

MP PSC Zoology Genetics Online Course

The document provides an extensive overview of genetics, covering key terms, inheritance patterns, chromosomal variations, and genetic disorders. It includes explanations of concepts such as incomplete dominance, crossing over, and population genetics, along with examples of genetic disorders like hemophilia and sickle-cell anemia. Additionally, it discusses the Hardy-Weinberg equilibrium and the significance of chromosome mapping in understanding gene linkage and inheritance.

Uploaded by

ranivarsha321983
Copyright
© All Rights Reserved
We take content rights seriously. If you suspect this is your content, claim it here.
Available Formats
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MP PSC ASSISTANT PROFESSOR

Zoology : 2024
M.S.
BIOLOGY Genetics Online
classes
available

Lecture-1
Terms and terminology
Mahendra Sir
M.S. BIOLOGY App and online classes
(online classes and test series for MP PSC AP Zoology)
For online class- 8318917792
Terms and terminology used in genetics
▪ Clone • Genotype
▪ Variation • Phenotype
▪ Individual • Sister and non sister chromatids
▪ Offspring • Homologous and heterologous
chromosome
▪ Gamets
▪ Gene
▪ Alleles
▪ Monohybrid cross
▪ Dihybrid cross
▪ Dominant and recessive character
▪ Homozygous and heterozygous
▪ F1 generation and self cross Note: Try to define itself and prepare a short notes on
these terms with suitable representation.
Incomplete dominance – Codominance-
➢ Both parental gene show their effect
➢Fi show intermediate character of both
simultaneously in fi generation
parents ➢ Phenotype of hybrid resemble both the
➢Phenotypic and genotypic ratio are same parents
➢New phenotype is formed ➢ New phenotype is not formed
➢Ex.- red white colour flower in snapdragon ➢ Ex.- AB blood group of human
Variation in chromosome number –
➢ Euploidy- Complete set of chromosome of an organism
➢ Aneuploidy- abnormalities in chromosome number either in complete set or single
➢Generation of aneuploidy- due to non disjunction in meiosis I or II
➢Type of aneuploidy-
➢Nullisomy- (2N-2)
➢Monosomy- (2N-1)
➢Trisomy- (2N+1)
➢Tetrasomy- (2N+2) non disjunction
➢Patau syndrome (trisomy : chromosome 13)
Autosome
➢Edward syndrome (trisomy : chromosome 18) chromosome
➢Down syndrome (trisomy : chromosome 21)
➢Turner syndrome ( monosomy in X chromosome: XO genotype) in female.
➢Klinefelter syndrome ( trisomy/ tetrasomy in X chromosome : XXY/XXYY)
in male.
➢Variation in chromosomal structure-
➢Types-
➢Deletion- change in amount of DNA (decreases)
➢Duplication – change in amount of DNA (increases)
➢Inversion- change in orientation of segment of DNA at 180* angle
➢Translocation- change in location of segment of DNA
Non mendelian inheritance:- ➢ Can be separated by CsCl density gradient
centrifugation
➢ Size of genome-
➢ In animals - < 20kb
➢ In yeast – 80kb to 80000bp
➢ In plants- 100000 to 2 million bp

➢Origin of mitochondria and chloroplast-


➢Organisation of extranuclear genome-
➢Mitochondrial genome –
➢Circular double-stranded supercoiled
➢In some protozoans and fungi- linear genome
➢No histone proteins are associated with mt DNA
➢GC content differs from nuclear DNA
➢Genome replication-
➢Semiconservative, specific DNA polymerase
➢Mt DNA replication process occurs throughout cell cycle
➢Two strands of mt DNA have different density, they called as H and L
strand
Model for mt DNA replication-: ( D loop model)
➢Synthesis of H strand started at ori to form D loop
➢As new H strand extended about half , initiation of L strand at another ori
➢Both strands are completed by continous replication
➢Transcription of mt DNA -:
➢Mt DNA contains genes of –t RNA , rRNA, cytochrome oxidase, NADH
dehydrogenase and ATPase
➢Other components found in mitochondria are encoded by nuclear genome.
➢Mitochondrial protein coding genes are found on both strand
➢Mt DNA have no 5* cap
➢Poly A tail is added to 3* end of mRNA
➢CCA is added to 3* end of tRNA
Crossing over – given by Morgan 1910
➢Reciprocal exchange of segments between non sister chromatids of homologous
chromosome.
➢Non sister chromatids called cross over or recombinant
➢Unchanged combinations are called non cross over or parental type
➢Crossing over occur in pachytene stage of meiosis I
➢Synopsis , divalent and synaptonemal complex formation –in zygotene
➢Chiasmata formation – diplotene
Mechanism of crossing over
Coupling and repulsion theory ––( Bateson and Punnet (1906)
Chiasmata type theory- Morgan
Modern theory – breakage and reunion

Coupling and repulsion theory Chiasmata type theory Modern theory


➢Single crossing over-
➢When non sister chromatids of a
homologous pair exchange segment only
at one place.

➢Double crossing over -


➢Exchange of segments between two non
sister chromatids at two place.
➢Reciprocal crossing over –
➢Crossing over occurs at two places between the same non sister chromosome.
➢It produces two parental type and two double cross over type.
➢Complementary crossing over – in this crossing over three or all four chromatids
are involved in exchange of segment.
➢However each crossing over occurs between non sister chromatids.
➢In double crossing over , a parental type may be absent and all are cross over
➢May be one parental type, two single cross over and one double cross over.
➢Multiple crossing over – in this 3,4 or more points of exchange occurs amongst
the non sister chromatids of the same homologous chromosome.
Factors affecting crossing over-
➢Distance
➢Temperature
➢Chemicals
➢Age
➢X- rays
➢Location

➢Importance of crossing over –


• The exchange of chromosome material results in variation in the offspring.
• It plays a crucial function in the evolution process.
• The creation of genetic maps is aided by crossing over frequency.
• Crossing over is evidence of a chromosome's linear organization of connected
genes.
➢ Chromosomal theory of linkage- (Morgan and Castle 1911)
➢ Main features-
➢ Linked genes are stay together during transmission from one to another generation.
➢ Linked genes occur on the same chromosome.
➢ Linked genes are arranged in linear fashion over the chromosome.
➢ Each linked gene occupy specific place over the chromosome.
➢ Genes tends to maintain original parental combination.
➢ Crossing over occurs due to weakening of linkage
➢ Types of linkage –
➢ Complete linkage and incomplete linkage
➢ Complete linkage – linkage which is not altered and inherited as such without any crossing
over
➢ Complete linkage is rare but has been reported in Drosophila
➢ Phenotypic ratio of test cross is 1:1
➢ Incomplete linkage –phenomenon of occasional crossing over between two homologous
chromosome .
➢ One ore more alleles present is linkage group are replaced by other .
➢ Produces both parental and recombinant type
➢ % of parental type is more than 50% while recombinant is less than 50%
➢ Phenotypic ratio of test cross is 9:1:1:8
Test cross Ratio
Law of independent assortment 1:1:1:1
Complete linkage 1:1
Incomplete linkage 9:1:1:8

Arrangements of linked gene


➢ Cis and trans arrangement

A a A B

B b b a

Cis Trans
Genetical disorder
• Most common and prevalent genetic disorders are Haemophilia, Cystic
fibrosis, Sickle cell anaemia, Colour blindness, Phenylketonuria, Thalassemia,
etc.
• Colour Blindness:
• It is a sex-linked recessive disorder
• failure to discriminate between red and green colour
• Defect in either red or green cone of eye
Haemophilia:
• It is X linked recessive disease
• a single protein that is a part of the cascade of
proteins involved in the clotting of blood is
affected
• an affected individual a simple cut will result
in non-stop bleeding.
Sickle-cell anaemia
• This is an autosome linked recessive trait
• The disease is controlled by a single pair of allele, HbA and HbS
• Out of the three possible genotypes only homozygous individuals for HbS
(HbSHbS ) show the diseased phenotype
• The defect is caused by the substitution of Glutamic acid (Glu) by Valine (Val) at
the sixth position of the beta globin chain of the haemoglobin molecule
Phenylketonuria:
• It is an autosomal recessive trait
• The affected individual lacks an enzyme that converts the amino acid
phenylalanine into tyrosine
• As a result of this phenylalanine is accumulated and converted into
phenylpyruvic acid and other derivatives
• Accumulation of these in brain results in mental retardation
➢Chromosomal theory of inheritance : (Sutton and Boveri (1902)
➢Key points –
❖Mendelian factors are located on the chromosome which show
segregation and independent assortment at the time of transmission
from one generation to another.
❖Supporting points –
❖Gametes are the bridge between one generation to next.
❖Equal contribution of both gametes in genetic of offspring.
❖Out of sperm only nucleus passes into egg for fertilization.
❖Nucleus contain chromatin which organise to from chromosome.
❖There are two chromosome of each type in an individual.
❖Gamete contain only one chromosome out of two.
Population genetics: study of gene frequencies in population
• Population: group of individuals present in a geographical are which
share common gene pool
• Gene frequency : percentage of an allele in relation to the total
alleles of a gene present in an interbreeding population
• Gene pool: all the gene and their alleles found in an interbreeding
population
• Genetic equilibrium : criteria
• Population is stable throughout the year
• No immigration or emigration
• Size of the population is large
• Mutations are negligible
• Sex ratio is unity and mating is random
Hardy –Weinberg equilibrium :
The allelic frequencies in a non evolving populations are stable and remain
constant from generation to generation
p+q=1 or (p+q)2 = p 2 + q 2 + 2pq=1

Five basic process which change hardy –Weinberg equilibrium and bring about
variation at the genetic level
• Mutation
• Recombination
• Genetic drift
• Gene flow
• Natural selection
➢ Epistasis: Interaction between two or more gene to control a single phenotype
➢ Interaction involve one gene masking or modified the phenotypic expression of another gene
➢ No new type of phenotypes are produced by this interaction
➢ Epistatic, hypostatic
➢Cause of epistasis:
➢ By presence of homozygous recessive of one gene pair
➢ By presence of one dominant allele in a gene pair
➢Dominant epistasis :
➢ 12:3:1
➢Recessive epistasis/ supplementary gene:
➢ 9:3:4
➢Duplicate recessive interaction:
➢ 9:7
➢Duplicate dominant interaction:
➢ 15:1
chromosome mapping-
➢ A linear graphic representation of the sequence of gene present over a chromosome and the
relative distance between them.

➢ Basis for chromosome mapping-


➢ Crossing over or recombination is directly proportional to the physical distance.
➢ Genes present farther apart on a chromosome undergo crossing over more frequently.
➢ F= total no of recombinant /total no of progeny
➢ 1 cM (Centi Morgan)= 1% recombination or 0.01 recombination frequency.
➢ 1dM (deci Morgan)= 10% recombination or 0.1 recombination frequency.
➢ 1 M (Morgan)= 100% recombination or 1 recombination frequency.
➢ Importance of linkage map-
➢ Provide information about location of different gene on various chromosome.
➢ Chances of crossing over between two genes can be known.
➢ Drawbacks of chromosome map-
➢ Chromosome maps are not accurate map.
➢ One crossing over interferes and reduces the frequency of nearby crossing over .
➢Sex linkage- linked group of gene found along with sex controlling
genes as present on sex chromosome .
➢Sex link inheritance- inheritance of certain gene together along with
sex determining genes.
➢Sex linked gene-the additional gene present on sex chromosome along
with sex determining gene are called sex linked gene.
➢Autosome – determine morphological traits
➢Allosome/ sex chromosome- determine the sex of individual.
➢Homomorphic – two sex chromosome having similar shape-
➢Heteromorphic – morphologically different sex chromosome
➢Androsome –sex chromosome determine male sex
➢Gynosome –sex chromosome determine female sex
Characteristics feature of sex linked inheritance-
➢Sex linked inheritance is criss cross inheritance.
➢Diagynic inheritance-male passes sex linked traits to grandson through his
daughter
➢Female passes its X chromosome equally into son and daughter.
➢Most of the sex linked traits are recessive.
➢Female are less affected with sex linked disorder- carrier
➢Males are more affected –hemiygous

➢Importance-

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