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Week 2 Notes

The document discusses the differences between acute and chronic health effects caused by pollution, outlining their symptoms, development times, and treatment requirements. It also details the steps in risk assessment, including hazard characterization, exposure assessment, dose-response assessment, and risk characterization. Additionally, it provides formulas for calculating average daily dose and cancer risk associated with pollutants, using trichloroethylene as a case study.

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0% found this document useful (0 votes)
3 views22 pages

Week 2 Notes

The document discusses the differences between acute and chronic health effects caused by pollution, outlining their symptoms, development times, and treatment requirements. It also details the steps in risk assessment, including hazard characterization, exposure assessment, dose-response assessment, and risk characterization. Additionally, it provides formulas for calculating average daily dose and cancer risk associated with pollutants, using trichloroethylene as a case study.

Uploaded by

p25vagish
Copyright
© All Rights Reserved
We take content rights seriously. If you suspect this is your content, claim it here.
Available Formats
Download as PDF, TXT or read online on Scribd

CVL100:

Environmental
Science
Risk assessment
Are my health effects caused by pollution???
What's the difference between acute and
chronic health effects?
Acute health effects Chronic health effects
● generally develop suddenly ● are slower to develop
● are accompanied by distinct ● may have any number of warning
symptoms signs or no signs at all
● require urgent or short-term care, ● may worsen over an extended
and get better once they are treated. period of time—months to years
Acute Chronic

● Immediately after, or shortly after ● Exposure occurring over a longer


exposure, symptoms and side effects period of time than acute.
appear in an individual. ● Chronic health effects are often
● Acute side effects often are a result to a associated with low levels of exposure
high level exposure over a short period over a long period of time.
of time. ● For many that experience this type of
● In most cases, the effects can be exposure, the effects are not
reversed after exposure has ceased. reversible.
Acute health effects Chronic health effects

● Eye irritation ● Internal organ damage

● Rashes ● Birth defects (Teratogenicity)

● Nausea ● DNA damage, mutation (Genotoxocity)

● Headache ● Cancer causing (Carcinogenecity)

● Vomiting
● Respiratory issues
● Gastro-intestinal issues
Health effects of Arsenic

Source: [Link]
chronic-exposure-to-inorganic-arsenic-from-contaminated_fig1_313448493
How can we estimate the risks?
Steps in risk assessment
● Hazard characterization (e.g. pollutants in air, water or soil)

● Exposure assessment (e.g. PM inhaled, fluoride in drinking water, pesticides in


vegetables)

● Dose-response assessment (adverse health effects based on degree of exposure)

● Risk characterization (Potential health impacts and estimation of risks)


Source- Exposure pathway- Receptor

[Link]
Exposure routes
● Ingestion
● Inhalation
● Dermal absorption

Source: WHO
USEPA
Dose-Response curve

No observed adverse effect level


Lethal dose (LD) or Lethal concentration (LC)

Test organisms
Uncertainty factors

Source: [Link]
Average daily dose or intake
𝐶 × 𝐸𝑅 × 𝐸𝐷 × 𝐸𝐹
𝐴𝐷𝐷 𝑜𝑟 𝐴𝐷𝐼 =
𝐵𝑊 × 𝐴𝑇
Mostly used for acute exposure
ADD- Average daily dose, mg/kg/day
C- Pollutant concentration, mg/L or mg/g or mg/cm2 or mg/m3
ER- Exposure rate, L/day or g/day or cm2/day or m3/day
ED- Exposure duration, days/year
EF- Exposure frequency, year
BW- Body weight, kg
AT- Averaging time, day (Pathway specific period of exposure)
Chronic daily intake
𝐶 × 𝐸𝑅 × 𝐸𝐷 × 𝐸𝐹
𝐶𝐷𝐼 =
𝐵𝑊 × 𝐴𝑇
CDI- Chronic daily intake or LADD, life-time average daily dose (calculated for a life-time)

AT- Life expectancy (Normally taken as 70 years)

Absorption ratio to differentiate potential and applied dose


Non-Carcinogenic pollutants
● Hazard quotient, HQ=ADD/RfD

● RfD= Reference dose or acceptable daily intake (Acceptable dose which could be
consumed without any adverse effects)

HQ<1

● Hazard index (HI) for multiple pollutants=σ 𝐻𝑄𝑖


Carcinogenic pollutants
● Risk= CDI*SF

● SF=slope factor (also known as potency factor, PF)

● Risk <10-6
● An industrial spill has resulted in the release of trichloroethylene (TCE) into the
environment. The concentration of TCE in the groundwater and the
surrounding air are estimated to be 5 µg/L and 3 µg/m3, respectively. What is
the cancer risk for a local resident who consumes 2 L of groundwater per day
and spends 1 h every day in the playground near the spill?

EF- 350 days/year


ED- 30 years (Duration of stay at one residence)
AT- 70 years (Life expectancy)
Body weight- 70 kg
Air intake- 21 m3/day
Oral PF- 0.046 kg-day/mg
Inhalational PF- 0.006 kg-day/mg
Ingestion route

2𝐿 𝑑𝑎𝑦
5 µg/L × × 350 𝑦𝑒𝑎𝑟 × 30𝑦𝑒𝑎𝑟
𝑑𝑎𝑦
𝐶𝐷𝐼 =
70 𝑘𝑔 × 70 𝑦𝑒𝑎𝑟

mg
𝐶𝐷𝐼 = 0.02
kg. day

𝑅𝑖𝑠𝑘 = 0.046 × 0.02 = 9.2 × 10−4


Inhalation route

21 𝑚3 1ℎ 𝑑𝑎𝑦
3 µg/m3 × × × 350 × 30𝑦𝑒𝑎𝑟
𝑑𝑎𝑦 24ℎ 𝑦𝑒𝑎𝑟
𝐶𝐷𝐼 =
70 𝑘𝑔 × 70 𝑦𝑒𝑎𝑟

mg
𝐶𝐷𝐼 = 0.135
kg. day
𝑅𝑖𝑠𝑘 = 0.135 × 0.006 = 8.1 × 10−4

𝑇𝑜𝑡𝑎𝑙 𝑟𝑖𝑠𝑘 = 9.2 × 10−4 + 8.1 × 10−4 = 1.73 × 10−3 > 10−6
● What must be the maximum allowable concentrations in the air and water if the
cancer risk is to be maintained below 10−6 ?

● How will the results vary for a 40 kg woman?

● What would be the risk for an infant if soil ingestion is also taken into account?
Suggested reading
Integrated Risk Information System
[Link]

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