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Alpha Helix and R-Group Interactions

Chapter 4 discusses the native conformation of proteins, emphasizing the importance of structure for function and the role of various interactions, including covalent bonds and weak interactions, in stabilizing protein structures. It details secondary structures like alpha helices and beta sheets, their formation, and the constraints imposed by amino acid residues. The chapter also touches on protein classification, proteostasis, and the implications of misfolding in diseases such as cystic fibrosis and prion disorders.

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0% found this document useful (0 votes)
6 views3 pages

Alpha Helix and R-Group Interactions

Chapter 4 discusses the native conformation of proteins, emphasizing the importance of structure for function and the role of various interactions, including covalent bonds and weak interactions, in stabilizing protein structures. It details secondary structures like alpha helices and beta sheets, their formation, and the constraints imposed by amino acid residues. The chapter also touches on protein classification, proteostasis, and the implications of misfolding in diseases such as cystic fibrosis and prion disorders.

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theresa4444allen
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Chapter 4

Native Proteins – biologically active (preferred) conformation of a protein


o Any structure is acceptable as long as the covalent binds aren’t
broken.
o Having lowest free energy; most stable conformation, proteins
need multiple stable forms to be deemed stable
o Structure is critical to function/ exceptions do occur that rely on
entropy
o Physiological conditions range 5-65 kJ/mol
o Unfolded protein is subject to higher conformational entropy –
bummer
Interactions and bonds play large roles
 Covalent disulfide
o Uncommon but very strong
o Fading away due to the environments more than likely containing
oxygen
o Found in extracellular proteins with reductants (glutathione) I.E
hormone insulin
 Weak interactions are important in production of different structures;
secondary, tertiary, quaternary
 200-46 kJ/mol to break a SINGLE covalent bond.
o Contrast a weak bond 0.4-30 kJ/mol
Hydrophobic amino acids
o Solvation layer – ultimate layer of hydrogen bonds that act as a shield
to protect the protein
o Account for physiological conditions; hydrogen bonds in a protein is
made by the same entropic effect
Ion pairs or salt bridges can be 50/50
o Focus on dipoles +/-
o salt bridges increases closer to a LOWER dielectric constant
o salt bridge (ionic bonds)
Vander-waals interactions are put together to promote the folding function
Structural pattern RULES :
1. Hydrophobic residues are largely buries in the protein interior away
from water.
2. # of hydrogen bond/ionic interactions in a protein is maximized->
reducing the # of unpaired ionic groups
Peptide bonds
o Rigid and planar
o C-C-N-C alpha carbon residues
o 6 atoms of a peptide group lie in a single plane

Secondary Structures – polypeptide focused on the alpha helix and beta


sheets
o 1948 Pauling – simplest for of maximized used of the internal hydrogen
bonding.
o A basic spiral – left helix rotates counter clockwise and right helix
rotates clockwise
o Characteristics of dihedral angles
Alpha helix
 Constraint produces Proline and Glycine residue
 Proline
o Has a amine bond which cannot bend thus ‘kink’ in the
proceeded helix
o Thus not commonly found in secondary structures
 Glycine
o is only C-H bonds which in turn bends MORE than other amino
acids
 Constraints: bulkiness of the R groups, interactions/ lack there of
between spaced out R groups
Beta formation
 1951 Pauling and Corey – extended the conformation of polypeptide
chains
o Zig-zag rather than helix
o Backbone is hydrogen bonds
 Adjacent backbones produce ‘sheets’
 R-Groups of adjacent AA protrude creating an alternating pattern
structure
o Beta and Alpha exsist unanimously as the protein develops

In class :
Single monomers don’t present with anything above tertiary structure
Beta sheets are mostly more stable than alpha helixes
Beta-alpha-Beta arranged a/b barrel

Motif (fold) of proteins can be a basis for a protein structural classification:


organized by families and superfamilies.
Families ~4000 strong evolutionary connection
Superfamilies – folded similarly but not much coding in common
Oligomer – multi-
repeating structural unit=promoter

Proteostasis ( Protein maintenance of the active set of cellular Pro under


given set of condition ) protein ‘cell cycle’

Renaturation (refolding) Anfinsen experiment added urea to disulfide,


completely unfolded,
Due to protein folds and unfolds - > neurodegenerative disorders.
 Cystic Fibrosis – defects in membrane bound protein CFTransmembrane
conductance regulator)
Prion – misfolded brain proteins – chronic/death

Ideals to discover and observe protein isolation/activation


Protein Crystallography
Nuclear spins NMR Spectrophy
 2D NMR techs.
o NOESY – distance heavy
o TOCSY – bonds observation coupling
Cryo-EM ; good for extremely large structures (i.e ribosomes)

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