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Chapter 11 Notes

Chapter 11 discusses gene regulation, focusing on how proteins interact with DNA to control gene expression in prokaryotes and eukaryotes. It explains the structure and function of operons, the role of regulatory genes, and the impact of chromosomal modifications and epigenetics on gene expression. Additionally, it highlights the mechanisms of gene expression regulation, the implications of mutations in cancer development, and the influence of lifestyle choices on cancer risk.

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0% found this document useful (0 votes)
3 views22 pages

Chapter 11 Notes

Chapter 11 discusses gene regulation, focusing on how proteins interact with DNA to control gene expression in prokaryotes and eukaryotes. It explains the structure and function of operons, the role of regulatory genes, and the impact of chromosomal modifications and epigenetics on gene expression. Additionally, it highlights the mechanisms of gene expression regulation, the implications of mutations in cancer development, and the influence of lifestyle choices on cancer risk.

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shrinikethsiv
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© All Rights Reserved
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How Genes Are

Controlled
Chapter 11
11.1 Proteins interacting with DNA turn prokaryotic
genes on or off in response to environmental change

● Gene regulation - the turning on and off of genes


● Gene expression - the overall process by which genetic information flows
from genes to proteins
○ Central dogma of molecular biology
Operons
● Cluster of genes with related functions + control sequences
● Exist in prokaryotes
The lac Operon

1. 3 adjacent lactose-utilization genes


2. A promoter sequence, where RNA polymerase binds
3. An operator sequence, where a repressor can bind and block the binding of
RNA polymerase

Regulatory gene
● Located outside the operon
● Continually codes for a repressor protein
The lac Operon - when lactose is NOT present
The lac Operon - when lactose is present
Inducible vs. Repressible Operons
Activators

● Proteins that turn operons on by:


○ Binding to DNA
○ Stimulating gene transcription
11.2 Chromosome structure and chemical
modifications can affect gene expression

● Differentiation
○ Involves cell specialization
○ Controlled by turning specific sets of genes on or off
● Almost all of the cells in an organism contain an identical genome
○ Differences between cell types due to selective gene expression
DNA Packing

● Multiple levels of folding and coiling


● Tends to prevent gene expression by preventing RNA polymerase from
binding
Epigenetic Inheritance

● Inheritance mechanisms not directly related to nucleotide sequence


● Chemical modifications
○ Methylation - more tightly packed
○ Acetylation - less tightly packed
X Chromosome Inactivation

● In female mammals, one X chromosome is highly compacted


○ Early in embryonic development
○ Barr body

[Link]
room-resources/x-inactivation
11.3 Complex assemblies of proteins control
eukaryotic transcription

● Initiation of gene expression most important in


regulating gene expression
● Eukaryotes use regulatory proteins - activators
and repressors - that promote or block the
binding of RNA polymerase
● Default mode for most genes is typically “off”
○ Must be turned “on”
● RNA polymerase requires transcription factors
11.4 Alternative RNA splicing

● Produces different mRNAs from same transcript


● More than one polypeptide from the same gene
11.6 Later stages of gene expression are also
subject to regulation

● Gene expression can be regulated by


○ Breakdown of mRNA
○ Initiation of translation
○ Protein activation
○ Protein breakdown
11.7
Multiple mechanisms
regulate gene expression in
eukaryotes

Gene expression can be:


● Turned on or off
● Sped up
● Slowed down
11.10 Signal Transduction
Pathways
● Series of molecular changes that convert a
signal on the target cell’s surface to a
specific response within the cell
11.15 Cancer results from mutations in genes that
control division
● Control systems have malfunctioned
● Often due to changes in gene expression
● Mutations in 2 types of genes
○ Proto-oncogenes mutate to oncogenes
○ Tumor-suppressor genes inactivated by mutations
11.16 Multiple genetic changes underlie the
development of cancer

Colon cancer
● Oncogene results in increased cell
division in colon lining
● Additional mutations cause the
growth of a small tumor (polyp) in
colon wall
● Additional mutations lead to a
malignant tumor with the potential
to metastasize
11.17 Faulty proteins can
interfere with normal signal
transduction pathways
● Proto-oncogenes and tumor
suppressor genes often code for
proteins involved in signal transduction
pathways → gene expression

● 2 main types of signal transduction


pathways lead to the synthesis of
proteins that influence cell division
11.18 Lifestyle choices can reduce cancer risk

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