Growth and Development Principles Explained
Growth and Development Principles Explained
DEVELOPMENT
FACTORS INFLUENCING GROWTH AND
TERMS DEVELOPMENT
GROWTH A. GENETIC INHERITANCE
➢ Increase in physical size or structure that is 1. GENDER
measurable.
• Girls are born lighter.
➢ Quantitative change in the body.
DEVELOPMENT • Girls begin puberty growth spurt 6 months to 1
yr. earlier than boys.
✓ Increase in skill and capacity to function
occurring in an orderly fashion. • Boys tend to be taller & heavier than girls by
end of puberty (14 – 16 y.o.).
✓ Child’s ability to perform specific tasks.
2. HEALTH
✓ Qualitative change.
MATURATION • Disorders included in the NB screening:
1. Congenital hypothyroidism
• Increase in competence or ability to function at 2. Congenital Adrenal Hyperplasia
a higher level. (CAH)
• The process by which the traits carried by the 3. Galactosemia (GAL)
child through his genes begin to unfold and are 4. Phenylketonuria (PKU)
realized. 5. Glucose-6-Phosphate
COGNITIVE DEVELOPMENT Dehydrogenase Deficiency (G6PD
➢ Acquiring concepts of time and space, abstract Def)
thought. • Hemophilia
➢ Ability to learn or understand from experience, • Type 1 Diabetes Mellitus
to acquire and retain knowledge, to respond to 3. INTELLIGENCE
new situations, to solve problems.
• Intellectual level affects vocabulary and ability
➢ Measured by: to encode & decode messages.
o Intelligence tests
o Observing child’s ability to function • Influences the number of languages a child
speaks, reading ability.
effectively in his/her environment.
• Depth of explanation that a child is capable of
PRINCIPLES OF GROWTH AND DEVELOPMENT understanding.
1. G & D are continuous processes from Examples:
conception until death. o Mental retardation
2. G & D proceed in an orderly sequence. o Autism
3. G & D are continuous processes from o ADHD (attention deficit
conception until death. hyperactivity disorder)
4. G & D proceed in an orderly sequence. 4. TEMPERAMENT
5. Different children pass through the • Usual reaction pattern of an individual, or an
predictable stages at different rates. individual’s characteristic manner of thinking,
6. All body systems do not develop at the behaving or reacting to stimuli in the
same rate. environment.
7. Development is cephalocaudal.
8. Development proceeds from proximal to
• Children are not alike:
distal body parts. o Others adapt quickly
9. Development proceeds from gross to o Some adapt slowly
refined skills. o Some react intensely
10. There is an optimum time for initiation of o Some react passively
experiences or learning.
11. Neonatal reflexes must be lost before REACTION PATTERNS
development can proceed. a) ACTIVITY LEVEL
12. A great deal of skill and behavior is learned • Some babies are constantly on the go.
by practice.
• Others stay where they are placed.
GROWTH PATTERN
b) RHYTHMICITY
HEAD CONTROL
• Some wake up at the same time each morning.
• Newborn to 6 months
SITTING UP • Hungry at regular intervals.
• 2 months to 8 months • Nap same time each day.
FINE MOTOR DEVELOPMENT • BM same time each day.
c) APPROACH 3. Parents must focus on preparing the child for
• Child’s response on initial contact with a new new activities.
stimulus. 4. Bring these characteristics to the parent’s
attention, because understanding the child is
• Some smile and “talk” to strangers.
the beginning of accepting & respecting the
• Others cry at the sight of strangers. child as an individual.
d) ADAPTABILITY B. ENVIRONMENT
• Ability to change one’s reaction to stimuli over 1. SOCIOECONOMIC LEVEL
time. ➢ Low socioeconomic status.
e) INTENSITY OF REACTION 2. PARENT-CHILD RELATIONSHIP
• Some children cry loudly. ✓ Children who are loved to thrive better
than those who are not.
• Some have mild or low-intensity reactions to
✓ Loss of love from a primary caregiver can
stress. interfere with a child’s desire to eat,
f) DISTRACTIBILITY improve, and advance.
• Children who are easily distracted or can easily 3. ORDINAL POSITION IN THE FAMILY
shift their attention to a new situation can be ➢ Only child or oldest child generally excels in
easily managed. language development.
• Others cannot be distracted. ➢ Youngest child may develop language more
• Parents may describe them as stubborn, willful, slowly.
or unwilling to compromise. 4. HEALTH
g) ATTENTION SPAN & PERSISTENCE • Children with physical disabilities may be
• Ability to remain interested in a project or limited in their ability to play.
activity. THEORIES OF DEVELOPMENT
A. SIGMUND FREUD
• Some play with their toy for 1 hr.
• Some play no more than 1 – 2 min. with each
• Austrian neurologist.
toy. • Founder of psychoanalysis.
h) THRESHOLD OF RESPONSE • Offered the first real theory of personality
• Intensity level of stimulation necessary to evoke development.
a reaction. • Describes child development as a series of
i) MOOD QUALITY psychosexual stages in which a child’s sexual
• The child who is always happy and laughing gratification becomes focused on a particular
can be said to have a (+) mood quality. body part.
WELLNESS PROMOTION
A. General
CHAPTER 33: NURSING CARE OF A FAMILY WITH AN
• Encourage well-childcare, screenings, ADOLESCENT
immunizations, and safety.
B. Nutrition ADOLESCENCE
• Promote healthy eating, limit junk food. • Transition period between childhood and
• Educate on the food pyramid and differentiate adulthood, characterized by rapid physical,
nutritious vs. junk foods. cognitive, social, and emotional changes.
C. Sleep • A time when boys prepare for manhood and
• Establish a consistent bedtime; allow flexibility girls for womanhood.
on non-school nights.
D. Growth & Development Subphases of Adolescence
• Encourage skill development in various 1. Early adolescence (13 – 14 years)
activities, promote safety for latchkey children. 2. Middle adolescence (15 – 16 years)
E. Family 3. Late adolescence (17 – 20 years)
• Foster open communication, responsibility,
decision-making, and individuality. Puberty
F. Health • A maturational and hormonal process marking
• Promote self-care and hygiene. sexual maturity and the development of
G. Anticipatory Guidance secondary sex characteristics.
• Teach about puberty, substance abuse, and • Normal Parameters:
sex education. o PR = 70 bpm
o RR = 20 bpm
o BP = 120/70 mmHg • Value system: Establishing personal values,
identity, and making career decisions.
Stages of Puberty
1. Prepubescence (2 years before puberty):
• Emancipation from parents: Becoming
independent, though it may be difficult for both
Preliminary physical changes.
adolescents and parents.
2. Puberty: Sexual maturity is reached, and the
first menstrual flow occurs in girls.
Socialization:
3. Postpubescence (1–2 years after puberty):
Skeletal growth completes, reproductive • Varies by age, from impulsive and emotional
functions are well established. behavior at 13 years to thoughtful interactions
at 17 years.
Physical Growth
Cognitive Development:
• Girls:
o Typically, 1–2 inches taller than boys • Formal operational thought: Ability to think
at the start of adolescence. abstractly, reason scientifically, and plan.
o Growth stops within 3 years of • Brain development: Pre-frontal cortex still
menarche (around 16–17 years). maturing, leading to impulsive behavior and
o Weight gain: 15–55 lbs. emotional swings.
Discipline:
• Boys:
o Grow 4–12 inches during • Important to set firm but reasonable limits and
adolescence. maintain a supportive, non-interfering family
o Weight gain: 15–65 lbs. environment.
Substance Abuse:
• Adolescents may use substances like tobacco,
alcohol, marijuana, or illicit drugs to cope with
stress or fit in with peers.
• Signs of abuse: Mood swings, declining
physical appearance, academic problems, and
changes in peer groups.
Runaways:
• Adolescents may run away due to family
conflict, loneliness, or other personal struggles.
• Interventions: Support from family and
community resources to resolve underlying
issues.
CHAPTER 08: NURSING CARE OF A FAMILY HAVING 1. Female Infertility Tests
DIFFICULTY CONCEIVING A CHILD • Ovulation Testing: Blood test for hormone
levels
• Hysterosalpingography (HSG): X-ray to
STRUCTURE OF AN OVUM check for fallopian tube blockage
A. Nucleus
• Pelvic Ultrasound: Detects ovarian and
• The heart of the egg cell. uterine abnormalities
• It contains most of the genetic material in the 2. Male Infertility Tests
form of chromosomes. • Semen Analysis: Examines sperm count,
• This is where the genes are situated. movement, and shape
• An egg, like a sperm, contains half the number • Testicular Biopsy: If sperm production is in
of chromosomes as a normal cell, i.e., 23 question
chromosomes each. • Hormone Testing: Assesses testosterone and
B. Cytoplasm other hormones
• A gel-like substance that holds all the cell’s
other internal structures, called organelles. TREATMENT OPTIONS FOR INFERTILITY
• It is in the cytoplasm that all the cell’s activities 1. Medications
take place to keep it alive and functioning • Clomiphene Citrate: Stimulates ovulation
properly. • Metformin: Used for PCOS
• Among the more important organelles are • Gonadotropins: Stimulates the ovaries to
structures called mitochondria, which supply produce eggs
most of the energy for the cell. 2. Surgical Procedures
C. Zona Pellucida
• Laparoscopy: Removes blockages, fibroids,
• The zona pellucida (or egg wall) is an outer or endometriosis
membrane of the egg.
• Varicocele Repair: Surgery for enlarged veins
• This structure helps the sperm to enter the egg in the testicles affecting sperm production
through its hard outer layers. 3. Assisted Reproductive Technologies (ART)
D. Corona Radiata
• Intrauterine Insemination (IUI): Sperm is
• It surrounds an egg and consists of follicle placed directly into the uterus
cells that provide necessary proteins for the
egg’s growth. • In Vitro Fertilization (IVF): Eggs are fertilized
outside the body and implanted in the uterus
FACTORS AFFECTING FERTILITY • Intracytoplasmic Sperm Injection (ICSI): A
1. Female Factors single sperm is injected into an egg
a. Ovulation Disorders
• Hormonal imbalances ROLE OF THE NURSE IN INFERTILITY CARE
• Polycystic ovarian syndrome (PCOS) • Provide emotional support and counseling
• Premature ovarian failure • Educate couples about fertility awareness and
b. Tubal Factors lifestyle changes
• Blockage or damage to the fallopian tubes due • Assist in fertility treatments and procedures
to: • Advocate for patient mental well-being
o Pelvic Inflammatory Disease (PID)
o Endometriosis
o Previous surgeries CHAPTER 18: NURSING CARE OF A FAMILY WITH A
NEWBORN
D. Implementation:
• The inability of the mitral valve (MV) to push
blood forward causes:
• For pregnant women with chronic illness: o Back pressure on the pulmonary
o Focus on teaching her new or circulation causing it to be distended.
additional measures to maintain o Systemic BP decreases.
health. o Pulmonary hypertension – pressure
• For women who developed a new illness: in pulmonary vein reaches 25 mmHg,
o Provide an opportunity to talk about fluid passes from pulmonary capillary
the event after her initial care is membranes into interstitial spaces
complete to identify concerns. and alveoli (pulmonary edema).
o Pulmonary edema.
E. Outcome Evaluation: o Pulmonary capillaries rupture.
• If the expected outcome is not met, High-Risk Outcomes:
reassessment, analysis, and planning need to • Women with pulmonary hypertension are at
be done. extremely high risk for:
• Make evaluation ongoing to ensure whether o Spontaneous miscarriage.
interventions are successful. o Preterm labor.
• Examples of outcomes: o Maternal death.
o Client states she rests for 2 hrs • If complications result in impaired blood flow to
morning and afternoon; dependent the uterus, the following events can occur:
edema remains at 1+ or less at next o Poor placental perfusion.
prenatal visit. o Intrauterine (IU) growth restriction.
o Family members state they are all o Fetal mortality.
participating in an exercise program
since mother developed gestational
• Serial Ultrasounds and Non-Stress Tests:
diabetes. o Woman needs serial ultrasounds and
non-stress tests done after 30-32
o Client reports no burning on urination
weeks to monitor fetal and maternal
or flank pain at next prenatal visit.
health.
o Client states she understands the
importance of taking daily thyroid
Signs and Symptoms of Left-Sided Heart Failure:
medicine for the total length of
1. Increased respiratory rate (RR).
pregnancy.
2. Increased fatigue, weakness, and dizziness.
3. Systemic decrease in blood pressure (BP).
Identifying a High-Risk Pregnancy
Cardiovascular Disorders:
Medical Management for Left-Sided Heart Failure:
• The cardiovascular disorders that most 1. If Mitral Valve (MV) Stenosis is Present:
commonly cause difficulty during o Thrombus formation can occur due to
pregnancy are: non-circulating blood.
o Valve damage concerns caused by o Anticoagulants:
rheumatic fever or Kawasaki disease. ▪ Heparin (does not cross
o Congenital anomalies such as atrial the placenta) for early
septal defect or uncorrected pregnancy.
coarctation of the aorta.
High-Risk Pregnancy: Cardiovascular System
▪ Warfarin (Coumadin) can • Cardiomegaly (heart enlargement).
be used after 12 weeks but Therapy:
return to heparin during the • Sharply reduce physical activity.
last month of pregnancy.
o Balloon Valve Angioplasty (for • Diuretic therapy.
treatment). • Arrhythmia agents, and digitalis therapy to
2. If Coarctation of the Aorta is Present: maintain heart function.
o Aortic dissection may occur. • Low-molecular-weight heparin may be
o Prescribed medications: administered.
▪ Antihypertensive • Immunosuppressive therapy (e.g.,
medications. corticosteroids).
▪ Diuretics to reduce blood • Postpartum Considerations:
volume. o If cardiomegaly persists, future
▪ Beta-blockers to improve pregnancies may be discouraged as
ventricular filling. the condition may recur or worsen.
o Oral contraceptives are
Right-Sided Heart Failure contraindicated due to the risk of
thromboembolism.
Common Causes:
• Pulmonary valve stenosis. Classification of Heart Disease in Pregnancy:
• Atrial & ventricular septal defects. • Heart disease in pregnancy is classified into
• Eisenmenger syndrome (R to L atrial or four categories based on criteria established by
ventricular septal defect with pulmonary the New York Heart Association (NYHA):
stenosis).
Pathophysiology: Class Description
• Occurs when output from the right ventricle Class
Uncompromised: Ordinary physical activity
(RV) is less than the blood volume received by causes no discomfort. No symptoms of cardiac
1
the right atrium (RA) from the vena cava. insufficiency or anginal pain.
• Back-pressure results in: Class
Slightly compromised: Ordinary physical activity
o Congestion of systemic venous 2
causes excessive fatigue, palpitation, dyspnea,
circulation. or anginal pain.
o Decreased cardiac output to the Markedly compromised: Less than ordinary
lungs. Class
activity causes excessive fatigue, palpitation,
3
o Decreased BP in the aorta. dyspnea, or anginal pain.
o High pressure in the vena cava. Severely compromised: Woman is unable to
o Jugular venous distention. Class carry out any physical activity without discomfort.
o Increased portal circulation (liver & 4 Symptoms of cardiac insufficiency or anginal
spleen distention). pain present even at rest.
o Ascites (fluid accumulation in the
peritoneal cavity). Assessments for Cardiac Disease in Pregnancy:
1. Thorough Health History:
Medical Management for Right-Sided Heart Failure: o Document pre-pregnancy cardiac
1. Eisenmenger Syndrome (uncorrected status.
anomaly): 2. Exercise Performance:
o Women with uncorrected anomalies o Assess the woman’s level of exercise
may be advised against pregnancy performance.
due to the high risks associated with 3. Symptoms Assessment:
this condition.
o Ask if the woman experiences a
cough or edema:
Peripartum Heart Disease:
▪ Coughing could be a sign
• Peripartum Cardiomyopathy can develop of pulmonary edema.
during pregnancy in women with no prior
history of heart disease. The cause is unknown Distinguishing Edema:
but may be related to pregnancy stress on the
circulatory system. • Innocent Edema vs. Heart Failure:
o Mortality rate = 50%. o The usual innocent edema of
Signs & Symptoms: pregnancy must be distinguished
from edema caused by heart failure,
• Shortness of breath. which is a more serious condition.
• Chest pain. Baseline Vital Signs:
• Non-dependent edema. • Assessment Considerations:
o Irregular pulse, rapid or difficult • Deficient Knowledge:
respirations, and chest pain on o Women may have insufficient
exertion may be present in women knowledge on how to reduce the
with heart disease. effects of maternal cardiovascular
o Baseline Measurements: disease on pregnancy and fetal
▪ Record blood pressure, health.
pulse rate, and respiratory Outcome Evaluation:
rate in sitting or lying • Goals:
position at the first prenatal o Patient identifies danger signs, such
visit and continue as angina pain, and knows the
throughout pregnancy. appropriate actions to take.
▪ Always obtain these in the o Maternal blood pressure maintained
same position at future above 100/60 mmHg.
visits for accurate o Fetal heart rate maintained at 110-
comparison. 160 beats per minute.
o Additional Assessments:
• Realistic Expectations:
▪ Nail bed filling should be o Not all women with heart disease will
<5 seconds.
be able to complete pregnancy
▪ Assess for jugular venous successfully.
distention.
o Positive actions, like increased rest
periods, can help reduce
Liver Size:
complications.
• Right-Sided Heart Failure:
o If the heart disease involves right- Related Interventions:
sided heart failure, assess liver size Promote Rest:
at prenatal visits for signs of • Rest Periods:
congestion.
o Women with cardiac disease need
two rest periods a day and a full
ECG and Echocardiogram:
night’s sleep to avoid exertion.
• Cardiac Monitoring: o Women should stop exercising
o ECG (electrocardiogram) or before cardiac output becomes
echocardiogram may be done insufficient.
periodically. o Some women may need to
o Both procedures are safe for the discontinue employment early in
fetus: pregnancy to reduce strain on the
▪ ECG measures cardiac heart.
electrical discharge and o Activity restrictions may vary:
poses no harm to the fetus. "Normal housework" may involve
▪ Echocardiography uses light tasks like dusting for some, but
ultrasound, which is also more strenuous work for others.
safe for the fetus. Promote Healthy Nutrition:
• Weight Management:
Fetal Assessment: o Ensure that the woman doesn’t gain
• Maternal Blood Pressure and Fetal Health: excessive weight to prevent
o When maternal blood pressure overburdening the heart and
becomes insufficient, fetal health circulatory system.
may be compromised, leading to o Confirm the woman is taking prenatal
poor placental perfusion. vitamins and maintaining nutritional
o Potential Fetal Risks: balance.
▪ Preterm labor may occur. • Anemia Prevention:
▪ Late decelerations on fetal o Sodium-restricted diet may be
heart monitoring may recommended but should not be
indicate poor response to excessively restrictive.
labor.
▪ Cesarean birth may be Educate Regarding Medication:
necessary, increasing risks • Cardiac Medication Adjustments:
for both the mother and o Women taking cardiac medications
fetus. like digoxin may need to increase
their maintenance dose due to the
Nursing Diagnosis and Related Interventions: expanded blood volume during
Nursing Diagnosis: pregnancy.
o Digoxin can also be used to slow Postpartum Care and Interventions
fetal heart rate if fetal tachycardia A. Assess for Heart Failure:
occurs. • The postpartum period is a critical time for
• Antihypertensive and Arrhythmia Agents: women with heart disease.
o Adenosine, beta-blockers, and • After birth, there is a rapid increase in
calcium channel blockers may be pressure within 5 minutes, which requires the
used to manage hypertension. heart to make a major adjustment quickly.
o Nitroglycerin, often prescribed for
angina, is considered safe during B. Decreased Activity:
pregnancy (Category C drug). • A program of decreased activity may be
• Penicillin Prophylaxis: necessary during the postpartum period to
o If the woman had rheumatic fever prevent additional strain on the heart.
and was on penicillin prophylaxis,
she should continue this during C. Medication Therapy:
pregnancy, as penicillin is not • Continue anticoagulant therapy and digoxin
teratogenic (Category B drug). until circulation stabilizes.
o Pre-birth Antibiotics: Providers may
prescribe ampicillin, amoxicillin D. Antiembolic Measures:
(Amoxil), or clindamycin (Cleocin) • Antiembolic stockings or intermittent
near birth to prevent infection at the pneumatic compression (IPC) boots may be
placental site. prescribed to increase venous return from the
legs and reduce the risk of clot formation.
Educate Regarding Infection Prevention:
• Infection Risks: E. Prophylactic Antibiotics:
o Systemic infections can increase • Prophylactic antibiotics should be
cardiac output and metabolism, administered immediately after birth to prevent
putting stress on the heart. subacute bacterial endocarditis due to
o Advise women with heart disease to potential microorganism introduction through
avoid exposure to infections and the placental site.
report any symptoms of upper
respiratory or urinary tract infections F. Stool Softener:
(UTIs).
• A stool softener may be prescribed to avoid
o Early Antibiotic Therapy can be straining during bowel movements, which
started to prevent complications. could increase cardiovascular stress.
o Monthly Screening for Bacteriuria
should be done with a clean-catch G. Use of Oxytocin (Pitocin):
urine test at prenatal visits to detect
UTIs early. • Oxytocin should be used with caution as it can
increase blood pressure, which places
Nursing Interventions During Labor and Birth: additional strain on the heart.
1. Frequent Monitoring:
H. Kegel Exercises:
o Assess the woman's blood pressure,
pulse, and respirations frequently. • Kegel exercises can be safely performed for
2. Fetal Monitoring: perineal strengthening immediately
o Continuously monitor fetal heart rate postpartum.
to assess fetal well-being.
3. Monitor Uterine Contractions:
o Monitor contractions to ensure proper I. Postpartum Exercises:
placental filling and to prevent • Postpartum exercises aimed at improving
circulatory system failure. abdominal tone can begin once approved by
4. Side-Lying Position: the woman's primary care provider.
o Advise a side-lying position to reduce
the risk of supine hypotension J. Assess Baby:
syndrome. • Acrocyanosis (peripheral cyanosis) in
5. Oxygen Administration: newborns is normal. The mother should not
o Oxygen may be administered during interpret this as cardiac inadequacy in her
labor due to increased oxygen baby.
demand.
6. Hemodynamic Monitoring: Hematologic Disorders: Anemia and Diabetes in
o Continuous monitoring may be Pregnancy
required using a Swan-Ganz catheter
to assess heart function during labor. TRUE ANEMIA
• Hemoglobin Levels: • Type 1 Diabetes: An autoimmune disorder
o <11 g/dL (hematocrit <33%) in 1st or where the pancreas fails to produce adequate
3rd trimester insulin.
o <10.5 g/dL (hematocrit <32%) in 2nd • Type 2 Diabetes: Gradual loss of insulin
trimester production, but some ability to produce insulin
remains.
Iron-Deficiency Anemia Clinical Manifestations in Pregnancy:
Causes: • For women with Type 1 or Type 2 diabetes
1. Diet low in iron who had good glucose control before
2. Heavy menstrual periods pregnancy, pregnancy causes changes in
3. Unwise weight-reduction programs glucose metabolism, making control more
4. Pregnant within <2 years before current difficult.
pregnancy
5. Low socio-economic levels with poor iron-rich • Insulin resistance increases as pregnancy
diets progresses due to hormones like human
Associated Risks: placental lactogen, cortisol, estrogen,
progesterone, and catecholamines.
• Low birth weight & preterm birth
• Women with pre-existing kidney disease
• Extreme fatigue & poor exercise tolerance (proteinuria, decreased creatinine clearance,
Blood Characteristics: hypertension) have a higher risk of pregnancy-
• Microcytic (small red blood cells) related hypertension.
• Hypochromic (less hemoglobin than average
red blood cells) Complications Associated with Diabetes in
Medical Management: Pregnancy:
1. Prophylaxis: 27 mg/day iron supplement 1. Hydramnios:
during pregnancy o High glucose levels cause extra fluid
2. Diet: High in iron and vitamins (green leafy to shift, enlarging amniotic fluid.
vegetables, meat, legumes) 2. Macrosomia:
3. Treatment: 120-200 mg elemental iron (ferrous o Large infants (>10 lb), leading to
sulfate or ferrous gluconate) potential birth issues (CPD).
Reminders: 3. Congenital Anomalies:
• Iron is absorbed best in an acid medium, so o Increased risk of caudal regression
take with orange juice or vitamin C. syndrome, spontaneous
• Side effects include constipation or gastric miscarriage, or stillbirth if diabetes
is poorly controlled.
irritation, alleviated by increasing roughage or
Clinical Manifestations in Newborns:
taking with food.
• Ferrous sulfate can cause black stools. • Hypoglycemia
• If severe, intravenous iron may be prescribed. • Respiratory distress syndrome
• Hypocalcemia
Folic Acid-Deficiency Anemia • Hyperbilirubinemia
Importance of Folic Acid (B9):
• Normal RBC formation in the mother Monitoring a Woman with Diabetes During
• Prevention of neural tube defects in the fetus Pregnancy:
1. Pre-pregnancy:
Most Common in:
1. Multiple pregnancies o Meet with primary healthcare
2. Women with secondary hemolytic illness provider to ensure optimal glucose
3. Women taking hydantoin (anticonvulsant) control before conception.
4. Women with gastric bypass surgery or B12 o Early pregnancy glucose control is
deficiency vital to minimize congenital
Megaloblastic Anemia: anomalies.
2. Tests:
• Enlarged RBC that don't function properly. o Glycosylated Hemoglobin
Medical Management:
(HbA1c): Reflects average glucose
1. Pre-pregnancy: Supplement with 400 µg folic
over the past 4-6 weeks. A level of
acid daily
6% is considered normal.
2. During pregnancy: 600 µg folic acid daily
3. Eat folate-rich foods (e.g., leafy greens, citrus, o Urine culture: To detect
fortified cereals). asymptomatic UTIs due to increased
glucose in urine.
High-Risk Pregnancy: Endocrine System (Diabetes o Ophthalmic examination: Once
Mellitus) during pregnancy for gestational
Diabetes in Pregnancy: diabetes and each trimester for
known diabetics.
3. Home Pregnancy Test: o A 100g glucose solution is ingested,
o Important for detecting pregnancy and blood samples are taken at 1, 2,
early, as glucose control is crucial and 3 hours after ingestion.
during the early weeks. o A diagnosis of diabetes is made if
two of the four blood samples are
Important Notes: abnormal, or if the fasting value is
• Women with diabetes often require increased >95 mg/dL.
insulin around week 24 of pregnancy to
prevent hyperglycemia. Nursing Diagnoses for Gestational Diabetes:
• Women with poorly controlled diabetes face • Risk for ineffective tissue perfusion due to
significant risks during pregnancy, including reduced vascular flow
larger babies and increased risk of • Imbalanced nutrition related to the inability to
complications such as stillbirth or miscarriage. use glucose effectively
• Risk for ineffective coping related to required
lifestyle changes
CLASSIFICATION OF DIABETES
• Risk for infection related to impaired healing
• Deficient fluid volume due to polyuria
• Deficient knowledge about managing diabetes
during pregnancy
• Health-seeking behaviors related to the need
for home glucose monitoring
ASSESSMENT PATHOPHYSIOLOGY
1. Uterus unusually large for the date of pregnancy.
2. Sonogram is used to confirm 1. Low or absence of surfactant
3. Non-stress test 2. The hyaline-like (fibrous) membrane
4. Amniocentesis formed from an exudate of an infant’s
✓ To determine lung maturity blood that begins to line the terminal
5. CPD or shoulder dystocia bronchioles, alveolar ducts & alveoli
3. This membrane prevents exchange of
✓ Baby cannot descend through the outlet of the pelvis.
O2 & CO2 at the alveolar capillary
membrane
APPEARANCE
4. Alveoli collapse with each expiration
1. Immature reflexes
(areas of hypoinflation occurs)
2. Extensive bruising or a birth injury such as broken clavicle
5. Pulmonary resistance increases
or Erb-Duchenne paralysis
6. Blood then shunts through the foramen
✓ Due to trauma to the cervical nerves if he/she
ovale and the ductus arteriosus
was born vaginally. 7. The lungs become poorly perfuse
3. Caput succedaneum, cephalhematoma 8. Tissue hypoxia
9. Carbohydrate breaks down for energy
OTHER PROBLEMS and
1. CARDIOVASCULAR DYSFUNCTION 10. Release of lactic acid
• Observe for signs of hyperbilirubinemia which 11. Severe acidosis
resulted from absorption of blood from bruising 12. Acidosis causes vasoconstriction and
and polycythemia. decreases pulmonary perfusion from
• Closely monitor HR vasoconstriction further limits surfactant
• If cyanosis is present, it may be a sign of production
transposition of the great vessels. 13. Alveoli are collapsing
2. HYPOGLYCEMIA 14. Oxygen–carbon dioxide exchange in the
• Infants use up nutritional stores readily to sustain alveoli is no longer adequate to sustain
his/her weight. life without ventilator support.
• Increase blood glucose in utero causes infant to
produce elevated levels of insulin. Cause: Low level or absence of surfactant.
NURSING DX ASSESSMENT
1. Ineffective breathing pattern r/t possible birth trauma
• Increased ICP from birth could lead to pressure • Initial Signs After Birth:
on the [Link]. o Difficulty initiating respiration →
• Diaphragmatic paralysis may occur due to Resuscitation.
cervical nerve trauma. o After resuscitation, subtle signs may
• If delivered by C/S, transient fluid could remain in appear:
the lungs. ▪ Decreased body temperature
2. Risk for imbalanced nutrition less than body ▪ Nasal flaring
requirement ▪ Sternal & subcostal
r/t additional nutrients needed to maintain weight retractions
• Infant needs to be breastfed immediately to ▪ Tachypnea (60 bpm)
prevent hypoglycemia.
• May need supplemental formula feedings after
▪ Cyanotic mucous membranes
breastfeeding to supply enough fluid and glucose • Expiratory Grunting:
for the first few days. o Caused by closure of the glottis.
• Additional Distress Indicators:
RESPIRATORY DISTRESS SYNDROME (HYALINE o Seesaw respirations (chest wall
MEMBRANE SYNDROME) retraction & abdominal protrusion with
inspiration, sternum rise on expiration).
Occurs in: o Heart failure (edema, low urine output).
o Preterm infants o Cyanosis, pale gray skin.
o Infants of diabetic mother
o Periods of apnea, bradycardia.
o Infants born by C/S
o Possible pneumothorax (air in pleural
cavity).
Diagnostic Criteria o The weight of the fluid helps open the
1. Clinical Signs: alveoli.
o Grunting o As the liquid enters the lungs, it carries
o Cyanosis (room air) oxygen along, allowing for oxygen
o Tachypnea exchange.
o Nasal flaring 8. Nitric Oxide
o Retractions • Action:
o Shock o Nitric oxide causes pulmonary
2. Diagnostic Tests: vasodilation, which increases blood
o Chest X-ray: Haziness. flow to the alveoli, improving
o Blood Gas Studies: Respiratory oxygenation.
acidosis.
o Culture of Blood, CSF, & Skin: Rule Prevention of RDS
out infections like Group B • Preventing Preterm Delivery:
Streptococcus. 1. Tocolytic Agents (e.g., Terbutaline):
Used to delay preterm labor.
Therapeutic Management 2. Glucocorticosteroids (e.g.,
1. Surfactant Replacement: Betamethasone):
o Administered via syringe or catheter ▪ Accelerate the production of
through an endotracheal tube at birth. lecithin, a surfactant
o Note: Avoid suctioning after surfactant component.
administration. ▪ Typically administered
o Monitor closely for excessive ventilator between 24-34 weeks of
pressure. pregnancy to improve lung
2. Oxygen Administration (O2): development in premature
o Continuous Positive Airway Pressure infants.
(CPAP) or Positive End-Expiratory
Pressure (PEEP) to keep alveoli open. Meconium Aspiration Syndrome (MAS)
o Complications: Retinopathy of • Meconium is present in the fetal bowel as early as
prematurity, bronchopulmonary 10 weeks of gestation.
dysplasia.
• Cause:
3. Ventilation Methods:
o Hypoxia can trigger a vagus reflex,
o Normal: Inspiration shorter than relaxing the rectal sphincter and
expiration (I/E ratio 1:2).
releasing meconium into the amniotic
o Non-compliant lungs: Reversed I/E fluid.
ratio (2:1) or high-frequency
o In breech deliveries, pressure on the
oscillatory/jet ventilation.
buttocks may also cause meconium to
4. Indomethacin/Ibuprofen:
be expelled into the amniotic fluid.
o Helps close patent ductus arteriosus o This leads to meconium-stained
(PDA) to improve ventilation efficiency.
amniotic fluid, which appears green to
5. Muscle Relaxants (e.g., Pancuronium):
greenish black at birth.
o Reduces the need for high ventilator
pressures.
• Occurrence:
o Important: Keep Atropine & Prostigmin o Meconium staining occurs in 10%-20%
of births.
available to reverse effects if needed.
6. Extracorporeal Membrane Oxygenation o 2%-4% of these infants will develop
(ECMO): Meconium Aspiration Syndrome
(MAS).
o Blood removed from the baby,
oxygenated in ECMO machine, and o Extremely Low Birth Weight (ELBW)
returned to body. infants are less likely to aspirate
meconium because the substance
o Used in severe cases when ventilation is
hasn’t reached the rectum in these early
insufficient.
stages.
7. Liquid Ventilation
• Mechanism: • Aspirated Meconium:
o Perfluorocarbons are introduced into the o In utero or at birth when the infant
takes their first breath.
lungs, helping to distend poorly inflated
lungs that lack surfactant. o Meconium causes severe respiratory
distress in affected infants.
Signs & Symptoms 2. Cesarean Delivery:
• Immediate post-birth: o If deeply stained amniotic fluid is
1. Difficulty establishing respirations, noted during labor, consider scheduling
especially if not born breech. a cesarean section to reduce the risk of
2. Low Apgar score. meconium aspiration.
3. Tachypnea, retractions, and cyanosis 3. Post-Birth Management:
begin almost immediately. o Oxygen administration and assisted
▪ Resuscitation with positive ventilation may be required to manage
pressure ventilation may be respiratory distress.
required. 4. Antibiotic Therapy:
• After Resuscitation:
o Antibiotics may be prescribed to
prevent pneumonia, a common
1. Tachypnea persists.
secondary issue in MAS.
2. Coarse bronchial sounds are heard on
5. Surfactant Therapy:
auscultation.
3. Retractions continue: o If lung compliance is poor, surfactant
may be administered to help open the
▪ Inflammation in the bronchi
alveoli and improve oxygenation.
traps air in the alveoli,
6. High Inspiratory Pressure:
hindering oxygen entry.
o If lung noncompliance continues, higher
▪ This can lead to air trapping, inspiratory pressures may be needed.
increasing the
anteroposterior diameter of
o However, this can cause
pneumothorax (air in the pleural space)
the chest (resulting in a barrel
or pneumomediastinum (air in the
chest).
chest cavity).
• Respiratory Monitoring: 7. Air Trapping Monitoring:
o Pulse oximetry or blood gases reveal o Close observation is necessary to
poor gas exchange: monitor for air trapping in the alveoli,
▪ Decreased PO2 and which can cause rupture and result in
increased PCO2. pneumothorax.
▪ This indicates difficulty in o Increased pulmonary resistance can
oxygenating the blood keep the ductus arteriosus open,
properly. causing blood shunting from the
• Bilateral coarse infiltrates in the lungs. pulmonary artery to the aorta, which
increases hypoxia and compromises
• Hyper-aerated areas may be visible, creating a
cardiac efficiency.
honeycomb effect.
o Signs of heart failure (increased heart
• The diaphragm is often pushed downward due to rate, respiratory distress) should be
overexpanded lungs. monitored to detect a patent ductus
arteriosus.
Management Considerations 8. Temperature Maintenance:
• The infant should be placed under a warmer, and o Ensure a temperature-neutral
resuscitation should begin as needed (positive environment to reduce the infant’s
pressure ventilation). metabolic oxygen demand.
• Oxygen supplementation may be required to 9. Chest Physiotherapy:
maintain oxygen saturation in the mid to upper o Percussion and vibration may help
90s. remove meconium remnants from the
• Continued monitoring and respiratory support may lungs, aiding in better respiratory
be necessary as the infant recovers from the function.
distress caused by meconium aspiration. 10. Advanced Support:
o Some infants may require nitric oxide
Therapeutic Management of MAS therapy or may need to be placed on
1. Amnioinfusion: ECMO (Extracorporeal Membrane
o Isotonic fluid (Normal Saline) is Oxygenation) to ensure adequate
instilled into the uterine cavity using a oxygenation if conventional methods are
transcervical catheter. insufficient.
o Helps dilute meconium in the amniotic
fluid.
o May also improve oligohydramnios
(low amniotic fluid).
Sepsis Neonatorum (Neonatal Sepsis) Treatment
• Sepsis is a severe infection that spreads through • Antibiotics:
the bloodstream and throughout the body. o Babies in the hospital and those
• Neonatal Sepsis (Sepsis Neonatorum) refers to younger than 4 weeks are often started
sepsis occurring in newborns, which can affect on antibiotics even before lab results
various organs, such as the lungs, brain, bones, come back (usually takes 24-72 hours
and joints. for results).
o Ampicillin administered IV during
Causes & Risk Factors pregnancy and labor helps reduce the
• Microorganisms causing sepsis: risk of neonatal exposure to Group B
Streptococcus.
o Bacteria: E. coli, Candida, Group B
Streptococcus (GBS).
Nursing Interventions
o Viruses: Rubella, RSV, CMV, Herpes 1. Monitor Respirations:
simplex virus, Hib, Listeria,
Enterovirus.
o Watch for periods of apnea or irregular
respirations.
o Fungi & Parasites.
o Administer oxygen as prescribed.
• Early-Onset Neonatal Sepsis (within 24 hours of 2. Vital Signs Monitoring:
birth): o Monitor temperature, heart rate, and
o Infection acquired before or during respiratory rate.
delivery. 3. Maintain Warmth:
o Risk factors for early-onset sepsis: o Ensure a temperature-neutral
1. Group B Streptococcus environment to prevent temperature
infection during pregnancy. instability.
2. Preterm delivery. 4. Isolation:
3. Rupture of membranes o Provide isolation as necessary,
lasting longer than 24 hours. especially if sepsis is suspected to be
4. Infection of placenta tissues contagious.
(chorioamnionitis). 5. Monitor Hydration:
• Late-Onset Neonatal Sepsis (after delivery): o Keep track of input & output to watch
o Risk factors for late-onset sepsis: for dehydration.
1. Catheters in blood vessels for o Weigh daily to monitor growth and fluid
extended periods. balance.
2. Extended hospital stays. 6. Assess Feeding:
o Observe feeding and sucking reflex.
Symptoms of Infection 7. Monitor for Jaundice:
1. Apnea (stopping breathing) or difficulty breathing. o Assess the baby for yellowing of the
2. Bradycardia (decreased heart rate). skin (jaundice).
3. Temperature instability or hypothermia. 8. Assess for CNS Involvement:
4. Weak suck.
o Look for signs of irritability, lethargy,
5. Jaundice.
or any abnormal neurological
symptoms.
Diagnostic Tests
9. Diarrhea Monitoring:
1. Blood Cultures: To identify infection-causing
microorganisms.
o Watch for diarrhea as it may indicate
gastrointestinal issues.
2. C-reactive protein (CRP): Checks for
10. Administer Antibiotics:
inflammation in the body.
3. Complete Blood Count (CBC): To assess • Give antibiotics as ordered to treat the infection.
infection markers.
4. Lumbar Puncture: To examine cerebrospinal Preventive Measures
fluid (CSF) for infection. • Ampicillin administration during pregnancy and
5. Chest X-ray: If the baby has respiratory issues labor to reduce the risk of neonatal Group B
(e.g., cough, trouble breathing). Streptococcus exposure.
6. Urine Culture: For babies older than a few days,
to check for urinary infections. Newborn Priorities in the First Days of Life
1. Initiation and Maintenance of Respirations:
Ensuring the newborn's ability to breathe
effectively after birth.
2. Establishment of Extrauterine Circulation: 3. Lack of Prenatal Care: Limited or no medical
Transitioning from placental circulation to supervision during pregnancy.
independent circulation. 4. Multiple Pregnancy: Increased risk in twins,
3. Maintenance of Fluid and Electrolyte Balance: triplets, or more.
Monitoring hydration levels and electrolyte balance 5. Previous Early Birth: If the mother has had a
to prevent imbalances. previous preterm birth, the likelihood of it
4. Control of Body Temperature: Regulating the happening again increases.
newborn's body temperature to prevent 6. Race: Non-white populations (especially African
hypothermia or hyperthermia. American) have a higher incidence of prematurity.
5. Intake of Adequate Nourishment: Ensuring 7. Cigarette Smoking: Smoking during pregnancy
sufficient feeding, whether breast milk or formula, increases the risk of preterm birth.
for proper growth. 8. Age of the Mother: Teen mothers (under 20) are
6. Establishment of Waste Elimination: Monitoring at a higher risk for preterm birth.
for normal urine and stool output, indicating proper 9. Order of Birth: First pregnancies and pregnancies
organ function. beyond the fourth are at higher risk.
7. Prevention of Infection: Implementing measures 10. Closely Spaced Pregnancies: Short intervals
to protect the newborn from infections during the between pregnancies can increase the likelihood
first critical days. of preterm birth.
8. Establishment of Infant-Parent/Caregiver 11. Reproductive System Abnormalities: Issues
Relationship: Encouraging bonding and such as an intrauterine septum can contribute to
attachment for emotional and social development. preterm birth.
9. Developmental Care: Balancing physiological 12. Infections: Urinary tract infections and other
needs with appropriate stimulation for optimal infections are linked to preterm birth.
development.
• High-Risk Newborns: These same priorities Pregnancy Complications Increasing Risk of Preterm
apply to high-risk infants, though they may face Labor or Early Delivery:
challenges due to complications during pregnancy, 1. Premature Rupture of Membranes: Early
at birth, or adjusting to life outside the womb. breaking of the amniotic sac can lead to preterm
labor.
• Dysfunctional Transition: Some newborns may
2. Premature Separation of the Placenta
struggle with the transition to extrauterine life,
(Abruptio Placenta): Early detachment of the
possibly indicated by a low Apgar Score.
placenta can lead to preterm birth.
3. Incompetent Cervix: A weakened cervix that
Introduction
begins to open early, increasing the likelihood of
• Risk Factor Screening During Pregnancy: preterm labor.
Identifying maternal risk factors (e.g., age, 4. Birth Defects of the Uterus: Abnormalities in the
diabetes, HIV, placenta previa, drug use) helps uterus may contribute to preterm birth.
identify newborns who may need special care after 5. History of Preterm Delivery: A previous preterm
birth. delivery increases the risk for future preterm births.
• Dysmature Infants: Babies whose birth weight is 6. Infections: Infections, such as urinary tract
disproportionately low for their gestational age, infections or infections in the amniotic membrane,
whether preterm, term, or postterm, are at higher increase the risk of preterm labor.
risk for complications in the first days of life. 7. Poor Nutrition: Lack of proper nutrition right
before or during pregnancy can affect the baby's
Preterm Newborn development and increase the risk of preterm
• A premature infant is born before 37 weeks of birth.
gestation, which is more than 3 weeks before the 8. Preeclampsia: A condition characterized by high
due date. blood pressure and protein in the urine after the
20th week of pregnancy, increasing the risk of
Levels of Preterm Birth (WHO Classification): preterm delivery.
1. Extremely Preterm: Born before 28 weeks of 9. Premature Rupture of Membranes (PROM): A
gestational age (AOG). significant risk factor for preterm birth due to the
2. Very Preterm: Born between 28–32 weeks AOG. rupture of the amniotic sac before labor begins.
3. Late Preterm: Born between 32–37 weeks AOG.
Other Factors Increasing the Risk of Preterm Labor:
Common Factors Associated with Preterm Birth 1. Age of the mother:
1. Low Socioeconomic Status: Economic o Mothers younger than 16 or older than
difficulties that affect access to prenatal care. 35 years are at higher risk.
2. Poor Nutritional Status: Lack of proper nutrition
during pregnancy.
2. Lack of Prenatal Care: Insufficient prenatal care Acute Bilirubin Encephalopathy (ABE)
increases the likelihood of complications, including • Definition: ABE is the destruction of brain cells
preterm labor. due to the invasion of indirect or unconjugated
3. Low Socioeconomic Status: Limited access to bilirubin into the brain tissue.
resources and healthcare can lead to higher
preterm birth rates.
• Cause: This occurs because of high
concentrations of indirect bilirubin in the
4. Substance Use: Tobacco, cocaine, or
bloodstream, which result from excessive
amphetamines use during pregnancy increases
breakdown of red blood cells at birth.
the risk of preterm labor and complications.
• Risk in Preterm Infants:
Physical Characteristics of a Premature Infant: o Preterm infants are more prone to ABE
1. Disproportionately Large Head: The head may than term infants due to acidosis
be ≥3 cm larger than the chest size. caused by poor respiratory exchange,
2. Ruddy Skin: The skin is typically red due to low which makes brain cells more
subcutaneous fat, which makes veins visible. susceptible to indirect bilirubin.
3. Acrocyanosis: The hands and feet may appear o Preterm infants also have less serum
blue due to poor circulation (common in premature albumin, which is responsible for
infants). binding and inactivating indirect bilirubin,
4. Vernix Caseosa: Newborns born after 28 weeks increasing the likelihood of bilirubin
will usually have a covering of vernix, though those toxicity.
born earlier may lack it. • Management:
5. Lanugo: A fine, downy hair; in late preterm o Phototherapy or exchange
infants, it may cover the back, forearms, forehead, transfusion can be used to lower
and sides of the face, but it’s sparse in very bilirubin levels and prevent brain
preterm infants. damage.
6. Fontanelles: The anterior and posterior o Preterm infants may require these
fontanelles (soft spots on the skull) are often small interventions at lower bilirubin levels
in premature infants. than term infants to prevent ABE.
7. No Creases on the Soles: Very preterm infants
may have smooth soles without the usual creases. Persistent Patent Ductus Arteriosus (PDA)
Potential Problems in Premature Infants • Cause: Preterm infants often lack surfactant,
Anemia of Prematurity: which makes their lungs noncompliant. This
makes it harder for blood to move from the
• Characteristics: pulmonary artery into the lungs, leading to
o Many premature infants develop pulmonary artery hypertension and interfering
normochromic, normocytic anemia with the closure of the ductus arteriosus.
(normal red blood cells but fewer in
number).
• Management:
o This can cause symptoms like o Intravenous therapy must be
administered cautiously because
paleness, lethargy, and poor feeding.
increasing blood pressure could worsen
• Causes: the condition.
o Immaturity of the hematopoietic o Indomethacin or ibuprofen can be
system, where the production of red used in term infants to help close a
blood cells doesn’t start effectively until patent ductus arteriosus (PDA),
after 32 weeks of gestation. improving lung ventilation. However,
o Destruction of red blood cells due to low these medications are given cautiously
levels of vitamin E, which normally in preterm infants due to potential
protects red blood cells from oxidation. adverse effects:
• Risk Factors: ▪ Decreased renal function
o Excessive blood draws for tests such ▪ Decreased platelet count
as complete blood counts or blood gas ▪ Gastric irritation
analysis after birth can exacerbate the o If indomethacin is used, urine output
anemia. should be carefully monitored, and the
o It is essential to limit blood draws in infant should be observed for bleeding
preterm infants and carefully track any at injection sites.
blood loss.
Periventricular/Intraventricular Hemorrhage (PVH/IVH) 1. Group B strep infection during
• Cause: Preterm infants are at higher risk of pregnancy
periventricular hemorrhage (PVH) or 2. Premature delivery
intraventricular hemorrhage (IVH) due to fragile 3. Prolonged rupture of
capillaries and immature cerebral vascular membranes
development. 4. Chorioamnionitis (infection of
o Sudden changes in cerebral blood the placental tissues)
pressure (due to hypoxia, intravenous o Late-onset neonatal sepsis: Occurs
infusion, ventilation, or pneumothorax) after birth and may be caused by
can lead to capillary rupture and brain hospital-acquired infections, prolonged
anoxia, resulting in bleeding. catheter use, or extended hospital stays.
• Classification of IVH:
Laboratory Tests for Sepsis:
o Grade 1: Bleeding in the periventricular 1. Blood culture: To identify the microorganism
germinal matrix or one ventricle. causing the infection.
o Grade 2: Bleeding within the lateral 2. C-reactive protein (CRP): Measures inflammation
ventricle without ventricle dilation. levels in the body.
o Grade 3: Bleeding causing enlargement 3. Complete blood count (CBC): To assess white
of the ventricles. blood cell counts and signs of infection.
o Grade 4: Bleeding in the ventricles and 4. Lumbar puncture: To examine cerebrospinal fluid
surrounding brain tissue. for bacteria if meningitis is suspected.
• Complications: Long-term complications of IVH 5. Chest x-ray: Used if there are respiratory
include hydrocephalus (fluid buildup in the brain), symptoms (e.g., cough or breathing problems).
especially if bleeding occurred into the narrow 6. Urine culture: Performed in babies older than a
aqueduct of Sylvius. few days to check for urinary tract infection.
• Monitoring and Prognosis:
Symptoms of Infection in Newborns:
o Preterm infants often have a cranial
1. Apnea (stopping breathing) or difficulty
ultrasound after birth and at regular
breathing
intervals to detect IVH.
2. Bradycardia (low heart rate)
o Infants with Grade 1 or 2 IVH usually 3. Temperature instability (either low body
have a good long-term prognosis.
temperature or fever)
o The prognosis for Grade 3 or 4 IVH is 4. Weak suck (difficulty feeding)
more guarded, and further complications 5. Jaundice (yellowing of the skin)
need to be ruled out.
Treatment:
Infection or Neonatal Sepsis (Sepsis Neonatorum)
• Immediate Antibiotics: Newborns younger than 4
• Definition: Sepsis neonatorum refers to a severe weeks, especially those in the hospital, are started
infection that enters the bloodstream and spreads on broad-spectrum antibiotics before laboratory
throughout the body, potentially affecting organs results are available.
such as the lungs, brain, bones, joints, or other
o Common antibiotics include ampicillin,
tissues.
often administered intravenously during
• Causes: Neonatal sepsis can be caused by labor to prevent infection.
microorganisms such as:
1. Bacteria: Escherichia coli (E. coli), Nursing Interventions:
Group B Streptococcus (GBS), Candida 1. Assess respiratory status: Monitor for apnea or
2. Viruses: Rubella, Respiratory Syncytial irregular breathing.
Virus (RSV), Cytomegalovirus (CMV), 2. Administer oxygen: As prescribed, to support
Varicella-zoster virus (chickenpox), oxygenation.
Herpes simplex virus, Haemophilus 3. Monitor vital signs (temperature, heart rate,
influenzae type b (Hib), Listeria respiratory rate, and blood pressure).
monocytogenes, Enterovirus 4. Maintain warmth: Keep the newborn warm to
3. Fungi and Parasites prevent hypothermia.
• Types: 5. Provide isolation: To prevent the spread of
o Early-onset neonatal sepsis: Appears infection if necessary.
within 24 hours of birth, typically 6. Monitor temperature: Watch for signs of
transmitted from the mother during hypothermia or hyperthermia.
pregnancy or delivery. Risk factors 7. Observe input and output: Monitor hydration
include: status and for signs of dehydration.
8. Weigh the baby daily: Track growth and fluid substance that helps maintain the surface tension
retention. of the alveoli and prevents them from collapsing.
9. Monitor for diarrhea: As a sign of gastrointestinal • Hyaline Membranes: Without sufficient surfactant,
involvement. an exudate of the infant’s blood lines the terminal
10. Assess feeding and sucking reflex: To ensure bronchioles, alveolar ducts, and alveoli, forming a
adequate nourishment. hyaline-like (fibrous) membrane. This
11. Assess jaundice: For early detection of bilirubin- membrane prevents the exchange of oxygen (O2)
related complications. and carbon dioxide (CO2) at the alveolar-capillary
12. Monitor for irritability or lethargy: These may membrane.
indicate central nervous system involvement.
13. Administer antibiotics as prescribed and adjust
• Alveolar Collapse: As a result, the alveoli
collapse with each expiration, leading to
based on culture results.
hypoinflation and pulmonary resistance. This
worsens blood flow to the lungs, causing blood to
Low Blood Sugar (Hypoglycemia)
shunt through the foramen ovale and ductus
• Definition: Hypoglycemia in neonates refers to arteriosus.
low blood sugar levels. Preterm neonates,
particularly those with extremely low birth weight
• Tissue Hypoxia: Inadequate oxygenation and
high lactic acid production cause severe acidosis,
(ELBW), are especially vulnerable.
which can result in vasoconstriction and further
• Causes: Preterm neonates are at a higher risk for impair pulmonary perfusion. The lack of surfactant
hypoglycemia due to: continues to worsen lung function.
1. Limited glycogen and fat stores
2. Reduced ability to produce glucose Causes:
through gluconeogenesis
3. Higher metabolic demands,
• Primary Cause: Low level or absence of
surfactant in preterm infants.
particularly due to a larger brain relative
to body size • Risk Factors:
Categories of Hypoglycemia: o Preterm birth
1. Mild Hypoglycemia: 40 to 50 mg/dL (2.2 to 2.8 o Infants born to diabetic mothers
mmol/L) o Cesarean section delivery
2. Moderate Hypoglycemia: 20 to 40 mg/dL (1.1 to o Conditions like meconium aspiration,
2.2 mmol/L) which reduces blood flow to the lungs.
3. Severe Hypoglycemia: Below 20 mg/dL (1.1
mmol/L) Assessment and Clinical Signs:
1. At Birth: Difficulty initiating respiration may require
Factors Contributing to Hypoglycemia in Preterm immediate resuscitation.
Neonates: 2. After Resuscitation, the following signs may
• Extremely low birth weight (ELBW): These appear:
infants have low glycogen and fat reserves. o Decreased body temperature
• Enzyme deficiency: Enzymes involved in o Nasal flaring
gluconeogenesis (such as PEPCK, glucose-6- o Sternal and subcostal retractions
phosphatase, fructose-1,6-diphosphatase, and o Tachypnea (rapid breathing, typically
pyruvate carboxylase) are often at very low over 60 breaths per minute)
levels, making it difficult for the body to produce o Cyanotic mucous membranes (bluish
new glucose from non-carbohydrate sources. color of the lips and mouth)
o Expiratory grunting due to closure of
Neonatal Respiratory Distress Syndrome (Hyaline the glottis
Membrane Syndrome) o Rales (crackling sounds) and
Definition: diminished breath sounds due to poor
Neonatal Respiratory Distress Syndrome (RDS), also known air entry
as Hyaline Membrane Syndrome, is a condition 3. As Respiratory Distress Increases:
predominantly affecting preterm infants and those with o Seesaw respirations: The chest wall
certain risk factors such as infants of diabetic mothers or retracts during inspiration, and the
those born via cesarean section. It is caused by a lack of abdomen protrudes; during expiration,
surfactant in the lungs, which is essential for lung function the sternum rises.
after birth. o Heart failure: Reduced urine output and
extremity edema may develop.
Pathophysiology:
o Pale, gray skin indicating poor
• Surfactant Deficiency: The primary cause of oxygenation.
RDS is the low level or absence of surfactant, a
o Periods of apnea (temporary cessation o Muscle relaxants like Pancuronium
of breathing). may be used to allow mechanical
o Bradycardia (slow heart rate). ventilation at lower pressure. This helps
o Pneumothorax (presence of air in the reduce the risk of pneumothorax while
pleural cavity, leading to lung collapse). improving oxygenation (pO2).
o Emergency Medications: Atropine
Diagnosis: and Prostigmin should be available to
1. Clinical Signs: reverse the effects of pancuronium if
o Grunting, cyanosis (on room air), necessary.
tachypnea, nasal flaring, retractions,
and shock. Extracorporeal Membrane Oxygenation (ECMO)
2. Chest X-ray: Shows haziness in the lungs.
3. Blood Gas Studies: Often reveal respiratory ECMO is a life-saving intervention for infants with severe
acidosis. respiratory or cardiac failure. It temporarily takes over the
4. Culture of Blood, CSF, and Skin: To rule out heart and lung functions to provide oxygenation and
infections such as Group B Streptococcus that circulation.
could mimic RDS.
How It Works:
Therapeutic Management: 1. Blood is drawn from the baby using a venous
1. Surfactant Replacement: catheter that is placed into the right atrium of the
o Surfactant is administered through an heart.
endotracheal tube at birth to help lower 2. The blood is then oxygenated and rewarmed by an
surface tension in the lungs and prevent ECMO machine.
alveolar collapse. 3. After being oxygenated, the blood is returned to
o Important: Avoid suctioning the infant's the aortic arch through a catheter inserted into
airway after surfactant administration. the carotid artery.
Indication:
o Monitoring: Infants on surfactant
ECMO is typically used in cases of severe respiratory
therapy may be placed on a ventilator,
distress syndrome (RDS) or other critical conditions that do
and close observation is necessary
not respond to conventional treatments.
because lung expansion can rapidly
improve, and ventilator pressure may
Liquid Ventilation
need adjustment.
2. Oxygen Administration:
Liquid ventilation is an advanced therapeutic technique that
o Oxygen is delivered to maintain introduces a fluid, typically perfluorocarbons, into the lungs
appropriate partial pressure of oxygen
of infants with surfactant deficiencies. This technique helps
(pO2) and pH levels.
improve lung function by expanding poorly inflated alveoli.
o Methods include Continuous Positive
Airway Pressure (CPAP) or Positive
How It Works:
End-Expiratory Pressure (PEEP) to
prevent alveolar collapse during • The perfluorocarbons help to distend the lungs
exhalation. by their weight, enabling better oxygenation.
o Complications: Retinopathy of • The liquid carries oxygen into the lungs, facilitating
prematurity and bronchopulmonary an exchange of oxygen and carbon dioxide, even
dysplasia are potential risks from in the absence of sufficient surfactant.
oxygen therapy.
3. Ventilation: Nitric Oxide Therapy
o Normal Breathing Pattern: Inspiration
is shorter than expiration (I/E ratio 1:2). Nitric oxide is used to vasodilate pulmonary blood vessels,
o In Non-Compliant Lungs: A reversed improving blood flow to the alveoli and enhancing
I/E ratio (2:1) may be necessary. High- oxygenation.
frequency oscillatory or jet ventilation Effect:
may also be used. • It helps reduce pulmonary hypertension and
4. Indomethacin or Ibuprofen: increases blood flow to areas of the lungs
o These medications can be used to help where oxygen exchange is most effective.
close the patent ductus arteriosus
(PDA), improving ventilation and Prevention of Preterm Delivery:
oxygenation. 1. Tocolytic Agent (Terbutaline):
5. Muscle Relaxants (Pancuronium): o Used to stop contractions and delay
preterm labor.
2. Glucocorticosteroid (Betamethasone): 2. Body Hair (Lanugo):
o Given between 24–34 weeks of o Fine hair covering the body, common in
pregnancy to speed up the production preterm infants.
of surfactant and improve lung 3. Enlarged Clitoris (in female infants):
development in preterm infants. o Due to hormonal influences during fetal
development.
Extra Air in the Tissue of the Lungs (Pulmonary 4. Lower Muscle Tone:
Interstitial Emphysema) o Premature infants typically have lower
muscle tone and are less active.
Pulmonary interstitial emphysema occurs when air leaks 5. Feeding Difficulties:
from the alveoli into the tissues surrounding the lungs, o Difficulty coordinating sucking,
causing air pockets to form. This condition is most swallowing, and breathing, which makes
associated with ventilator use in premature infants. feeding challenging.
6. Undescended Testicles (in male infants):
Risk Factors: o A common occurrence in premature
• Mechanical ventilation male infants.
• High pressures in the lungs 7. Soft, Flexible Ear Cartilage:
o Preterm infants may have soft ear
Bleeding in the Lungs (Pulmonary Hemorrhage) cartilage that can be molded easily.
8. Thin, Shiny Skin:
Pulmonary hemorrhage refers to bleeding in the lungs, o Skin may appear transparent, and veins
which can occur in premature infants, especially those who are visible underneath due to insufficient
are on mechanical ventilation. fat stores.
ASSESSMENT
1. Frothy saliva in the mouth and nose, and drooling
2. Coughing and choking during feeding 13. Instruct parents to identify behaviors that indicate
3. Unexplained cyanosis the need for:
4. Regurgitation and vomiting • Suctioning
5. Abdominal distention
6. Inability to pass a small gauge (no. 5 French)
• Signs of respiratory distress
orogastric feeding tube • Signs of constricted esophagus such as:
o Poor feeding
TREATMENT o Dysphagia
1. Maintenance of a patent airway o Drooling
2. Prevention of pneumonia o Regurgitated undigested food
3. Gastric or blind pouch decompression
4. Supportive therapy
5. Surgical repair CARE OF AN INFANT WITH CHEST TUBE
STEP 1
IMPLEMENTATION • Examine the area around the tubing often to help
prevent potential infection.
PREOPERATIVE PERIOD
o Make sure the area surrounding the
1. Intubation and mechanical ventilation may be
chest tube is as clean and sterile as
necessary if respiratory distress occurs
possible
2. Maintain NPO status
3. Monitor and regulate IV fluids as prescribed
o The area surrounding the incision and
chest tube should be covered and
4. Suction secretions
protected from the surrounding
5. A double-lumen catheter is placed into the upper
environment
esophageal pouch and attached to intermittent or
continuous low suction to keep the pouch empty o Ensure that tubing is not kinked or
6. Maintain in an upright position to facilitate otherwise bent
drainage and to prevent aspiration of gastric STEP 2
secretions • Lift infant by placing one hand beneath her
7. A gastrostomy tube may be placed and is left head and shoulders, the other beneath her
open so that air entering the stomach through the buttocks
fistula can escape, minimizing the danger of o Don't lift an infant with a chest tube by
regurgitation clasping her under the arms and lifting,
8. Administer broad-spectrum antibiotics as as this may cause pain and dislodge
prescribed because of the high risk for aspiration the tube placement
pneumonia o You may hold the child on your lap or
encourage her to breast feed or bottle
POSTOPERATIVE PERIOD feed, always taking care to provide
1. Monitor respiratory status adequate back and neck support
2. Maintain IVFs, antibiotics, and parenteral o Don't place the infant on her stomach
nutrition as prescribed STEP 3
3. Monitor I & O and weigh daily • You can start giving the infant a tub bath or
4. Inspect surgical site shower after the stitches have been removed or
5. Assess for signs of pain after about one week post-surgery
6. Assess for dehydration and possible fluid
o Make sure the bathing area and water
overload
are clean
7. Provide care to the chest tube if in place
8. Monitor for anastomotic leaks as evidenced by:
HYPERTROPHIC PYLORIC STENOSIS
o Purulent chest drainage
o Increased temperature • Hypertrophy of the circular muscles of the
pylorus, causing narrowing of the pyloric canal
o Increased WBC count between the stomach and the duodenum
9. If a gastrostomy tube is present, it is attached to
gravity drainage until the infant can tolerate
o Baby fails to grow
feedings (usually 5–7th day postop) o Less fluid can pass through this defect
10. Before oral feedings and removal of the chest o Majority of absorption in the small
tube, a barium swallow is performed to verify the intestine
integrity of the esophageal anastomosis o Because of distention, baby will vomit
11. Feedings through the gastrostomy tube may be (characteristics: projectile vomiting);
prescribed until the anastomosis is healed fast intake, slow discharge
12. Oral feedings are begun with sterile water, o Tracheoesophageal – non-projectile;
followed by small frequent feedings of formula equal to the amount that is ingested
• Usually develops in the first few weeks of life, PREOPERATIVE CARE
causing: 1. Monitor hydration by daily weights, I&O, urine
o Projectile vomiting specific gravity
o Dehydration o General anesthesia causes muscle
relaxation – danger of aspiration and
o Metabolic alkalosis
airway obstruction
o Failure to thrive 2. Correct fluid and electrolyte imbalances
• Vomiting is the first symptom in most children: o Administer IVF as prescribed
o May occur after every feeding or only 3. Maintain NPO status
after some feedings 4. Monitor number and character of stools
o Usually starts around 3 weeks of age, 5. Maintain NGT patency for stomach
but may occur between 1 week to 5 decompression
months o NGT is not for feeding – removes air
o Vomiting is forceful (projectile and fluids from stomach
vomiting) = stomach is overdistended
o Infant is hungry after vomiting and POSTOPERATIVE CARE
wants to feed again 1. Monitor I&O
2. Maintain IVF until infant takes and retains oral
Other Symptoms fluids
• Abdominal pain (overdistention) o Wait for doctor's signal (return of
• Belching (dug-ab) peristalsis)
o Auscultate for bowel sounds
• Constant hunger (less nutrients absorbed)
3. Begin small, frequent feedings of glucose water
• Dehydration (gets worse with severity of vomiting) or electrolyte solution 4–6 hrs. postop
o Less fluid enters small intestine = less 4. Advance to formula 24 hrs. postop as prescribed
fluid 5. Gradually increase amount and interval until full
• Failure to gain weight or weight loss feeding schedule resumed (48 hrs. postop)
• Wave-like motion of the abdomen shortly after 6. Feed slowly, burping frequently to prevent
feeding and just before vomiting tension on suture line
7. Monitor for abdominal distention
SIGNS AND TESTS 8. Monitor surgical wound for infection:
o Redness
• Diagnosed before baby is 6 months old
o Drainage
• Physical exam may reveal signs of dehydration o Pus formation
• Swollen abdomen 9. Instruct parents on wound care and feeding
• Doctor may detect abnormal pylorus = olive-
shaped mass DIAPHRAGMATIC HERNIA
• Ultrasound of abdomen is the first imaging test • Definition: A diaphragmatic hernia is a birth defect
• Other tests:
where there is an abnormal opening in the
diaphragm, allowing abdominal organs to move
o Barium x-ray → reveals swollen into the chest cavity. This condition occurs before
stomach & narrowed pylorus
birth during fetal development.
▪ Upper GI (barium swallow) – o Cyanosis and abnormal appearance
baby swallows liquid for
may be observed in the baby after birth.
tracing
o An issue that develops in-utero due to
o Blood chemistry panel → shows the abnormal formation of the
electrolyte imbalance
diaphragm.
SURGERY • Cause:
• Pyloromyotomy – pyloroplasty (ultimate
o Genetic and environmental factors, such
as exposure to teratogens.
intervention)
o Widen the opening in the pylorus so o Diaphragm formation occurs between
the 7th and 10th week of pregnancy;
contents can empty into small intestine
any disruption can lead to congenital
• Balloon dilation doesn’t work as well, but may be anomalies.
used if anesthesia is risky o In Bochdalek hernia, improper
• Fluids via IV are given before surgery (NPO baby) diaphragm formation or trapped
intestines during development.
o In Morgagni hernia, improper o The abdominal organs are moved back
development of the diaphragm’s tendon. into the abdominal cavity, and the
diaphragm is repaired.
TWO TYPES OF DIAPHRAGMATIC HERNIA
1. BOCHDALEK HERNIA: LONG-TERM CARE:
o Most commonly (85%) occurs on the left • Babies with diaphragmatic hernias often face
side of the diaphragm. chronic lung disease and may need long-term
o Stomach, liver, spleen, and/or intestines oxygen support or medications.
may move into the chest cavity, leading • Regular follow-up care is essential after hospital
to more severe symptoms. discharge for continued support of respiratory
2. MORGAGNI HERNIA: health.
o Occurs on the right side of the
diaphragm.
o Liver and/or intestines move into the TREATMENT/NURSING CARE FOR DIAPHRAGMATIC
chest cavity. HERNIA (CDH)
• Physiologic Emergency: Diaphragmatic hernia is
SYMPTOMS:
a physiologic emergency rather than a surgical
• Bochdalek Hernia: one. The critical concern is preventing or
o Difficulty breathing improving Persistent Pulmonary Hypertension of
o Rapid breathing the Newborn (PPHN).
o Fast heart rate o Medical Management:
o Cyanosis ▪ Stabilize the infant’s condition.
o Abnormal chest development (one side ▪ Manage pulmonary
larger than the other) dysfunction before
o Concave abdomen considering surgery.
• Morgagni Hernia: ▪ Delay surgery if needed (from
o Symptoms may or may not appear after a few days up to two weeks),
birth. depending on the infant's
oxygenation and nutritional
DIAGNOSIS: status.
1. Physical examination ▪ The goal is to decrease
2. Chest x-ray: Used to assess lung, diaphragm, and pulmonary hypertension and
intestine abnormalities. stabilize pulmonary
3. Arterial blood gas: To evaluate the baby’s dysfunction.
breathing ability. o Respiratory Management:
4. Other tests: ▪ Provide adequate ventilation
o Chromosome blood test (to check for and oxygenation while
genetic problems). avoiding permanent lung
o Echocardiogram (ultrasound of the damage.
heart).
Treatment Options:
TREATMENT: • Gastric Decompression:
1. Neonatal Intensive Care (NICU): - Use of oro/nasogastric tubes with low
o Life-threatening condition, requiring care continuous suction to prevent air from
in NICU. entering the intestines, which could
o Mechanical ventilators may be needed obstruct the lungs and cause
to assist with breathing due to regurgitation.
underdeveloped lungs. • Inhaled Nitric Oxide:
2. ECMO (Extracorporeal Membrane - A vasodilator that decreases pulmonary
Oxygenation): hypertension (PPHN).
o A heart-lung bypass machine that helps - It dilates the pulmonary vascular bed,
temporarily support the heart and lungs. improving oxygenation.
o Used to stabilize the baby’s condition. • Pressors (Hypertensive Drugs):
3. Surgery: - Increase systemic blood pressure to
o Once the baby stabilizes, surgery is override pulmonary vascular resistance.
performed to repair the diaphragmatic - Reduce right-to-left shunting and
hernia. increase pulmonary blood flow.
- Common pressors include:
Dopamine: Increases peripheral vascular resistance. Diagnosis
Administer at 2 mcg/kg/min, up to 20 mcg/kg/min via • Sonography to determine type of imperforate
continuous infusion. anus.
Dobutamine: Increases cardiac contractility. Administer at 2 Treatment
mcg/kg/min, up to 20 mcg/kg/min via continuous infusion.
• Surgery: Open passage for feces or use fistula
o Parent Education: until corrective surgery.
Pre-operative Education:
Inform parents about surgery
o Perineal anoplasty or colostomy (higher
defect).
plans and post-operative
Postoperative Care
expectations.
1. Monitor for infection.
▪ Post-operative Education: 2. Position side-lying with legs flexed or prone to
Update parents on the infant's reduce edema.
status, and review any new 3. Keep incision clean and dry.
equipment, medications, or 4. Maintain NPO status and NGT if in place.
therapies. 5. Maintain IV fluids until GI motility returns.
6. Provide colostomy care, keeping the area dry.
INTESTINAL OBSTRUCTION: IMPERFORATE ANUS 7. Fresh colostomy stoma may be red and
edematous, will decrease over time.
Imperforate anus is a congenital defect where the anus is 8. Instruct parents on anal dilatation (if prescribed) to
either missing or blocked. maintain bowel patency:
• Forms of Imperforate Anus: o Use only prescribed dilators with water-
1. Low Lesion: soluble lubricant, inserting 1-2 cm to
▪ The colon is close to the skin, avoid mucosal damage.
possibly covered by a
membrane. Hirschsprung’s Disease
▪ The anus may be narrowed or • Absence of ganglionic innervation, usually in the
missing, and the rectum may sigmoid colon, leading to chronic constipation and
end in a blind pouch. ribbon-like stools.
2. High Lesion:
• Cause: Abnormal gene on chromosome 10.
▪ The colon is higher in the Assessment
pelvis, with a fistula 1. Failure to pass meconium by 24 hours.
connecting the rectum to the 2. Abdominal distention.
bladder, urethra, or vagina. 3. History of constipation, intermittent
▪ Symptoms may include constipation/diarrhea.
meconium mixed in urine 4. Empty rectum on examination.
(green color). 5. Barium enema to assess affected area.
3. Persistent Cloaca: 6. Biopsy for confirmation.
▪ A rare anomaly where the Therapeutic Management
rectum, vagina, and urinary • Surgery: Dissection and removal of affected bowel
tract form a single channel. section, followed by anastomosis. Temporary
colostomy, bowel repair at 12-18 months.
ASSOCIATED ANOMALIES: Diagnosis
These conditions develop from the same germ layer during 1. Constipation related to reduced bowel function.
fetal development. Some anomalies include: 2. Imbalanced nutrition due to bowel issues.
• V: Vertebral anomalies 3. Risk of compromised family coping due to chronic
• A: Anal atresia (absence or closure of the anus) illness.
Nursing Interventions
• C: Cardiovascular anomalies
Preoperative:
• T: Tracheoesophageal fistula 1. Empty bowel with enemas and colonic irrigations.
• R: Renal (kidney) and/or radial (bone) anomalies 2. If distention persists, consult doctor for NG tube.
• E: Esophageal atresia 3. Offer pacifier for infants on parenteral fluids.
4. Encourage parent-infant bonding.
• L: Limb defects
5. Position with head elevated to prevent discomfort.
6. Offer soothing stimulation (e.g., music, touch).
7. Provide small, frequent feedings (low residue diet).
8. Administer parenteral nutrition if needed.
9. Provide instructions for saline enema and stool
softeners for older children.
Postoperative: Therapeutic Management
1. Change dressings using sterile technique. 1. Non-surgical reduction: Water-soluble contrast
2. Prevent wound contamination from diapers. enema, barium or air contrast enema.
3. Prevent excoriation by cleaning and applying 2. Surgical reduction: Manual reduction or resection if
ointments. ischemia/necrosis present. Laparoscopic approach
4. Use strict handwashing. may be used.
5. Report any redness, swelling, drainage, evisceration,
or dehiscence immediately. Nursing Diagnosis & Interventions
6. Suction oral secretions frequently to prevent Risk for Deficient Fluid Volume due to Bowel Obstruction:
respiratory infections. 1. Keep infant NPO pre-surgery or non-surgical
7. Encourage older children to cough and deep breathe reduction (pacifier for comfort).
to maintain respiratory status. 2. Administer IVF to re-establish fluids and
8. Allow infants to cry briefly to prevent atelectasis. electrolytes.
9. Change infant's position frequently to improve 3. After non-surgical reduction: Keep NPO for a few
circulation and lung aeration. hours, gradually introduce regular feedings.
10. Maintain NG tube patency to prevent abdominal 4. After surgery:
distention. o Keep NG tube on low suction until
11. Keep NPO until bowel sounds return, as per healing and return of peristalsis.
physician's orders. o Keep IVF, start oral feedings gradually
12. Provide frequent oral hygiene while NPO. when bowel sounds are present.
13. Administer fluids to maintain hydration and
electrolytes and begin oral feedings when ordered. Developmental Disorders of the Nervous System
14. Support parents in colostomy care, reassuring that 1. Hydrocephalus (abnormal CSF accumulation)
it won't delay normal development. 2. Neural tube closure abnormalities (e.g.,
15. Involve the whole family in teaching colostomy care meningocele, meningomyelocele)
for better acceptance.
Hydrocephalus
Intussusception
• Excess CSF in ventricles/subarachnoid space
leading to brain swelling.
• The invagination of one bowel segment into
• Normal ICP: 5-15 mmHg, with 500 ml CSF
another, common in infants, typically after 6
produced/day.
months.
Causes
• Causes include Meckel’s diverticulum, polyp,
• Overproduction of CSF (rare, due to tumor).
hypertrophy of Peyer’s patches, or tumors in older
infants. • Obstruction in fluid passage (e.g., aqueduct of
Symptoms Sylvius, foramina of Magendie and Luschka,
Early: hemorrhage, infection).
• Nausea, vomiting (sometimes bile-stained), pulling • Interference with CSF absorption (e.g., post-
legs to chest. surgery or hemorrhage).
Additional Causes
• Intermittent, severe abdominal cramping.
Later: • Genetic defects, infections during pregnancy,
bleeding, trauma, tumors.
• "Red currant jelly" stool (blood and mucus).
Types of Hydrocephalus
• "Sausage-shaped" mass in the upper right 1. Communicating (Extraventricular): Fluid
abdomen. reaches spinal cord.
• Risk of ischemia, necrosis, perforation, and sepsis. 2. Obstructive (Intraventricular): Blocked fluid
circulation.
3. Congenital: Present at birth, often with
meningomyelocele.
4. Acquired: Occurs later in life, such as from
prematurity or intraventricular hemorrhage.
Signs and Symptoms
Early signs (infants):
1. Widened, tense fontanelles and separated
sutures.
2. Enlarged head, shiny scalp, prominent veins.
3. Brow bulging (bossing), “sunset eyes” (sclera
above iris).
4. Seizures, lethargy, vomiting.
5. Increased ICP: Decreased pulse, increased BP, o Outcome: No signs of increased ICP or
hyperactive reflexes. altered vital signs.
6. Irritable or lethargic with shrill cry. o Interventions:
Symptoms in Older Children: ▪ Keep the bed flat or slightly
1. High-pitched cry, personality/memory changes. raised.
2. Facial changes crossed eyes.
▪ Avoid turning the child onto
3. Difficulty feeding, excessive sleepiness.
the side with the shunt.
4. Headaches, irritability, urinary incontinence.
5. Loss of coordination, muscle spasticity, slow ▪ Prevent exposure to magnets
growth. (affects shunt function).
Tests ▪ Monitor for signs of ICP (e.g.,
1. Head circumference measurements. tense fontanelles, vomiting).
2. CT scan, cranial ultrasound, arteriography. ▪ Check for infection signs (e.g.,
3. Brain scan, skull X-rays, lumbar puncture (rare). fever, irritability).
4. Transillumination to detect fluid-filled skull. ▪ Manage pain to prevent CSF
Treatment Goal pressure increase.
• To prevent brain damage by improving CSF flow, 2. Risk for Imbalanced Nutrition (related to
tailored to the cause and extent of hydrocephalus. increased ICP)
o Outcome: Child’s weight remains within
1. Overproduction of Fluid: the normal range; no vomiting.
o Acetazolamide (Diamox): Diuretic to o Interventions:
excrete excess CSF. ▪ NPO until bowel sounds
o Ventricular Endoscopy: Destroys part return; gradually introduce
of the choroid plexus. fluids.
o Tumor Removal: If a tumor is causing ▪ Ensure proper head support
fluid overproduction. while feeding.
2. Laser Surgery: ▪ Monitor for ineffective sucking
o Used to reopen fluid flow or bypass or vomiting as signs of
obstructions, especially in children increased ICP.
without neural tube involvement. 3. Risk for Impaired Skin Integrity (related to head
3. Ventriculoperitoneal Shunt: weight/immobility)
o A catheter from the ventricles to the o Outcome: Skin remains intact and free
peritoneum to drain and absorb CSF. of breakdown.
4. Endoscopic Third Ventriculostomy (ETV): o Interventions:
o Relieves pressure without the need for a ▪ Change the child’s head
shunt. position every 2 hours.
5. Cauterizing: ▪ Use a pressure-relieving
o Removing or burning away parts of the mattress or padding.
brain that produce excess CSF.
▪ Ensure surgical dressings
6. Regular Check-ups:
remain dry and secure.
o To monitor for developmental,
intellectual, neurological, and physical
▪ Prevent head flattening
(nonsynostotic plagiocephaly).
issues.
4. Deficient Knowledge (related to home care for
Other Treatments:
hydrocephalus)
• Antibiotics for infections. o Outcome: Parents feel confident in
• Shunt Removal: In severe infections like managing care.
meningitis. o Interventions:
Complications: ▪ Address parents’ anxieties
1. Surgical complications and provide support.
2. Infections (meningitis, encephalitis)
▪ Educate about the shunt, its
3. Intellectual impairment
care, and infection prevention.
4. Nerve damage (e.g., loss of movement, sensation)
5. Physical disabilities ▪ Teach signs of increased ICP
(e.g., irritability, vomiting).
Nursing Diagnoses and Interventions: ▪ Educate on the severity of
1. Risk for Ineffective Cerebral Tissue Perfusion shunt infections and
(related to increased ICP) necessary interventions
🧬 Neural Tube Defects (NTDs) 5. Abnormal eye movements
Latex allergy (up to 68%) — avoid latex in gloves,
• Group of congenital malformations from improper
6.
catheters, dental tools.
neural tube closure.
• Often referred to collectively as Spina Bifida. Neurological Complications
• Occurs early in intrauterine life due to failure of
posterior fusion of the embryo. Arnold-Chiari II Malformation:
• Cerebellum and brainstem herniate into foramen
Types of Neural Tube Defects: magnum.
1. Anencephaly
• Causes hydrocephalus due to CSF flow
• Absence of cerebral hemispheres. obstruction.
• Usually results in neonatal death (medulla intact— Hydrocephalus:
may breathe briefly). • Common in spina bifida due to cerebellar
• Diagnosed by elevated AFP, amniocentesis, or displacement.
prenatal ultrasound. • Often requires shunting.
• Emotional support is critical; allow grieving rituals Brain Structural Changes:
(e.g., holding, naming the baby). • Smaller cerebellum
2. Microcephaly
• Frontal cortex may be thickened; parietal/posterior
areas thinner.
• Brain is underdeveloped (more than 3 SD below
normal). 4. Encephalocele
• Causes: • Cranial defect where brain/meninges herniate.
o Intrauterine infections (e.g., rubella,
• Most common in the occipital region.
CMV, toxoplasmosis)
o Zika virus (via mosquitoes) • Covered by skin or dura; imaging (MRI/CT)
o Malnutrition or birth anoxia confirms diagnosis.