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Internal Resorption

The document discusses various dental conditions including inside resorption, pulp calcification, acute and chronic apical periodontitis, and abscesses, detailing their etiology, symptoms, processes, sensitivity tests, radiographic findings, histology, and treatments. It emphasizes the importance of root canal treatment for several conditions and provides differential diagnoses for dental pain. The document is authored by Dr. Herczegh Anna, an associate professor in the Department of Restorative Dentistry and Endodontics.

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0% found this document useful (0 votes)
2 views19 pages

Internal Resorption

The document discusses various dental conditions including inside resorption, pulp calcification, acute and chronic apical periodontitis, and abscesses, detailing their etiology, symptoms, processes, sensitivity tests, radiographic findings, histology, and treatments. It emphasizes the importance of root canal treatment for several conditions and provides differential diagnoses for dental pain. The document is authored by Dr. Herczegh Anna, an associate professor in the Department of Restorative Dentistry and Endodontics.

Uploaded by

camosundental
Copyright
© All Rights Reserved
We take content rights seriously. If you suspect this is your content, claim it here.
Available Formats
Download as PDF, TXT or read online on Scribd

Inside resorption

Etiology
• inflammation of the pulp (pulpitis chronica or granulomatosa clausa, Palazzi's granuloma)
• immune cells in the granulation tissue and dentinoclasts break down the inside dentin

Symptom
• asymptomatic purple patch on the crown

Process
• Progressive

Clinical appearance
• pink discoloration

Sensitivity test
• response similar to the intact tooth
• in general, it will retain their vitality

Dr. Herczegh Anna PhD


Department of Restorative Dentistry and Endodontics
associate professor
Inside resorption
Rtg
• Radiolucens laesio inside the root

Treatment
• Rootcanal treatment ( rapidly)
• Warm gutta-percha technique, MTA

Dr. Herczegh Anna PhD


Department of Restorative Dentistry and Endodontics
associate professor
Pulp calcificatio
Etiology
• Advancing age
• Persistent stimuli (eg. caries) calcification
• Secondary dentin formation →the cavity system narrows
• At the end of the root cement apposition → foramen apical narrowing
• reduction in amount of blood flow →arteriosclerotic changes
• The number→ of cellular elements is decreasing → collagen bundles can be detected → pulp
fibrosis
• number of blood vessels and nerves decreasing → dentin permeability↓

Symptom
• Asymptomatic
Process
• Progressive
Clinical appearance
• the crown may be discolored ( yellowish)
Sensitivity test
• No response for stimuli or reduced

Dr. Herczegh Anna PhD


Department of Restorative Dentistry and Endodontics
associate professor
Pulp calcificatio
Rtg
• pulp chamber and root canal narrowing, obstruction
• pulp stones
• calcification metamorphosis

Treatment
• If it is necessery rootcanal treatment

Dr. Herczegh Anna PhD


Department of Restorative Dentistry and Endodontics
associate professor
Differencial diagnosis

Dentin sensitivity
• The pain is similar to reversible pulpitis
But the pain is caused by a hydrostatic pressure difference in the dentin which is
mechanically irritates the nerve endings
Irreversible pulpitis
• need to be allocated from periapical inflammation
• in case of apical periodontitis there is axial percussion sensitivity
• in case of periapical abscess there is fistula
• the apical processes in general there are also x-ray findings
Sinusitis
thermal and electrical pain stimuli can not elicit in tooth
typical symptoms of sinusitis are present: pain when you drive forward the head
and pressure cause pain in the front wall of the maxilla
Unlocalizable pain
separation from: jaw, neuralgic pain, otitis media, osteomyelitis, inflammation of
the parotid
Dr. Herczegh Anna PhD
Department of Restorative Dentistry and Endodontics
associate professor
Periodontal diseases

Dr. Herczegh Anna PhD


Department of Restorative Dentistry and Endodontics
associate professor
Acute apical periodontitis
Etiology
• Pulp necrosis
• Pulp inflammation reach the periapical tissue
• Endodontic procedure
• Paradontal process

Symptoms
• Intense pain bite on it or touch to biting
• inflammation→edema fluid → pressure on the nerve
• Elongation (feeling)
Process
• Depends from virulence and exposure time
• Stronger infection than the body’s defenses→ intense acute inflammation
• Increased edema fluid → pain
• Untreated case→abcessus

Dr. Herczegh Anna PhD


Department of Restorative Dentistry and Endodontics
associate professor
Acute apical periodontitis

Sensitivity test
• Cold –
• Warm can be+
Palpation: the tooth can be movable
Percussion: Intense pain Do not use it !!!
Rtg
• Negative
• Root membrane gap widening ( after10-12 days)
• Positive, after chronic inflammation : resorption bone defense

Treatment
• Reduce occlusion
• Root canal treatment ( open???? close?????) 2 sites

Dr. Herczegh Anna PhD


Department of Restorative Dentistry and Endodontics
associate professor
Chronic apical periodontitis

Etiology
• Permanent presence of moderate virulence bacteria
• Untreated pulp necrosis
• Incomplete rootcanal treatment (dead space)
• Periodontitis apicalis acuta

Symptom
• Discomfort for biting

Process
• Chronical inflammation → reabsorption of lamina dura and spongiest asymptomatic or
mild discomfort
Sensitivity test
• Cold- , electrical stimulus-
• Warm: can be + ( closed tooth)
Palpation, Percussion: can cause sensitivity

Dr. Herczegh Anna PhD


Department of Restorative Dentistry and Endodontics
associate professor
Chronic apical periodontitis
Rtg
• Destruction of periapical tissues

Histology
• Granulomatic tissue + macrophag, histiocyta plasma cell
• Cyst: eosinophil fluid multilayered epithelium, connective tissue,, granulomatic cells
Treatment
• Rootcanal treatment (1 or 2 sites)

Dr. Herczegh Anna PhD


Department of Restorative Dentistry and Endodontics
associate professor
Dr. Herczegh Anna PhD
Department of Restorative Dentistry and Endodontics
associate professor
Dr. Herczegh Anna PhD
Department of Restorative Dentistry and Endodontics
associate professor
Acute apical abscess
Etiology
• Flare-up of a chronic process - the body reacts severely to irritativ factors from
necrotized pulp. It rarely develops from an acute process.
Symptoms
• Pain, systemic manifestation (fever, malaise, leukocytosis)
Process
• Inflammation leads to abscess → melts or spreads diffusely in the periapical space.

Sensitivity test
• No response to heat and electrical stimuli
Palpation
• The tooth may become loose, swelling
Percussion
• Intense pain, forbidden to use it

Dr. Herczegh Anna PhD


Department of Restorative Dentistry and Endodontics
associate professor
Abscessus apicalis acuta
Rtg
• Thickened ,widened root membrane gap, then a picture typical of chronic apical
periodontitis.
Histology
• Abscess PMN leukocyte
• Granulomatous tissue
• Purulent exudate

Treatment
• Drainage
• Rootcanal treatment (Closing in 2 sessions?????)
• Antibiotic
• Oral surgeon

Dr. Herczegh Anna PhD


Department of Restorative Dentistry and Endodontics
associate professor
Chronic apical abscess (periodontitis apicalis
suppurativa)
Etiology
• abscess formation after pulp necrosis or periodontitis apicalis chronica

Symptoms
• fistula → drainage → no pain

Process
Chronic apical abscess →fistula,
parulis

Dr. Herczegh Anna PhD


Department of Restorative Dentistry and Endodontics
associate professor
Chronic apical abscess (periodontitis
apicalis suppurativa)
Sensitivity test
• no response to heat and electrical stimuli
Rtg
• Similar to chronic apical periodontitis
Histology
• Similar to chronic apical periodontitis + fistula
Treatment
• Rootcanal treatment (1 site)

Dr. Herczegh Anna PhD


Department of Restorative Dentistry and Endodontics
associate professor
Condensing osteitis

Etiology
• Inflammation spreads into the root membrane gap
• Pulp necrosis or irreversible pulpitis cause it
• Persistent minor infection, mild toxin effect →local defense mechanism reduces
inflammation

Symptom
• Asymptomatic, discomfort

Process
• Inflammation → Increased osteoblast activity → hyperossification

Sensitivity test -
Percussion -

Dr. Herczegh Anna PhD


Department of Restorative Dentistry and Endodontics
associate professor
Condensing osteitis
Rtg
• Irregular, diffuse, concentric radiopaque bone around the root apex, hyperossificacio
• It most often develops around the roots of lower molar teeth.

Histology
• Irregular bone structure

Treatment
• Rootcanal treatment

Dr. Herczegh Anna PhD


Department of Restorative Dentistry and Endodontics
associate professor
Differencial diagnosis

Anatomical formulas:
• canalis mandibulae, foramen mentale, sinus maxillaris, foramen incisivum,

Benign lesions that do not originate from the periodontium:


• cementoma, monostaticus fibroticus
• dysplasia, traumatic bone cyst, ameloblastoma,
• central hemangioma

Malignant lesions not originating from the dental pulp:


• chondrosarcoma, myeloma

Dr. Herczegh Anna PhD


Department of Restorative Dentistry and Endodontics
associate professor

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