Name Of Patient : Mr. VEENIT YADAV Reg.

No : 0012601070113
Age/Gender : 31 Years/Male Collected On : 07-Jan-2026 02:52 PM
Referred By : Dr. SELF Received On : 07-Jan-2026 03:53 PM
Sample Type/ID : EDTA/694453 Reported On : 07-Jan-2026 04:01 PM
Client Panel : DL175-- Report Status : Final Report
Referred By Lab : Self

HAEMATOLOGY
Test Name Results Unit [Link]

CBC, COMPLETE BLOOD COUNT

HAEMOGLOBIN (HB) 16.2 gm/dL 13.0-17.0
Method Name: Photometry
R B C (Red Blood Cell Count) 4.9 millions/cumm 4.50-5.50
Method Name: Impedance
PCV (Packed cell volume)/Hct 41.80 % 30.0-55.0
Method Name: Calculated
M C V (Mean Corpuscular Volume) 85.9 fL 70.0-101.0
Method Name: Calculated
M C H (Mean Corpuscular Hb) 33.2 pg 27-32
Method Name: Calculated
M C H C (Mean Corp Hb Conc) 38.70 g/dL 30.0-36.0
Method Name: Calculated
RDW (CV) 13.2 % 12.0-14.0
Method Name: By Automated/Calculated
RDW (SD) 40.1 fL 35.0- 46.0
Method Name: Calculated
MENTZER INDEX 17.53
Method Name: Calculated
PLATELET COUNT 254 10^3/µL 150-410
Method Name: Impedance
PCT 0.20 % 0.12-0.40
Method Name: By Calculated
MPV 9.70 fL 7.10-12.50
Method Name: Plt Histogram
PDW 12.00 fL 8.30-18.0
Method Name: Calculated
P-LCC 72.00 10^3/µL 45-95
Method Name: Calculated
P-LCR 28.40 % 11-45

TLC (Total Leucocyte Count) 13.7 10^3/µL 4.50-11.00

Method Name: Impedance /manual
DIFFERENTIAL LEUCOCYTE COUNT
NEUTROPHIL 74.00 % 40.0-80.0
Method Name: Impedance & absorbance/manual
LYMPHOCYTES 20.00 % 20.0-40.0
Method Name: Impedance & absorbance/manual
EOSINOPHIL 2.00 % 1.0-6.0
Method Name: Impedance & absorbance/manual
MONOCYTES 4.00 % 2.0-10.0

DR. UMA SHANKAR DR. ANKIT [Link] KUMAR SINGH

MBBS, MD (PATH) MBBS. MD (PATH) PhD. in Microbiology
Page 1 of 12 CONSULTANT PATHOLOGIST CONSULTANT PATHOLOGIST Consultant Microbiologist
 Name Of Patient : Mr. VEENIT YADAV Reg. No : 0012601070113
Age/Gender : 31 Years/Male Collected On : 07-Jan-2026 02:52 PM
Referred By : Dr. SELF Received On : 07-Jan-2026 03:53 PM
Sample Type/ID : EDTA/694453 Reported On : 07-Jan-2026 04:01 PM
Client Panel : DL175-- Report Status : Final Report
Referred By Lab : Self

HAEMATOLOGY
Test Name Results Unit [Link]
Method Name: Impedance & absorbance/manual
BASOPHILS 0.00 % 1.0-2.0
Method Name: Impedance & absorbance/manual
ANC (ABSOLUTE NEUTROPHIL COUNT) 10.14 10^3/µL 2.0-7.0
Method Name: Calculated
ALC (ABSOLUTE LYMPHOCYTE COUNT) 2.74 10^3/µL 1.0-3.0
Method Name: Calculated
AEC (ABSOLUTE EOSINOPHIL COUNT) 0.27 10^3/µL 0.02-0.50
Method Name: Calculated
AMC (ABSOLUTE MONOCYTE COUNT) 0.55 10^3/µL 0.20-1.00
Method Name: Calculated
ABC (ABSOLUTE BASOPHIL COUNT) 0.00 10^3/µL 0.00-0.10
Method Name: Calculated
NEUTROPHIL/LYMPHOCYTE RATIO (NLR) 3.70 Ratio 0.43-2.75
Method Name: Calculated
PLATELET/LYMPHOCYTE RATIO (PLR) 92.70 Ratio 36.63-149.13
Method Name: Calculated

Interpretation:

A complete blood count (CBC) is a blood test used to evaluate your overall health and detect a wide range of disorders, including anemia, infection and
leukemia.

Detect abnormalities in your blood that may be signs of disease.

Diagnose or monitor many different disorders, conditions and infections.
Evaluate your overall health.
Rule out conditions, disorders and disease.
Monitor various blood diseases.

Disorder related to having a high white blood count Disorder related to having a low white blood count
include include:
Autoimmune and inflammatory diseases, conditions
Disease of the immune system, such as the HIV/AIDS
that cause the immune system attack healthy tissues.
Bacterial or viral infection Lymphoma, a cancer of the bone morrow
Cancers such as leukemia and Hodgkin disease Diseases of the liver or spleen
Allergic reactions

Neutrophil to lymphocyte ratio (NLR) was identified as an independent risk factor for critical illness in patients with COVID-19 [Link]-
lymphocyte ratio (PLR) is a novel inflammatory marker, which may be used in many diseases for predicting inflammation and mortality.
Note :- The result obtained relate only to the sample given/ received & tested. A single test result is not always indicative of a disease, it has
to be correlated with clinical data for interpretation.

*** End Of Report ***

Laboratory is NABL Accredited

DR. UMA SHANKAR DR. ANKIT [Link] KUMAR SINGH

MBBS, MD (PATH) MBBS. MD (PATH) PhD. in Microbiology
Page 2 of 12 CONSULTANT PATHOLOGIST CONSULTANT PATHOLOGIST Consultant Microbiologist
 Name Of Patient : Mr. VEENIT YADAV Reg. No : 0012601070113
Age/Gender : 31 Years/Male Collected On : 07-Jan-2026 02:52 PM
Referred By : Dr. SELF Received On : 07-Jan-2026 03:53 PM
Sample Type/ID : Whole Blood EDTA/694453 Reported On : 07-Jan-2026 04:56 PM
Client Panel : DL175-- Report Status : Final Report
Referred By Lab : Self

HAEMATOLOGY
Test Name Results Unit [Link]

ESR (ERYTHROCYTE SEDIMENTATION RATE)

ESR (Wintrobes METHOD) 14 mm/1st hr. 0 - 15
Method Name: Wintrobe
CLINICAL COMMENTS:

Erythrocyte sedimentation rate (ESR) is a relatively simple, inexpensive, non-specific test that indirectly measures the degree of inflammation present in
the body. Inflammation is part of the body's immune response. It can be acute, developing rapidly after trauma, injury or infection, for example, or can
occur over an extended time (chronic) with conditions such as autoimmune diseases or cancer. Moderately elevated ESR occurs with inflammation but also
with anemia, infection, pregnancy, and with aging. A very high ESR can be seen in severe infection, marked by an increase in globulins, systemic
vasculitis, polymyalgia rheumatica or temporal arteritis. People with multiple myeloma or Waldenstrom's macroglobulinemia (tumors that make large
amounts of immunoglobulins) typically have very high ESRs

Factors increasing ESR:

Advanced age
Anemia
Pregnancy
High fibrinogen
Macrocytosis
Kidney problems
Thyroid disease
Some cancers, such as multiple myeloma
Infection

Factors decreasing ESR :

Microcytosis
Low fibrinogen
Polycythemia
Marked leukocytosis

Note :- The result obtained relate only to the sample given/ received & tested. A single test result is not always indicative of a disease, it has
to be correlated with clinical data for interpretation.

*** End Of Report ***

Laboratory is NABL Accredited

DR. UMA SHANKAR DR. ANKIT [Link] KUMAR SINGH

MBBS, MD (PATH) MBBS. MD (PATH) PhD. in Microbiology
Page 3 of 12 CONSULTANT PATHOLOGIST CONSULTANT PATHOLOGIST Consultant Microbiologist
 Name Of Patient : Mr. VEENIT YADAV Reg. No : 0012601070113
Age/Gender : 31 Years/Male Collected On : 07-Jan-2026 02:52 PM
Referred By : Dr. SELF Received On : 07-Jan-2026 03:53 PM
Sample Type/ID : EDTA/694453 Reported On : 07-Jan-2026 04:29 PM
Client Panel : DL175-- Report Status : Final Report
Referred By Lab : Self

HAEMATOLOGY
Test Name Results Unit [Link]

HBA1C (GLYCOSYLATED HEMOGLOBIN)

HbA1c (NGSP) 5.80 % Non-Diabetic < 6.0
Good Control 6.0-7.0
Weak Control 7.0-8.0
Poor control > 8.0
Estimated Average Glucose 119.76 mg/dL 68-125
Method Name: Calculated

CLINICAL COMMENT:-
American Diabetes Association
Reference Group HbA1c in %
Non diabetic adults >=18 years < 6.0
At risk (Prediabetes) 5.7 - 6.4
Diagnosing Diabetes >= 6.5

1. HbA1c is used for monitoring diabetic [Link] reflects the mean plasma glucose over three months.
2. Trends in HbA1c are a better indicator of diabetic control than a solitary test.
3. HbA1c target in pregnancy is to attain level <6 % .
4. HbA1c target in pediatric age group is to attain level < 7.5 %.

REMARKS:-

1. Shortened RBC life span –HbA1c test will not be accurate when a person has a condition that affects the average lifespan of red blood cells (RBCs), such
as hemolytic anemia or blood loss. When the lifespan of RBCs in circulation is shortened, the A1c result is falsely low and is an unreliable
measurement of a person's average glucose over time.
2. Abnormal forms of hemoglobin – The presence of some hemoglobin variants, such as hemoglobin S in sickle cell anemia, may affect certain methods
for measuring A1c. In these cases, fructosamine can be used to monitor glucose control.

Method :- Ion-exchange high-performance liquid chromatography (HPLC).

Reference :- American Diabetes Associations. Standards of Medical Care in Diabetes 2015

Note :- The result obtained relate only to the sample given/ received & tested. A single test result is not always indicative
of a disease, it has to be correlated with clinical data for interpretation.

*** End Of Report ***

Laboratory is NABL Accredited

DR. UMA SHANKAR DR. ANKIT [Link] KUMAR SINGH

MBBS, MD (PATH) MBBS. MD (PATH) PhD. in Microbiology
Page 4 of 12 CONSULTANT PATHOLOGIST CONSULTANT PATHOLOGIST Consultant Microbiologist
 Name Of Patient : Mr. VEENIT YADAV Reg. No : 0012601070113
Age/Gender : 31 Years/Male Collected On : 07-Jan-2026 02:52 PM
Referred By : Dr. SELF Received On : 07-Jan-2026 03:53 PM
Sample Type/ID : Serum/694451 Reported On : 07-Jan-2026 04:47 PM
Client Panel : DL175-- Report Status : Final Report
Referred By Lab : Self

BIOCHEMISTRY
Test Name Results Unit [Link]

LFT,LIVER FUNCTION TESTS

BILIRUBIN TOTAL,Serum 0.54 mg/dL 0.2-1.2
Method Name: Malloy-Evelyn Modified
BILIRUBIN (Direct) 0.18 mg/dL 0.0-0.30
Method Name: Malloy- Evelyn modified
BILIRUBIN (Indirect) 0.36 mg/dL 0.2-1.0
Method Name: Calculated
SGOT (AST) ,Serum 102.30 U/L 0-35
Method Name: Kinetic UV
SGPT (ALT), Serum 83.90 U/L 5-45
Method Name: Kinetic UV
ALKALINE PHOSPHATASE ,Serum 97.20 U/L 53.0-128.0
Method Name: IFCC-Enzymatic-kinetic
GAMMA G.T. ,Serum 31.30 U/L 5.0-55.0
Method Name: G-glutamyl-p-nitroanili
TOTAL PROTEIN , Serum 7.14 g/dL 6.4-8.3
Method Name: Biuret
ALBUMIN,Serum 4.6 g/dL 3.5-5.2
Method Name: Bromocresol Green (BCG)-colorimetric
Globulin 2.54 g/dL 2.50 - 3.5
Method Name: Calculated
A/G Ratio ,Serum 1.81 Ratio 1.0 - 2.3
Method Name: Calculated
AST/ALT Ratio, Serum 1.22 Ratio 0-5
Method Name: Calculated
CLINICAL COMMENT:-
ALT is the best test for detecting hepatitis. People with acute Hepatitis often have very high levels of ALT. while those chronic have moderately raise
[Link] is found in the liver, heart and other muscles in the body. When liver, heart or muscles are damaged they release AST into the [Link]
is an orange pigment formed when RBCs break down as a natural part of the body’s ongoing renewal. Raised level show the liver isn’t functioning too
[Link] is found mainly in the liver and can be used to confirm results if you have high ALP [Link] ALP and GGT are higher when there is liver or bile
duct [Link] is found in the liver and bones. If ALP levels are increased and levels of some of the other LFT substances are high it can be assumed that
higher ALP is coming from the [Link] PROTIEN measure albumin and all other proteins in blood. Including antibodies made to help fight off
infections. ALBUMIN is the main protein made by the liver and low levels may mean the liver is damaged or the albumin is being lost through damaged
kidneys.
Reference ranges are from Teitz fundamental of clinical chemistry 8th ed (2018). Reference ranges vary between laboratories.
Note :- The result obtained relate only to the sample given/ received & tested. A single test result is not always indicative of a disease, it has
to be correlated with clinical data for interpretation.

*** End Of Report ***

Laboratory is NABL Accredited

DR. UMA SHANKAR DR. ANKIT [Link] KUMAR SINGH

MBBS, MD (PATH) MBBS. MD (PATH) PhD. in Microbiology
Page 5 of 12 CONSULTANT PATHOLOGIST CONSULTANT PATHOLOGIST Consultant Microbiologist
 Name Of Patient : Mr. VEENIT YADAV Reg. No : 0012601070113
Age/Gender : 31 Years/Male Collected On : 07-Jan-2026 02:52 PM
Referred By : Dr. SELF Received On : 07-Jan-2026 03:53 PM
Sample Type/ID : Serum/694451 Reported On : 07-Jan-2026 04:47 PM
Client Panel : DL175-- Report Status : Final Report
Referred By Lab : Self

BIOCHEMISTRY
Test Name Results Unit [Link]

KFT,KIDNEY FUNCTION TESTS

UREA,Serum 22.8 mg/dL 17.0 -44.0
Method Name: Enzymatic UV Kinetic (Urease, UV)
CREATININE , Serum 0.72 mg/dl 0.70-1.30
Method Name: Enzymatic kinetic
URIC ACID ,Serum 6.10 mg/dL 3.5 - 7.2
Method Name: Uricase Enzymatic-colorimetric
SODIUM ,Serum 137.9 mmol/L 135-146
Method Name: ISE
POTASSIUM ,Serum 3.65 mmol/L 3.5-5.1
Method Name: ISE
CHLORIDE,Serum 96.60 mmol/L 96-107
Method Name: ISE
BLOOD UREA NITROGEN (BUN),Serum 10.65 mg/dl 6 - 20
Method Name: Urease – UV
BUN/CREATININE RATIO 14.79 Ratio 10-20
Method Name: Calculated
UREA/CREATININ RATIO 31.67 Ratio 20-35
Method Name: Calculated
CALCIUM, Serum 10.30 mg/dL 8.50-10.50
Method Name: Arsenazo III- Colorimetric

Clinical comments: -
Urea/creatinine: High levels of urea & creatinine indicate kidney disease or some from damage due to blocked blood flow to kidneys. Increased in any renal
functional [Link] Acid: A high uric acid level occurs when your kidneys don't eliminate uric acid efficiently. Things that may cause this slow-
down in the removal of uric acid include rich foods, being overweight, having diabetes, taking certain diuretics (sometimes called water pills) and drinking
too much alcohol. Electrolyte: Levels of electrolytes in the blood are affected by kidney disease in different ways, When they are out of balance this can
affect your body’s fluids levels and your pH. [Link]: Help with muscle contraction, nerve signaling, blood clotting, cell division and maintaining
bone and teeth.
Reference ranges are from Teitz fundamental of clinical chemistry 8th ed (2018).Reference ranges vary between laboratories
Note: - The result obtained relate only to the sample given/ received & tested. A single test result is not always indicative of a disease, it has
to be correlated with clinical data for interpretation.

*** End Of Report ***

Laboratory is NABL Accredited

DR. UMA SHANKAR DR. ANKIT [Link] KUMAR SINGH

MBBS, MD (PATH) MBBS. MD (PATH) PhD. in Microbiology
Page 6 of 12 CONSULTANT PATHOLOGIST CONSULTANT PATHOLOGIST Consultant Microbiologist
 Name Of Patient : Mr. VEENIT YADAV Reg. No : 0012601070113
Age/Gender : 31 Years/Male Collected On : 07-Jan-2026 02:52 PM
Referred By : Dr. SELF Received On : 07-Jan-2026 03:53 PM
Sample Type/ID : PLASMA F/694452 Reported On : 07-Jan-2026 04:25 PM
Client Panel : DL175-- Report Status : Final Report
Referred By Lab : Self

BIOCHEMISTRY
Test Name Results Unit [Link]

BLOOD GLUCOSE FASTING

GLUCOSE FASTING 105.10 mg/dL 70.0 - 100.0
Method Name: Enzymatic-Colorimetric (GOD-PAP)

Comment:-
Conditions that can result in an elevated blood glucose level include: Acromegaly, Acute stress (response to trauma, heart attack, and stroke for
instance),Chronic kidney disease, Cushing syndrome, Excessive consumption of food, Hyperthyroidism, Pancreatitis.A low level of glucose may indicate
hypoglycemia, a condition characterized by a drop in blood glucose to a level where first it causes nervous system symptoms.(sweating, palpitations,
hunger, trembling, and anxiety), then begins to affect the brain (causing confusion, hallucinations, blurred vision, and sometimes even coma and death). A
low blood glucose level (hypoglycemia) may be seen with:Adrenal insufficiency, Drinking excessive alcohol, Severe liver disease,
Hypopituitarism,Hypothyroidism, Severe infections, Severe heart failure, Chronic kidney (renal) failure, Insulin overdose, Tumors that produce insulin
(insulinomas), Starvation.

NOTE-The result obtained relate only to the sample given/ received & tested. A single test result is not always indicative of a disease; it has
to be correlated with clinical data for interpretation.

*** End Of Report ***

Laboratory is NABL Accredited

DR. UMA SHANKAR DR. ANKIT [Link] KUMAR SINGH

MBBS, MD (PATH) MBBS. MD (PATH) PhD. in Microbiology
Page 7 of 12 CONSULTANT PATHOLOGIST CONSULTANT PATHOLOGIST Consultant Microbiologist
 Name Of Patient : Mr. VEENIT YADAV Reg. No : 0012601070113
Age/Gender : 31 Years/Male Collected On : 07-Jan-2026 02:52 PM
Referred By : Dr. SELF Received On : 07-Jan-2026 03:53 PM
Sample Type/ID : Serum/694451 Reported On : 07-Jan-2026 04:47 PM
Client Panel : DL175-- Report Status : Final Report
Referred By Lab : Self

BIOCHEMISTRY
Test Name Results Unit [Link]

LIPID PROFILE
TOTAL CHOLESTEROL, Serum 193.20 mg/dL Desirable<200
Method Name: Enzymatic-colorimetric (CHOD-PAP) Borderline 200-239
High > 239
TRIGLYCERIDE , Serum 100.00 mg/dL Normal <150
Method Name: Enzymatic (GPO-PAP)-colorimetric Borderline 150-199
High 200-499
Very High >499
HDL-CHOLESTEROL , Serum 46.30 mg/dL >40 Recommended range
Method Name: Direct measure
NON HDL CHOLESTEROL,Serum 146.90 mg/dL <130
Method Name: Calculated
LDL CHOLESTEROL,Serum 126.90 mg/dL Optimal <100
Method Name: Calculated Borderline 130-159
High 160-189
Very High >190
VLDL CHOLESTEROL ,Serum 20.00 mg/dL 0.0- 30.0
Method Name: Calculated
TOTAL CHOLESTEROL /HDL RATIO ,Serum 4.17 Ratio low Risk < 3.0
Method Name: Calculated Average Risk3.0-5.0
High Risk > 5.0
LDL / HDL CHOLESTEROL RATIO 2.74 Ratio 1.5-3.5
Method Name: Calculated
HDL/LDL CHOLESTEROL RATIO 0.36 Ratio <3.50
Method Name: Calculated
10-12 hours fasting is mandatory for lipid profile. In case of the lipemic sample (highly turbid due to lipoproteins mainly chylomicrons), the test
cannot be performed on the specimen but can be repeated after a week following fat free diet.

CLINICAL COMMENTS
Lipids, such as cholesterol, triglycerides, and fatty acids, are fat and substances like fat used as a source of fuel by the body. Lipid levels can be an important
measure of health. For example, a person who has high cholesterol has an increased risk of heart disease and stroke.
There are two main forms of cholesterol:-
Low-density lipoprotein (LDL) is called "bad cholesterol." Most efforts to lower cholesterol are aimed at reducing levels of LDL.
High-density lipoprotein (HDL) is called "good cholesterol." It can help remove excess cholesterol from the blood [Link] levels of triglycerides raise your risk for
heart disease and other serious problems.
Reference ranges are from Teitz fundamental of clinical chemistry 8th ed (2018). Reference ranges vary between laboratories.
Note :- The result obtained relate only to the sample given/ received & tested. A single test result is not always indicative of a disease, it has
to be correlated with clinical data for interpretation.

*** End Of Report ***

Laboratory is NABL Accredited

DR. UMA SHANKAR DR. ANKIT [Link] KUMAR SINGH

MBBS, MD (PATH) MBBS. MD (PATH) PhD. in Microbiology
Page 8 of 12 CONSULTANT PATHOLOGIST CONSULTANT PATHOLOGIST Consultant Microbiologist
 Name Of Patient : Mr. VEENIT YADAV Reg. No : 0012601070113
Age/Gender : 31 Years/Male Collected On : 07-Jan-2026 02:52 PM
Referred By : Dr. SELF Received On : 07-Jan-2026 03:53 PM
Sample Type/ID : Serum/694451 Reported On : 07-Jan-2026 04:47 PM
Client Panel : DL175-- Report Status : Final Report
Referred By Lab : Self

BIOCHEMISTRY
Test Name Results Unit [Link]

IRON PROFILE
Iron 103.00 µg/dL 59.0-158.0
Method Name: TPTZ
UIBC 215.30 µg/dL 120.0-347.0
Method Name: NiTRO-PSAP
TOTAL IRON BINDING CAPACITY 318.30 µg/dL 240.0-450.0
Method Name: Calculated
TRANSFERRIN SATURATION 32.36 % 20.0-50.0
Method Name: Calculated

CLINICAL COMMENTS:-
Iron is an essential nutrient that, among other functions, is needed in small quantities to help form normal red blood cells (RBCs).
Serum iron test: measures the level of iron in the liquid portion of the blood.
Transferrin test: directly measures the level of transferrin in the blood. Transferrin is the protein that transports iron around in the body. Under normal
conditions, transferrin is typically one-third saturated with iron. This means that about two-thirds of its capacity is eld in reserve.
TIBC (total iron-binding capacity): measures the total amount of iron that can be bound by proteins in the blood. Since transferrin is the primary iron-
binding protein, the TIBC test is a good indirect measurement of transferrin availability.
UIBC (unsaturated iron-binding capacity): The UIBC test determines the reserve capacity of transferrin, i.e., the portion of transferrin that has not yet
been saturated with iron. UIBC also reflects transferrin levels.
Transferrin saturation: a calculation that reflects the percentage of transferrin that is saturated with iron (100 x serum iron/TIBC).
Serum ferritin: reflects the amount of stored iron in the body.
INCREASED IN:

Hemosiderosis of excessive iron intake (e.g. repeated blood transfusion, iron therapy, iron containing vitamins).
Decreased formation of RBCs (thalassemia, pyridoxal deficiency anaemia).
Increased destruction of RBCs (hemolytic anaemia).
Acute liver damage
Progesteronal birth control pills & pregnancy
Premenstrual elevation
cute iron toxicity

DECREASED IN:

Iron deficiency anaemia

Normochromic anaemia of infections & chronic diseases
Nephrosis -Menstruation
Diurnal variation: Normal in mid morning, low values in mid afternoon, and very low values near midnight.

Note :- The result obtained relate only to the sample given/ received & tested. A single test result is not always indicative of a disease, it has
to be correlated with clinical data for interpretation

*** End Of Report ***

Laboratory is NABL Accredited

DR. UMA SHANKAR DR. ANKIT [Link] KUMAR SINGH

MBBS, MD (PATH) MBBS. MD (PATH) PhD. in Microbiology
Page 9 of 12 CONSULTANT PATHOLOGIST CONSULTANT PATHOLOGIST Consultant Microbiologist
 Name Of Patient : Mr. VEENIT YADAV Reg. No : 0012601070113
Age/Gender : 31 Years/Male Collected On : 07-Jan-2026 02:52 PM
Referred By : Dr. SELF Received On : 07-Jan-2026 03:53 PM
Sample Type/ID : Serum/694451 Reported On : 07-Jan-2026 06:51 PM
Client Panel : DL175-- Report Status : Final Report
Referred By Lab : Self

IMMUNOLOGY
Test Name Results Unit [Link]

VITAMIN B12 (CYANOCOBALAMIN)

VITAMIN B12 (Cyanocobalamin) 169.20 pg/mL 211-911
Method Name: Chemiluminescence immunoassay (CLIA)

CLINICAL COMMENT:-

Vitamin B-12 (Methylcobalamin) is an important vitamin for many body functions, such as brain health, blood cell production, and proper nerve
[Link] B12 deficiency may be due to lack of IF secretion by gastric mucosa (eg, gastrectomy, gastric atrophy) or intestinal malabsorption (eg,
ileal resection, small intestinal diseases). Vitamin B12 deficiency frequently causes macrocytic anemia, hyperthyroidism, glossitis, peripheral neuropathy,
weakness, hyperreflexia, ataxia, loss of proprioception, poor coordination, and affects behavioral changes. These manifestations may occur in any
combination; many patients have the neurologic defects without macrocytic anemia. Pernicious anemia is a macrocytic anemia caused by vitamin B12
deficiency that is due to a lack of IF secretion by gastric mucosa. High levels of B-12 may increase risk for cancer, liver disease, certain types of leukemia,
diabetes, kidney failure. Serum methylmalonic acid and homocysteine levels are also elevated in vitamin B12 deficiency states.
Decreased Levels:-
Lack of Intrinsic factor: Total or partial gastrectomy, Atrophic gastritis, Intrinsic factor antibodies
Malabsorption: Regional ileitis, resected bowel, Tropical Sprue, Celiac disease, pancreatic insufficiency, bacterial overgrowth & achlorhydria
Loss of ingested vitamin B12: fish tapeworm
Dietary deficiency: Vegetarians
Congenital disorders: Orotic aciduria & transcobalamine deficiency
Increased demand: Pregnancy specially last trimester
Increased Levels:-
Chronic renal failure, Congestive heart failure, Acute & Chronic Myeloid Leukemia, Polycythemia vera, Carcinomas with liver metastasis, Liver disease,
Drug induced cholestasis & Protein malnutrition.
Reference ranges are from Teitz fundamental of clinical chemistry 8th ed (2018). Reference ranges vary between laboratories
Note :- The result obtained relate only to the sample given/ received & tested. A single test result is not always indicative of a disease, it has
to be correlated with clinical data for interpretation.

*** End Of Report ***

Laboratory is NABL Accredited

DR. UMA SHANKAR DR. ANKIT [Link] KUMAR SINGH

MBBS, MD (PATH) MBBS. MD (PATH) PhD. in Microbiology
Page 10 of 12 CONSULTANT PATHOLOGIST CONSULTANT PATHOLOGIST Consultant Microbiologist
 Name Of Patient : Mr. VEENIT YADAV Reg. No : 0012601070113
Age/Gender : 31 Years/Male Collected On : 07-Jan-2026 02:52 PM
Referred By : Dr. SELF Received On : 07-Jan-2026 03:53 PM
Sample Type/ID : Serum/694451 Reported On : 07-Jan-2026 04:52 PM
Client Panel : DL175-- Report Status : Final Report
Referred By Lab : Self

IMMUNOLOGY
Test Name Results Unit [Link]

THYROID FUNCTION TEST(TFT)

T3 (Triiodothyronine) ,Serum 1.34 ng/mL 0.70-2.04
Method Name: Electrohemiluminescence immunoassay (CLIA)
T4 (Thyroxine) ,Serum 7.88 µg/dl 4.60-10.50
Method Name: Electrochemiluminescence immunoassay (CLIA)
Ultrasensitive TSH (Thyroid Stimulating 2.517 µIU/ml 0.40-4.50
Hormone),Serum
Method Name: Chemiluminescence immunoassay (CLIA)

Interpretation:-
TSH is a glycoprotein hormone secreted by the anterior pituitary. TSH is a labile hormone & is secreted in a pulsatile manner throughout the day and is
subject to several non-thyroidal pituitary influences. Significant variations in TSH can occur with circadian rhythm, hormonal status, stress, sleep
deprivation, caloric intake, medication & circulating antibodies.
REFERENCE RANGE in uIU/mL (As per Tietz
REFERENCE GROUP, Pregnancy
Fundamentals of clinical chemistry 7th ed)
1st Trimester 0.30– 4.50
2nd Trimester 0.50 – 4.60
3rd Trimester 0.80 – 5.20
REFERENCE GROUP REFERENCE RANGE in uIU/mL
1-4 days 1.00-39.00
5 days – 20 wks 1.70-9.10
21 wks – 20 years 0.70 – 6.40
Males > 20 years 0.40– 4.50
Adult Females (> 20 years) 0.40– 4.50

Clinical Use
· Diagnose Hypothyroidism and Hyperthyroidism
· Monitor T4 replacement or T4 suppressive therapy
· Quantify TSH levels in the subnormal range.

TSH levels are subject to circadian variation, reaching peak levels between 2-4 AM and min between 6-10 PM. The variation is the order of 50% hence time
of the day has influence on the measures serum TSH concentration. Dose and time of drug intake also influence the test result.

Disclaimer-TSH is an important marker for the diagnosis of thyroid dysfunction. Recent studies have shown that the TSH distribution progressively shifts
to a higher concentration with age ,and it is debatable whether this is due to a real change with age or an increasing proportion of unrecognized thyroid
disease in the elderly.

Reference ranges are from Teitz fundamental of clinical chemistry 7th ed.

NOTE-The result obtained relate only to the sample given/ received & tested. A single test result is not always indicative of a disease; it has
to be correlated with clinical data for interpretation.

*** End Of Report ***

Laboratory is NABL Accredited

DR. UMA SHANKAR DR. ANKIT [Link] KUMAR SINGH

MBBS, MD (PATH) MBBS. MD (PATH) PhD. in Microbiology
Page 11 of 12 CONSULTANT PATHOLOGIST CONSULTANT PATHOLOGIST Consultant Microbiologist
 Name Of Patient : Mr. VEENIT YADAV Reg. No : 0012601070113
Age/Gender : 31 Years/Male Collected On : 07-Jan-2026 02:52 PM
Referred By : Dr. SELF Received On : 07-Jan-2026 03:53 PM
Sample Type/ID : Serum/694451 Reported On : 07-Jan-2026 06:30 PM
Client Panel : DL175-- Report Status : Final Report
Referred By Lab : Self

IMMUNOLOGY
Test Name Results Unit [Link]

VITAMIN D3 TOTAL (25-HYDROXY CHOLECALCIFEROL)

VITAMIN D3 (25 OH) ,Serum 25.70 ng/mL < 20 Deficiency
Method Name: Chemiluminescence immunoassay (CLIA) 20- 30 Insufficiency
30-100 Sufficiently
>100 Toxicity

SUMMARY:-
This test is done to determine if you have too much or too little vitamin D in your [Link]-than-normal levels can be due to a vitamin D deficiency,
which can result from: Lack of exposure to sunlight Lack of enough vitamin D in the diet Liver and kidney diseases Poor food absorption Use of certain
medicines, including phenytoin, phenobarbital, and rifampin
Comments:-
Vitamin D total assay is used as an aid in the assesment of Vitamin D sufficiency in [Link] D is acquired either by exposure to sunlight or ingestion
of food containing vitamin D. It is metabolized to vit D, 25 hydroxy in the liver in the first step by vit D,25-hydroxylase system. A small amount of it further
gets metabolized by hydroxylation in kidney to vit D 1,25 dihydroxy. Since vit D, 25 hydroxy is the predominant circulating form of Vit D in normal
population, it is considered to be the most reliable index of vit D status. In children, severe deficiency leads to bone-malformation, known as rickets. Milder
degrees of insufficiecy are believed to cause reduced efficiency in the utilization of dietary calcium.
The measurement of 25-OH-D is becoming increasingly important in the management of patients with various disorders of calcium metabolism associated
with Rickets, neonatal hypocalcemia, pregnancy, nutritional and renal osteodystrophy, hypoparathyroidism, and postmenopausal state.
Increased levels are found in Vit. D intoxication.
Decreased levels are detected in Rickets, osteomalacia, secondary hyperparathyroidism, malabsorption of vit D (e.g. liver diseases, cholestasis), and
diseases that increase Vit D metabolism ( Tuberculosis, sarcoidosis, primary hyperparathyroidism).
Note:- I t s h o u l d b e t a k e n i n t o c o s i d e r a t i o n t h a t d i f f e r e n c e s i n V i t a m i n D ( 2 5 - O H ) l e v e l s m a y e x i s t w i t h r e s p e c t t o g e n d e r , a g e , s e a s o n ,
geographical latitude and ethnic groups. The result obtained relate only to the sample given/ received & tested. A single test result is not
always indicative of a disease, it has to be correlated with clinical data for interpretation .

*** End Of Report ***

Laboratory is NABL Accredited

DR. UMA SHANKAR DR. ANKIT [Link] KUMAR SINGH

MBBS, MD (PATH) MBBS. MD (PATH) PhD. in Microbiology
Page 12 of 12 CONSULTANT PATHOLOGIST CONSULTANT PATHOLOGIST Consultant Microbiologist