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Allergic Rhinitis LAQ-1

Allergic rhinitis is an IgE-mediated inflammatory disorder of the nasal mucosa triggered by specific allergens, leading to symptoms such as sneezing, rhinorrhea, and nasal obstruction. It can be classified by duration (intermittent or persistent) and severity (mild or moderate-severe), with management strategies including allergen avoidance, pharmacotherapy, immunotherapy, and surgery for structural issues. Common treatments involve antihistamines and intranasal corticosteroids, with immunotherapy indicated for persistent symptoms despite optimal medical treatment.

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0% found this document useful (0 votes)
3 views4 pages

Allergic Rhinitis LAQ-1

Allergic rhinitis is an IgE-mediated inflammatory disorder of the nasal mucosa triggered by specific allergens, leading to symptoms such as sneezing, rhinorrhea, and nasal obstruction. It can be classified by duration (intermittent or persistent) and severity (mild or moderate-severe), with management strategies including allergen avoidance, pharmacotherapy, immunotherapy, and surgery for structural issues. Common treatments involve antihistamines and intranasal corticosteroids, with immunotherapy indicated for persistent symptoms despite optimal medical treatment.

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siddiqueirfan235
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Long Essay Question (LAQ): Allergic Rhinitis

Definition
Allergic rhinitis is an IgE-mediated inflammatory disorder of the nasal mucosa occurring after exposure to a
specific allergen in a sensitized individual, characterized by sneezing, watery rhinorrhea, nasal obstruction, and
nasal itching.

Etiology
Common Allergens
Inhalant Allergens
• House dust mites
• Pollens (grass, weeds, trees)
• Animal dander
• Fungal spores
• Cockroach allergens

Occupational Allergens
• Flour dust
• Cotton dust
• Wood dust
• Latex

Predisposing Factors
• Family history of atopy
• Asthma
• Eczema
• Environmental pollution
• Passive smoking

Classification
According to Duration
Intermittent
• Symptoms <4 days/week or <4 weeks

Persistent
• Symptoms >4 days/week and >4 weeks

According to Severity
Mild
• Normal sleep
• Normal daily activities
• No troublesome symptoms

Moderate–Severe
• Sleep disturbance
• Impaired daily activities
• Reduced work or school performance
• Troublesome symptoms

(ARIA classification)

Pathophysiology
Sensitization Phase
• Allergen is processed by antigen-presenting cells.
• Th2 lymphocytes stimulate B cells.
• B cells produce allergen-specific IgE.
• IgE binds to mast cells in the nasal mucosa.

Re-Exposure Phase
Early Phase (Minutes)
Mast cell degranulation releases:
• Histamine
• Leukotrienes
• Prostaglandins

Causing:
• Sneezing
• Itching
• Watery rhinorrhea

Late Phase (4–8 Hours)


Eosinophils and other inflammatory cells infiltrate the mucosa, leading to:
• Persistent nasal obstruction
• Mucosal edema
• Hyperreactivity of the nasal mucosa

Clinical Features
Symptoms (Classical Tetrad)
1. Sneezing – repeated paroxysmal sneezing, especially in the morning.
2. Rhinorrhea – profuse watery nasal discharge.
3. Nasal obstruction – intermittent initially, later persistent.
4. Nasal itching – involves nose, palate, throat, and eyes.

Associated Symptoms
• Itchy, watery eyes (allergic conjunctivitis)
• Postnasal drip
• Cough
• Hyposmia or anosmia
• Headache and fatigue

Signs
Allergic Facies
• Open-mouth breathing
• Long face
• Dark circles under eyes (allergic shiners)

Allergic Salute
Frequent upward rubbing of the nose producing a transverse nasal crease.

Nasal Examination
Anterior Rhinoscopy
• Pale, bluish, edematous inferior turbinates
• Thin watery secretions
• Congested but non-friable mucosa

Investigations
• Peripheral blood eosinophil count – supportive but not diagnostic.
• Nasal smear cytology – eosinophils in nasal secretions.
• Serum IgE – total IgE may be elevated; specific IgE identifies allergens.
• Skin prick test – most useful investigation; wheal-and-flare reaction in 15–20 minutes.
• CT PNS if sinusitis or polyps suspected.
• Pulmonary function tests if asthma suspected.

Differential Diagnosis
• Viral rhinitis – fever, malaise, short duration.
• Vasomotor rhinitis – no allergy or eosinophilia.
• Chronic rhinosinusitis – thick mucopurulent discharge.
• Deviated nasal septum – structural obstruction.
• Nasal polyposis – persistent obstruction with polypoidal masses.
• CSF rhinorrhea – unilateral clear watery discharge.

Complications
• Chronic sinusitis
• Nasal polyps
• Otitis media with effusion
• Eustachian tube dysfunction
• Sleep disturbance
• Worsening of bronchial asthma
• Impaired quality of life and school performance in children

Management
Principles
1. Allergen avoidance
2. Pharmacotherapy
3. Immunotherapy
4. Surgery for selected cases

Allergen Avoidance
• Wash bedding in hot water weekly.
• Use mattress and pillow covers.
• Remove carpets and heavy curtains.
• Reduce indoor humidity.
• Keep windows closed during high-pollen seasons.
• Wear masks outdoors.
• Avoid outdoor activities during peak pollen periods.
• Remove or minimize exposure to pets and keep them out of the bedroom.

Pharmacotherapy
Antihistamines
Preferred oral second-generation agents:
• Cetirizine
• Levocetirizine
• Loratadine
• Fexofenadine

They reduce sneezing, itching, and rhinorrhea but are less effective for severe nasal obstruction.

Intranasal Corticosteroids (Most Effective)


• Fluticasone
• Mometasone
• Budesonide
• Beclomethasone

Benefits:
• Best control of nasal obstruction.
• Reduce mucosal inflammation.
• Improve all major nasal symptoms.

Important Exam Point


Intranasal corticosteroids are the single most effective treatment for persistent allergic rhinitis.

Other Drugs
• Intranasal antihistamines: Azelastine, Olopatadine.
• Topical decongestants: Xylometazoline, Oxymetazoline (use for not more than 5–7 days).
• Leukotriene receptor antagonist: Montelukast, especially when associated with asthma.

Immunotherapy
Indications
• Symptoms persist despite optimal medical treatment.
• Identifiable specific allergen.
• Patient wishes to reduce long-term drug use.

Types
• Subcutaneous immunotherapy (SCIT) – gradually increasing doses of allergen extract.
• Sublingual immunotherapy (SLIT) – allergen tablets or drops under the tongue.

Advantages:
• Alters the natural course of allergic disease.
• May reduce progression to asthma.

Surgical Treatment
Surgery is not curative for allergy; it is performed for structural problems.

Indications
• Persistent inferior turbinate hypertrophy.
• Nasal polyps.
• Deviated nasal septum causing obstruction.
• Chronic sinusitis associated with allergic rhinitis.

Procedures
• Inferior turbinate reduction
• Submucosal diathermy
• Radiofrequency turbinate reduction
• Septoplasty
• Functional endoscopic sinus surgery (FESS) when indicated

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