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Tiña: Micosis, Causas y Síntomas

La tiña, o dermatofitosis, es una micosis superficial causada por hongos parásitos que afectan la piel y anexos, siendo más común en niños y varones adultos. Se transmite a través del ambiente, animales y personas infectadas, y su cuadro clínico varía según la localización, presentando síntomas como eritema, escamas y prurito. Los factores predisponentes incluyen humedad, calor, diabetes y el uso prolongado de glucocorticoides.
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0% encontró este documento útil (0 votos)
3 vistas3 páginas

Tiña: Micosis, Causas y Síntomas

La tiña, o dermatofitosis, es una micosis superficial causada por hongos parásitos que afectan la piel y anexos, siendo más común en niños y varones adultos. Se transmite a través del ambiente, animales y personas infectadas, y su cuadro clínico varía según la localización, presentando síntomas como eritema, escamas y prurito. Los factores predisponentes incluyen humedad, calor, diabetes y el uso prolongado de glucocorticoides.
Derechos de autor
© All Rights Reserved
Nos tomamos en serio los derechos de los contenidos. Si sospechas que se trata de tu contenido, reclámalo aquí.
Formatos disponibles
Descarga como DOCX, PDF, TXT o lee en línea desde Scribd

TIÑA

o Sinónimo: dermatofitosis
o Definición: micosis superficial originada por hongos parásitoss de la keratina llamados dermatofitos que afecta a piel y anexos y rara vez afectan tejidos
profundos
o Epidemiología Cabeza Cuerpo Ingle Pies Onicomicosis
 Hongos causales ◦ Niños (98%) ◦ Cualquier ◦ Varones ◦ Varones ◦ Varones
◦ Mujeres adultas edad y adultos adultos adultos
◦ T. rubrum (36-52% hasta 80%)
◦ Nivel sexo ◦ 17% ◦ 20-51% ◦ 30%
◦ M. canis (14-24) socioeconómic ◦ 15-20%
◦ T. tonsurans (15-18%) o bajo
◦ Otros tipos no son comunes en México ◦ 3-28%
o Etiopatogenia
 Grupos ◦ Mala higiene o no secarse de forma adecuada los pies
◦ Trichophyton ◦ Malos hábitos de peinado
◦ Microsporum ◦ M. canis se adquiere a partir de perros y gatos enfermos
◦ Epidermophyton  La infección se limita a estructuras con queratina: capa cornea, pelos
 Formas de contagio: y uñas
◦ Ambiente  Al penetrar la capa cornea las esporas emites filamentos radiados
◦ Animales produciendo lesión anular
◦ Personas enfermas ◦ Pelo: no invade la zona queratógena (franja de Adamson)
 Factores predisponentes: ◦ Uñas:
◦ Humedad ⮡ Borde distal (hiponiquio)
◦ Calor ⮡ Borde lateral (onicomicosis subungueal distal)
◦ Diabetes ⮡ Bordean la cuticua y eponiquio (onicomicosis subungueal
◦ Uso prolongado de proximal)
glucocorticoides ⮡ Superficie dorsal de la lámina ungueal (lecuconiquia
◦ Calzado cerrado tricofítica)

o Cuadro clínico
Cabeza Cuerpo Ingle Pies Onicomicosis
◦ Predomina M. canis (80%) y ◦ M. canis, T. rubrum, T. ◦ En regiones ◦ Interdigital, plantas y ◦ Clasificación
T. tonsurans tonsurans, E. floccosum inguinocrurales y bordes de los pies • Subungueal
◦ Seca: ◦ Eritema y escamas en periné (hasta abdomen ◦ Puede causar grietas, • Distal-lateral
• Descamación y pelos placas redondeadas con y pene) fisuras, descamación, • Blanca superficial
tiñosos (pelos cortos 2- bordes vesiculares ◦ Placas eritoescamosas vesículas, ampollas y • Blanca proximal
3mm, gruesos, activos y prurito de borde vesicular costras melicéricas subungueal
quebradizos, ◦ La variedad microspórica ◦ Cronicidad y prurito (vesiculoampollar, • Distrófica total
deformados y a veces produce placas dan lugar a dishidróticao paroniquia
con una vaina pequeñas (0.5-2 cm) y pigmentación y eccematiforme) o zonas ◦ Engrosamiento, fragilidad,
quebradiza múltiples liquenificación con hiperqueratosis estrías, coloración
• Genera alopecia difusa ◦ Se presenta como ◦ Frecuente en zonas ◦ Se complica por impétigo, amarillenta o marrón
con placas irregulares epidemia familiar con calurosas y erisipela, ides oscura, de predominio
• Los pelos afectados origen común (gato o sedestación larga Tiña de manos (tinea distal
semejan granos de perro infectado) manuum) ◦ Predisponen traumatismos
pólvora (puntos negros) Corticoestropeo ◦ Afecta palmas (cuando y SIDA
◦ Inflamatoria (querión de ◦ Fenómeno inflamatorio afecta dorso parece tiña
Celso) (querión= panal) por dermatofito en sí o corpus)
• Puede afectar cualquier abuso de ◦ T. rubrum
parte de la piel, pero glucocorticoides ◦ Hiperqueratosis difusa,
más común en la cabeza ◦ Eritema acentuado y de descamación, anhidrosis
• Plastrón inflamatorio, extensión más allá de las y eritema
doloroso al tacto, hecho lesiones, placas y estrías ◦ Fenómeno ide
de varias pústulas, atróficas (dermatofítide o
abscesos, úlceras y tricofítide) depende de la
costras melicéricas tiña de pies y se
• Puede curar sola 2-5 manifiesta por vesículas
meses, pero puede (dishidrosis) o
quedar alopecia def. descamación

Tiña del cuerpo tricofítica (T. Tiña (T. rubrum) Tiña en mocasín Blanca superficial
Pelos tiñosos tonsurans)
Tiña tricofítica (T. rubrum) Intertriginosa
Subungueal proximal blanca
Tiña con liquenificación y
Tiña de la cabeza microspórica pigmentación

Tiña microspórica (M. canis) Impétigo secundario


Distrófica total

Querión de Celso (panal de


abejas) Ides por tiña de pies

Tiña corticoestropeada

Querión en etapa alopécica

Common questions

Con tecnología de IA

The dermatophytide phenomenon, also known as 'ide', manifests as vesicular eruptions, erythema, and scaling, often occurring on the hands or feet, depending on the primary site of infection. It is triggered by a hypersensitivity reaction to the dermatophyte antigens, particularly from tinea pedis (athlete’s foot), and does not involve direct fungal invasion at the eruption site. This reaction highlights the systemic immune response elicited by a localized dermatophyte infection .

Treatment strategies for onychomycosis vary depending on the type of fungal invasion and severity. Subungual infections generally require systemic antifungal therapy such as oral terbinafine or itraconazole, specifically for distal-lateral subungual onychomycosis. For milder infections like white superficial onychomycosis, topical antifungals such amorolfine or ciclopirox can be used. In cases of total dystrophic onychomycosis, a combination of oral and topical treatments may be required. It's crucial to consider patient factors, like potential drug interactions and liver function, when prescribing systemic treatments .

In rural settings, environmental transmission of dermatophytosis is more commonly associated with direct contact with infected animals such as livestockand domestic animals (e.g., cats and dogs), which are common carriers of fungi like Microsporum canis. In contrast, urban environments are more conducive to transmission through human-to-human contact due to close living quarters and communal facilities where skin particles and dander containing fungi may accumulate, thereby facilitating spread, especially with species like Trichophyton rubrum .

Untreated tinea pedis can lead to several complications, including secondary bacterial infections like impetigo and cellulitis. The maceration and fissuring between the toes provide an entry point for bacteria, leading to inflammation and possible ulceration. Chronic infection can also contribute to 'ides' or immune-mediated dermatophytide reactions, and if the infection advances, it may cause onychomycosis and spread to other body areas. Long-term, untreated infections may lead to significant skin disruption and lichenification .

The primary factors that predispose an individual to dermatophytosis include high humidity, heat, diabetes, prolonged use of glucocorticoids, wearing closed shoes, poor hygiene, and not drying feet properly. These factors create an environment conducive to fungal growth by increasing moisture and warmth, which are ideal conditions for dermatophyte survival and proliferation. Poor hygiene and direct contact with infected animals like dogs and cats, especially with M. canis, also facilitate the transmission and establishment of the infection by bringing fungi in contact with keratinized tissues like skin, nails, and hair .

Corticosteroids, when used inappropriately, exacerbate dermatophyte infections by dampening the immune response, leading to more extensive and severe skin manifestations known as tinea corticoestropeada. In this context, corticosteroids can cause the lesions to become more erythematous, extensive, and potentially ichthyotic due to immunosuppression, thereby allowing the fungus to proliferate and invade deeper tissues than typical superficial infections. This misuse can mask symptoms initially, resulting in delayed diagnosis and treatment .

Microsporum canis and Trichophyton tonsurans infections in the scalp have different clinical presentations. M. canis predominantly causes 'grey patch' ringworm, characterized by dry scaling and diffuse alopecia with broken hairs looking like black dots. In comparison, T. tonsurans often leads to 'black dot' tinea capitis, here the hair breaks off near the scalp surface creating black dot-like stubs. M. canis infection is often contracted from infected cats and dogs, while T. tonsurans is more commonly spread from human-to-human contact .

Tinea corporis and tinea cruris affect different areas and demographics but have similar fungal origins. Tinea corporis, also known as ringworm of the body, generally presents as round, erythematous, scaly plaques with clear centers on any skin surface except the scalp, feet, or groin, affecting all ages and both sexes. Tinea cruris, commonly known as jock itch, occurs in the groin and inner thighs, characterized by itchy, red, ring-shaped rashes, more commonly affecting adult males. Both conditions may have active vesicular borders, but tinea cruris often worsens in sweaty and hot conditions, commonly seen in athletes .

Socio-economic status significantly influences the prevalence of dermatophyte infections, particularly in children, who are more susceptible to conditions like tinea capitis. Lower socio-economic groups often face barriers to effective hygiene and healthcare access, increasing their risk of infections. Overcrowded living conditions, limited access to clean water, and insufficient education about infection prevention contribute to higher infection rates. Inadequate management of infected pets and lack of treatment also perpetuate the infection cycle within these communities .

Tinea manuum is characterized by diffuse hyperkeratosis, scaling, erythema, and anhidrosis predominantly affecting the palms, resembling chronic eczema. It is commonly caused by Trichophyton rubrum and often occurs as a result of secondary spread from tinea pedis, sharing a similar etiology and pathogenic fungi. The dermatophyte infection is often diagnosed late because it can mimic other skin conditions. It's related to other dermatophytosis types like tinea pedis and unguium through common fungal sources and transmission routes .

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