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Liver Function Tests

The document outlines liver function tests (LFTs), detailing the liver's structure, functions, and the significance of various parameters measured in LFTs. It discusses indications for LFTs, their limitations, and the interpretation of results related to liver diseases. Additionally, it provides information on bilirubin metabolism, liver enzyme studies, and profiles of common liver diseases.

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jinal8898
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0% found this document useful (0 votes)
5 views30 pages

Liver Function Tests

The document outlines liver function tests (LFTs), detailing the liver's structure, functions, and the significance of various parameters measured in LFTs. It discusses indications for LFTs, their limitations, and the interpretation of results related to liver diseases. Additionally, it provides information on bilirubin metabolism, liver enzyme studies, and profiles of common liver diseases.

Uploaded by

jinal8898
Copyright
© All Rights Reserved
We take content rights seriously. If you suspect this is your content, claim it here.
Available Formats
Download as PDF, TXT or read online on Scribd

LIVER FUNCTION TESTS

Liver is composed of three system

✓The hepatocytes – concerned with metabolic reactions, macromolecular

synthesis and degradation and metabolism of xenobiotics

✓The biliary system – metabolism of bilirubin and bile salts,

✓The reticuloendothelial system – concerned with immune system and

production of heme and globin metabolites


FUNCTIONS OF LIVER
•Storage of Glycogen, Iron and vitamins

•Deamination and transamination of amino acids

•Ammonia detoxification

•Fetal life - hematopoiesis occurs in the liver and immune hemolysis

•Catabolism of steroid hormones


LFT – INDICATIONS
•Screen for liver disease

•Identify nature of liver disease (hepatocellular, cholestatic, or infiltrative)

•Assess severity and prognosis of liver disease

•Follow up course of liver disease


LFT – LIMITATIONS
•Lack sensitivity (i.e. may be normal in some liver diseases like cirrhosis)

•Lack specificity (i.e. may be abnormal in non-liver disorders e.g. serum


albumin is low in nephrotic syndrome)
BILIRUBIN METABOLISM
Differential Diagnosis Based on Elevated LFTs

Hepatocellular pattern: Elevated aminotransferases out of proportion


to alkaline phosphatase

Cholestatic pattern: Elevated alkaline phosphatase +gamma glutamyl


transferase + bilirubin out of proportion to AST and ALT
PARAMETERS
SERUM BILURUBIN
Total serum bilirubin - Normal <1.2 mg/dl

Pre-hepatic/ unconjugated/ indirect – 0.2 – 0.9 mg/dl

Post-hepatic/ conjugated/ direct – 0.1 – 0.3 mg/dl


SYNTHETIC AND METABOLIC
FUNCTIONS OF LIVER
•Proteins –

✓Total serum protein ( 6- 8 g/dl)

✓Sr Albumin (3.50-5.20 g/dl)

✓Sr Globulin (1.80-3.40 g/dl)

✓A/G ratio (normal ratio is >1.5)

•Prothrombin time (PT) (11-16 sec)


SERUM ALBUMIN
✓60% of total proteins in serum

✓Determines synthetic capacity of hepatocytes

✓Half life-20 days

✓Acute liver disease (viral hepatitis)- little change

✓Chronic liver disease (cirrhosis)- decrease(marker of chronicity-low)


ALBUMIN GLOBULIN RATIO
Formula : albumin level
(Total protein- Albumin level)

Normal value : 1.5- 1.80


PROTHROMBIN TIME (PT)
Vitamin K dependent factors - II, VII, IX, and X

In hepatocellular disease-Deficient Synthesis of these factors

Obstructive jaundice - vitamin K (fat-soluble) not absorbed due to the absence of


bile in the intestine.

Prolonged PT - hepatocellular disease and obstructive jaundice.


BLOOD AMMONIA
GIT portal vein liver

Ammonia to nontoxic urea(urea cycle)

↑ Blood Ammonia levels -


Fulminant hepatic failure
Cirrhosis
Reye’s syndrome
“Shunting” of portal blood to systemic circulation
Gastrointestinal hemorrhage
inherited deficiencies of urea cycle enzymes
LIVER ENZYME STUDIES
•Serum aspartate aminotransferase or
AST (SGOT)

•Serum alanine aminotransferase or ALT


(SGPT)

•Serum alkaline phosphatase or ALP

•γ-Glutamyl transferase or GGT


ALT/AST
AST – Liver > cardiac muscle > skeletal muscle > kidneys > brain

ALT– specifically liver

Both normal – 0 – 30 IU/L

Hepatocellular injury → ↑ membrane permeability → ↑ Serum levels

AST is rapidly cleared by reticuloendothelial system ALT>AST always


HEPATIC CAUSES OF MARKEDLY ELEVATED
ALT/AST (>1000 IU/L)
AST/ALT RATIO- ALCOHOLIC
LIVER DISEASE
Normal – 0.7 – 1.4

2:1 → Suggestive of ALD

3:1 → Highly suggestive of ALD


ALP- ALKALINE PHOSPHATASE
Normal – 40-120 IU/L

ALP – Liver > bone > placenta > small intestine

Considered significant – if elevated 4 times normal

Physiological increase (upper limit -never 4 times )

 Pregnancy

 >60 years of age

 After a fatty meal

 Children and adolescents


ALP – MARKED ELEVATION
(>4X)
Cholestasis

Infiltrative liver disease (eg: cancer,


amyloidosis)

Paget’s disease of bone


ALT-predominant:

Acute or chronic viral hepatitis, steatohepatitis, acute Budd-Chiari syndrome,


ischemic hepatitis, autoimmune, hemochromatosis, medications/toxins,
autoimmune, alpha1-antitrypsin deficiency, Wilson disease, Celiac disease

AST-predominant:

Alcohol-related, cirrhosis, non-hepatic (hemolysis, myopathy, thyroid


disease, exercise)
SERUM GGT
Normal GGT (liver)– 10 – 47 IU/L

Elevated
 Cholestasis (more specific is GGT )

 ALD

 Acute hepatitis – recovery phase


LFTS PROFILES IN COMMON LIVER DISEASES
Disorder Bilirubin AST/ALT ALP Albumin PT

Hemolysis/ N/ Normal Normal Normal Normal


Gilbert unconjugated

Acute Both Elevated N/ <3 times N Normal Usually


hepatocellular elevated; ALT/AST Normal
disease bilirubinuria +

Chronic Both Elevated N/<3 times N Decrease Prolonged


hepatocellular elevated; <300 u/l
disease Bilirubinuria +
LFTS PROFILES IN COMMON LIVER DISEASES
THANK YOU

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